Mitochondrial abnormalities and disruption of the neuromuscular junction precede the clinical phenotype and motor neuron loss in hFUSWT transgenic mice.
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Energy metabolism in ALS: an underappreciated opportunity?Amyotrophic Lateral Sclerosis associated FUS mutation shortens mitochondria and induces neurotoxicityThe physiological and pathological biophysics of phase separation and gelation of RNA binding proteins in amyotrophic lateral sclerosis and fronto-temporal lobar degeneration
P2860
Mitochondrial abnormalities and disruption of the neuromuscular junction precede the clinical phenotype and motor neuron loss in hFUSWT transgenic mice.
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2017 nî lūn-bûn
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name
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
@en
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
@nl
type
label
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
@en
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
@nl
prefLabel
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
@en
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
@nl
P2093
P2860
P50
P356
P1476
Mitochondrial abnormalities an ...... oss in hFUSWT transgenic mice.
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P2093
Caroline Memmi
Gema Vizcay-Barrena
Jacqueline C Mitchell
Leanne Allison
P2860
P304
P356
10.1093/HMG/DDX415
P577
2018-02-01T00:00:00Z