Enhanced spontaneous DNA twisting/bending fluctuations unveiled by fluorescence lifetime distributions promote mismatch recognition by the Rad4 nucleotide excision repair complex.

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Enhanced spontaneous DNA twisting/bending fluctuations unveiled by fluorescence lifetime distributions promote mismatch recognition by the Rad4 nucleotide excision repair complex.

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2017 nî lūn-bûn

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Enhanced spontaneous DNA twist ...... otide excision repair complex.

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Enhanced spontaneous DNA twist ...... otide excision repair complex.

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Enhanced spontaneous DNA twist ...... otide excision repair complex.

@en

Enhanced spontaneous DNA twist ...... otide excision repair complex.

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Enhanced spontaneous DNA twist ...... otide excision repair complex.

@en

Enhanced spontaneous DNA twist ...... otide excision repair complex.

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P1476

Enhanced spontaneous DNA twist ...... otide excision repair complex.

@en

P2093

Anjum Ansari

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Debamita Paul

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Hong Mu

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Jung-Hyun Min

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Peter J Steinbach

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Sagnik Chakraborty

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Suse Broyde

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P2860

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Xeroderma pigmentosum group C protein complex is the initiator of global genome nucleotide excision repair

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The comings and goings of nucleotide excision repair factors on damaged DNA

DNA-tension dependence of restriction enzyme activity reveals mechanochemical properties of the reaction pathway.

Xeroderma pigmentosum group C sensor: unprecedented recognition strategy and tight spatiotemporal regulation

Recognition of Damaged DNA for Nucleotide Excision Repair: A Correlated Motion Mechanism with a Mismatched cis-syn Thymine Dimer Lesion.

Solution structure of a trans-opened (10S)-dA adduct of (+)-(7S,8R,9S,10R)-7,8-dihydroxy-9,10-epoxy-7,8,9,10-tetrahydrobenzo[a]pyrene in a fully complementary DNA duplex: evidence for a major syn conformation

P304

1240-1255

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P31

scholarly article

P356

10.1093/NAR/GKX1216

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English

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P478

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P577

2017-12-18T00:00:00Z

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29267981

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P932

5815138

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