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Genetic lineage tracing defines myofibroblast origin and function in the injured heart.c-kit+ cells minimally contribute cardiomyocytes to the heartNecroptosis Interfaces with MOMP and the MPTP in Mediating Cell Death.Genetic overexpression of Serpina3n attenuates muscular dystrophy in mice.Cathepsin S Contributes to the Pathogenesis of Muscular Dystrophy in Mice.Progenitors of skeletal muscle satellite cells express the muscle determination gene, MyoD.Thrombospondin expression in myofibers stabilizes muscle membranes.Differential expression of embryonic epicardial progenitor markers and localization of cardiac fibrosis in adult ischemic injury and hypertensive heart disease.Preexisting endothelial cells mediate cardiac neovascularization after injury.Dissection of Thrombospondin-4 Domains Involved in Intracellular Adaptive Endoplasmic Reticulum Stress-Responsive Signaling.Myofiber-specific inhibition of TGFβ signaling protects skeletal muscle from injury and dystrophic disease in mice.Fibroblast-specific TGF-β-Smad2/3 signaling underlies cardiac fibrosis.Autophagic cell death is dependent on lysosomal membrane permeability through Bax and Bak.BEX1 is an RNA-dependent mediator of cardiomyopathy.Identity Crisis for Regenerative Cardiac cKit+ Cells.A multifunctional reporter mouse line for Cre- and FLP-dependent lineage analysis.Gata4-Dependent Differentiation of c-Kit+ Derived Endothelial Cells Underlies Artefactual Cardiomyocyte Regeneration in the Heart.van Berlo et al. replyApplying Modern Transcriptomics to Interrogate the Human Cardiac FibroblastInhibition of Senescence-Associated Genes Rb1 and Meis2 in Adult Cardiomyocytes Results in Cell Cycle Reentry and Cardiac Repair Post-Myocardial Infarction
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wetenschapper
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հետազոտող
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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Onur Kanisicak
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0000-0002-8393-4874