CaMKIIα Mediates the Effect of IL-17 To Promote Ongoing Spontaneous and Evoked Pain in Multiple Sclerosis.

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CaMKIIα Mediates the Effect of IL-17 To Promote Ongoing Spontaneous and Evoked Pain in Multiple Sclerosis.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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name

CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

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CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

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Item

label

CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

@en

CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

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prefLabel

CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

@en

CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

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P1476

CaMKIIα Mediates the Effect of ...... ed Pain in Multiple Sclerosis.

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P2093

Fang Huang

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Xiaoyu Hu

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Zaijie Jim Wang

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P2860

Ca2+/calmodulin-dependent protein kinase II alpha is required for the initiation and maintenance of opioid-induced hyperalgesia

Interleukin-17 production in central nervous system-infiltrating T cells and glial cells is associated with active disease in multiple sclerosis

Autophosphorylation at Thr286 of the alpha calcium-calmodulin kinase II in LTP and learning

Multiple sclerosis

Quantitative assessment of tactile allodynia in the rat paw

A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia

Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis

Spinal upregulation of glutamate transporter GLT-1 by ceftriaxone: therapeutic efficacy in a range of experimental nervous system disorders.

Mortality in multiple sclerosis: a review.

Curcumin attenuates opioid tolerance and dependence by inhibiting Ca2+/calmodulin-dependent protein kinase II α activity

P304

232-244

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scholarly article

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10.1523/JNEUROSCI.2666-17.2017

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English

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P478

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2017-11-16T00:00:00Z

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29146590

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5761434

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