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Remyelination after cuprizone-induced demyelination in the rat is stimulated by apotransferrinProline substitutions and threonine pseudophosphorylation of the SH3 ligand of 18.5-kDa myelin basic protein decrease its affinity for the Fyn-SH3 domain and alter process development and protein localization in oligodendrocytesGolli myelin basic proteins regulate oligodendroglial progenitor cell migration through voltage-gated Ca2+ influxAssessment of human sperm protein tyrosine phosphorylation by immunocytochemistry in a clinical andrology laboratory. Preliminary data.Intranasal administration of aTf protects and repairs the neonatal white matter after a cerebral hypoxic-ischemic event.Modulation of canonical transient receptor potential channel 1 in the proliferation of oligodendrocyte precursor cells by the golli products of the myelin basic protein geneImpact of simulated microgravity on oligodendrocyte development: implications for central nervous system repair.Developmental activation of the proteolipid protein promoter transgene in neuronal and oligodendroglial cells of neostriatum in mice.STAT3-mediated astrogliosis protects myelin development in neonatal brain injury.Voltage-gated Ca2+ entry promotes oligodendrocyte progenitor cell maturation and myelination in vitro.Classical 18.5-and 21.5-kDa isoforms of myelin basic protein inhibit calcium influx into oligodendroglial cells, in contrast to golli isoformsRegulation of store-operated and voltage-operated Ca2+ channels in the proliferation and death of oligodendrocyte precursor cells by golli proteins.Oligodendrocytes and myelination: the role of iron.Conditional Deletion of the L-Type Calcium Channel Cav1.2 in Oligodendrocyte Progenitor Cells Affects Postnatal Myelination in Mice.The multiple roles of myelin protein genes during the development of the oligodendrocyte.Inhalation of growth factors and apo-transferrin to protect and repair the hypoxic-ischemic brain.Traumatically injured astrocytes release a proteomic signature modulated by STAT3-dependent cell survival.Increased expression of golli myelin basic proteins enhances calcium influx into oligodendroglial cells.Differential gene expression during development in two oligodendroglial cell lines overexpressing transferrin: a cDNA array analysis.Expression of myelin basic protein in two oligodendroglial cell lines is modulated by apotransferrin through different transcription factors.Overexpression of human transferrin in two oligodendroglial cell lines enhances their differentiation.Apotransferrin promotes the differentiation of two oligodendroglial cell lines.Multiple kinase pathways regulate voltage-dependent Ca2+ influx and migration in oligodendrocyte precursor cells.Golli Myelin Basic Proteins Modulate Voltage-Operated Ca(++) Influx and Development in Cortical and Hippocampal Neurons.L-type voltage-operated calcium channels contribute to astrocyte activation In vitro.Inhibition of the proteasome by lactacystin enhances oligodendroglial cell differentiation.Apotransferrin induces cAMP/CREB pathway and cell cycle exit in immature oligodendroglial cells.Conditional Deletion of the L-Type Calcium Channel Cav1.2 in NG2-Positive Cells Impairs Remyelination in Mice.Golli myelin basic proteins stimulate oligodendrocyte progenitor cell proliferation and differentiation in remyelinating adult mouse brain.Differential effects of apotransferrin on two populations of oligodendroglial cells.Morphological changes of myelin sheaths in rats intracranially injected with apotransferrin.The imidazoline I2 receptor agonist 2-BFI attenuates hypersensitivity and spinal neuroinflammation in a rat model of neuropathic pain.Erratum to: Golli Myelin Basic Proteins Modulate Voltage-Operated Ca++ Influx and Development in Cortical and Hippocampal Neurons.Apotransferrin decreases the response of oligodendrocyte progenitors to PDGF and inhibits the progression of the cell cycle.Regulation of L-type Ca++ currents and process morphology in white matter oligodendrocyte precursor cells by golli-myelin proteinsThe Divalent Metal Transporter 1 (DMT1) Is Required for Iron Uptake and Normal Development of Oligodendrocyte Progenitor CellsEnhanced oligodendrocyte maturation and myelination in a mouse model of Timothy syndromeMuscarinic receptor MR signaling prevents efficient remyelination by human and mouse oligodendrocyte progenitor cellsApotransferrin Decreases Migration and Enhances Differentiation of Oligodendroglial Progenitor Cells in an in vitro SystemCalcium Signaling in the Oligodendrocyte Lineage: Regulators and Consequences
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hulumtues
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հետազոտող
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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Pablo M Paez
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P106
P21
P31
P496
0000-0001-9362-9454