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The cell biology of fat expansionIsolation and quantitation of adiponectin higher order complexes.Dermal collagen and lipid deposition correlate with tissue swelling and hydraulic conductivity in murine primary lymphedema.Neuronal and nonneuronal cholinergic structures in the mouse gastrointestinal tract and spleenElevated resistin levels induce central leptin resistance and increased atherosclerotic progression in miceReceptor-mediated activation of ceramidase activity initiates the pleiotropic actions of adiponectin.Targeted deletion of adipocytes by apoptosis leads to adipose tissue recruitment of alternatively activated M2 macrophages.Proteinuria Increases Plasma Phosphate by Altering Its Tubular HandlingLack of "immunological fitness" during fasting in metabolically challenged animals.Impaired humoral immunity and tolerance in K14-VEGFR-3-Ig mice that lack dermal lymphatic drainageAdiponectin promotes functional recovery after podocyte ablation.A driving force for change: interstitial flow as a morphoregulator.Adiponectin, Leptin, and Fatty Acids in the Maintenance of Metabolic Homeostasis through Adipose Tissue Crosstalk.Secondary lymphedema in the mouse tail: Lymphatic hyperplasia, VEGF-C upregulation, and the protective role of MMP-9.VEGFR-3 neutralization inhibits ovarian lymphangiogenesis, follicle maturation, and murine pregnancy.Hypercholesterolemic mice exhibit lymphatic vessel dysfunction and degeneration.Characterization of lymphangiogenesis in a model of adult skin regeneration.Differential transendothelial transport of adiponectin complexes.The Role of Proprotein Convertase Subtilisin/Kexin Type 9 in Nephrotic Syndrome-Associated Hypercholesterolemia.Lymphangiogenesis: fuel, smoke, or extinguisher of inflammation's fire?Renal inflammation and injury are associated with lymphangiogenesis in hypertension.Normal dendritic cell mobilization to lymph nodes under conditions of severe lymphatic hypoplasia.Pathological Type-2 Immune Response, Enhanced Tumor Growth, and Glucose Intolerance in Retnlβ (RELMβ) Null Mice: A Model of Intestinal Immune System Dysfunction in Disease Susceptibility.Nav1.8 neurons are involved in limiting acute phase responses to dietary fat.Hyperplasia, de novo lymphangiogenesis, and lymphatic regression in mice with tissue-specific, inducible overexpression of murine VEGF-D.Retrograde Lymph Flow Leads to Chylothorax in Transgenic Mice with Lymphatic Malformations.Adiponectin alters renal calcium and phosphate excretion through regulation of klotho expression.Regulation of lymphatic capillary regeneration by interstitial flow in skin.VEGF-C promotes the development of lymphatics in bone and bone loss.An Endothelial-to-Adipocyte Extracellular Vesicle Axis Governed by Metabolic StateCooperative and redundant roles of VEGFR-2 and VEGFR-3 signaling in adult lymphangiogenesisComparison of ozone-specific (OZAC) and oxygen radical (ORAC) antioxidant capacity assays for use with nasal lavage fluidEnhancing Renal Lymphatic Expansion Prevents Hypertension in MiceImmune cell trafficking, lymphatics and hypertensionReduced lymphatic function contributes to age-related diseaseVascular Endothelial Growth Factor-D (VEGF-D) Overexpression and Lymphatic Expansion in Murine Adipose Tissue Improves Metabolism in ObesityKlotho regulation by albuminuria is dependent on ATF3 and endoplasmic reticulum stressTime-course of sodium transport along the nephron in nephrotic syndrome: The role of potassiumKidney-specific lymphangiogenesis increases sodium excretion and lowers blood pressure in miceAugmenting Renal Lymphatic Density Prevents Angiotensin II-Induced Hypertension in Male and Female Mice
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description
hulumtues
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Joseph M Rutkowski
@nl
Joseph M Rutkowski
@sl
Joseph M. Rutkowski
@en
Joseph M. Rutkowski
@es
type
label
Joseph M Rutkowski
@nl
Joseph M Rutkowski
@sl
Joseph M. Rutkowski
@en
Joseph M. Rutkowski
@es
prefLabel
Joseph M Rutkowski
@nl
Joseph M Rutkowski
@sl
Joseph M. Rutkowski
@en
Joseph M. Rutkowski
@es
P106
P1153
14424246600
P21
P31
P496
0000-0002-4197-0236