about
Cellular responses to Plasmodium falciparum erythrocyte membrane protein-1: use of relatively conserved synthetic peptide pools to determine CD4 T cell responses in malaria-exposed individuals in Benin, West AfricaExpanding the antimalarial toolkit: Targeting host-parasite interactionsCD4 T-cell subsets in malaria: TH1/TH2 revisitedIn vivo approaches reveal a key role for DCs in CD4+ T cell activation and parasite clearance during the acute phase of experimental blood-stage malariaExtramedullary myelopoiesis in malaria depends on mobilization of myeloid-restricted progenitors by IFN-γ induced chemokinesThe role of animal models for research on severe malariaPSLAP, a protein with multiple adhesive motifs, is expressed in Plasmodium falciparum gametocytesA comprehensive evaluation of rodent malaria parasite genomes and gene expressionEffector memory Th1 CD4 T cells are maintained in a mouse model of chronic malariaVector transmission regulates immune control of Plasmodium virulence.Recovery of an antiviral antibody response following attrition caused by unrelated infection.Screening cDNA expression libraries in lambda gt11 with a T cell hybridoma.Immunity against HIV/AIDS, malaria, and tuberculosis during co-infections with neglected infectious diseases: recommendations for the European Union research priorities.The role of the spleen in malaria.Rapid changes in transcription profiles of the Plasmodium yoelii yir multigene family in clonal populations: lack of epigenetic memory?Functional memory B cells and long-lived plasma cells are generated after a single Plasmodium chabaudi infection in mice.Antibody isotype analysis of malaria-nematode co-infection: problems and solutions associated with cross-reactivity.Tumor necrosis factor alpha p55 receptor is important for development of memory responses to blood-stage malaria infection.ICAM-1 is a key receptor mediating cytoadherence and pathology in the Plasmodium chabaudi malaria modelDistinct kinetics of memory B-cell and plasma-cell responses in peripheral blood following a blood-stage Plasmodium chabaudi infection in miceExpansion of IgG+ B-cells during mitogen stimulation for memory B-cell ELISpot analysis is influenced by size and composition of the B-cell pool.Transcriptomics identified a critical role for Th2 cell-intrinsic miR-155 in mediating allergy and antihelminth immunity.Different regions of the malaria merozoite surface protein 1 of Plasmodium chabaudi elicit distinct T-cell and antibody isotype responses.Complement contributes to protective immunity against reinfection by Plasmodium chabaudi chabaudi parasites.Homeostatic proliferation and IL-7R alpha expression do not correlate with enhanced T cell proliferation and protection in chronic mouse malariaDifferential requirement for cathepsin D for processing of the full length and C-terminal fragment of the malaria antigen MSP1.Targeted delivery of antigen processing inhibitors to antigen presenting cells via mannose receptors.Epitope-specific regulation of immunoglobulin class switching in mice immunized with malarial merozoite surface proteins.Characterization and gene expression analysis of the cir multi-gene family of Plasmodium chabaudi chabaudi (AS).Rodent malarias: the mouse as a model for understanding immune responses and pathology induced by the erythrocytic stages of the parasite.Replication of Plasmodium in reticulocytes can occur without hemozoin formation, resulting in chloroquine resistance.Sequestration and histopathology in Plasmodium chabaudi malaria are influenced by the immune response in an organ-specific mannerMemory B-cell and antibody responses induced by Plasmodium falciparum sporozoite immunization.Phenotypic and functional profiling of CD4 T cell compartment in distinct populations of healthy adults with different antigenic exposureThe relevance of non-human primate and rodent malaria models for humansMouse models of blood-stage malaria infections: immune responses and cytokines involved in protection and pathology.Restoration of pattern recognition receptor costimulation to treat chromoblastomycosis, a chronic fungal infection of the skinRole of gamma interferon during infection with Plasmodium chabaudi chabaudi.The plasma concentration of the B cell activating factor is increased in children with acute malaria.Disruption of IL-21 signaling affects T cell-B cell interactions and abrogates protective humoral immunity to malaria.
P50
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P50
subject
description
hulumtues
@sq
onderzoeker
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researcher
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ricercatrice
@it
հետազոտող
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name
Jean Langhorne
@ast
Jean Langhorne
@en
Jean Langhorne
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Jean Langhorne
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Jean Langhorne
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type
label
Jean Langhorne
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Jean Langhorne
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Jean Langhorne
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Jean Langhorne
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Jean Langhorne
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altLabel
J Langhorne
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J. Langhorne
@en
Langhorne J
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Langhorne J.
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Langhorne
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prefLabel
Jean Langhorne
@ast
Jean Langhorne
@en
Jean Langhorne
@es
Jean Langhorne
@nl
Jean Langhorne
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P1006
P214
P1006
P106
P1153
7004973281
P1960
q2214OQAAAAJ
P21
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P31
P496
0000-0002-2257-9733
P569
2000-01-01T00:00:00Z
P734
P735
P7859
lccn-nb2005014940