Blocking CCL5-CXCL4 heteromerization preserves heart function after myocardial infarction by attenuating leukocyte recruitment and NETosis.

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Blocking CCL5-CXCL4 heteromerization preserves heart function after myocardial infarction by attenuating leukocyte recruitment and NETosis.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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name

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

@en

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

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Item

label

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

@en

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

@nl

prefLabel

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

@en

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

@nl

P1476

Blocking CCL5-CXCL4 heteromeri ...... ocyte recruitment and NETosis.

@en

P2093

Adelina Curaj

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Christian Weber

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David Schumacher

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Delia Projahn

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Isabella Werner

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Julia Möllmann

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Mareike Staudt

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Tanja Vajen

http://www.w3.org/2001/XMLSchema#string

Tilman M Hackeng

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Tolga Taha Sönmez

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P2860

ESC working group cellular biology of the heart: position paper: improving the preclinical assessment of novel cardioprotective therapies

Platelet factor 4 is chemotactic for neutrophils and monocytes

CCR5 deficiency increases risk of symptomatic West Nile virus infection

Repair after myocardial infarction, between fantasy and reality: the role of chemokines

Interaction of risk factors, comorbidities, and comedications with ischemia/reperfusion injury and cardioprotection by preconditioning, postconditioning, and remote conditioning

CCL5-CCR5 interaction provides antiapoptotic signals for macrophage survival during viral infection

RANTES is required for ischaemia-induced angiogenesis, which may hamper RANTES-targeted anti-atherosclerotic therapy

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2

CCR5 signaling suppresses inflammation and reduces adverse remodeling of the infarcted heart, mediating recruitment of regulatory T cells

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10647

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scholarly article

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10.1038/S41598-018-29026-0

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English

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http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

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Elisa Anamaria Liehn

Rory R. Koenen

Philipp Von Hundelshausen

Sakine Simsekyilmaz

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2018-07-13T00:00:00Z

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P698

30006564

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P932

6045661

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