P2X7R antagonism after subfailure overstretch injury of blood vessels reverses vasomotor dysfunction and prevents apoptosis.

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Vascular surgical stretch injury leads to activation of P2X7 receptors and impaired endothelial function.

P2860

Q46856693-B19AB551-9E8A-47DA-8808-EE41B34052BA

P2860

P2X7R antagonism after subfailure overstretch injury of blood vessels reverses vasomotor dysfunction and prevents apoptosis.

Item

http://www.wikidata.org/entity/Q47769804

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh

2017年學術文章

@zh-hant

name

P2X7R antagonism after subfail ...... nction and prevents apoptosis.

@en

P2X7R antagonism after subfail ...... nction and prevents apoptosis.

@nl

type

Item

label

P2X7R antagonism after subfail ...... nction and prevents apoptosis.

@en

P2X7R antagonism after subfail ...... nction and prevents apoptosis.

@nl

prefLabel

P2X7R antagonism after subfail ...... nction and prevents apoptosis.

@en

P2X7R antagonism after subfail ...... nction and prevents apoptosis.

@nl

P1476

P2X7R antagonism after subfail ...... unction and prevents apoptosis

@en

P2093

Colleen Brophy

http://www.w3.org/2001/XMLSchema#string

Daniel Feldman

http://www.w3.org/2001/XMLSchema#string

Reid McCallister

http://www.w3.org/2001/XMLSchema#string

Weifeng Luo

http://www.w3.org/2001/XMLSchema#string

P2860

A novel tumor marker, Niban, is expressed in subsets of thyroid tumors and Hashimoto's thyroiditis

AKT-dependent phosphorylation of Niban regulates nucleophosmin- and MDM2-mediated p53 stability and cell apoptosis

Purinergic receptor-mediated rapid depletion of nuclear phosphorylated Akt depends on pleckstrin homology domain leucine-rich repeat phosphatase, calcineurin, protein phosphatase 2A, and PTEN phosphatases

Systemic administration of an antagonist of the ATP-sensitive receptor P2X7 improves recovery after spinal cord injury

Mechanical stretch: physiological and pathological implications for human vascular endothelial cells

The P2X7 receptor channel: recent developments and the use of P2X7 antagonists in models of disease

A novel gene "Niban" upregulated in renal carcinogenesis: cloning by the cDNA-amplified fragment length polymorphism approach

Hypertonic stress increases T cell interleukin-2 expression through a mechanism that involves ATP release, P2 receptor, and p38 MAPK activation

P2X receptors as cell-surface ATP sensors in health and disease

Pannexin membrane channels are mechanosensitive conduits for ATP

P2888

s11302-017-9585-0

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579-590

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scholarly article

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10.1007/S11302-017-9585-0

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P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Joyce Cheung-Flynn

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2017-09-13T00:00:00Z

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28905300

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P932

5714848

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P2X7R antagonism after subfailure overstretch injury of blood vessels reverses vasomotor dysfunction and prevents apoptosis.

about

P2860

P2860

P2X7R antagonism after subfailure overstretch injury of blood vessels reverses vasomotor dysfunction and prevents apoptosis.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P932