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Activity-Dependent Arc Expression and Homeostatic Synaptic Plasticity Are Altered in Neurons from a Mouse Model of Angelman Syndrome.Decreased Axon Caliber Underlies Loss of Fiber Tract Integrity, Disproportional Reductions in White Matter Volume, and Microcephaly in Angelman Syndrome Model Mice.Neuronal overexpression of Ube3a isoform 2 causes behavioral impairments and neuroanatomical pathology relevant to 15q11.2-q13.3 duplication syndrome.Enhanced Transmission at the Calyx of Held Synapse in a Mouse Model for Angelman Syndrome.Taurine Administration Recovers Motor and Learning Deficits in an Angelman Syndrome Mouse Model.Subcellular organization of UBE3A in human cerebral cortex
P2860
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
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2016年學術文章
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name
Subcellular organization of UBE3A in neurons.
@en
Subcellular organization of UBE3A in neurons.
@nl
type
label
Subcellular organization of UBE3A in neurons.
@en
Subcellular organization of UBE3A in neurons.
@nl
prefLabel
Subcellular organization of UBE3A in neurons.
@en
Subcellular organization of UBE3A in neurons.
@nl
P2093
P2860
P356
P1476
Subcellular organization of UBE3A in neurons.
@en
P2093
Alain C Burette
Benjamin D Philpot
Kristen D Phend
Matthew C Judson
Susan Burette
P2860
P304
P356
10.1002/CNE.24063
P407
P577
2016-06-23T00:00:00Z