Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
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Nanoparticle inhalation impairs coronary microvascular reactivity via a local reactive oxygen species-dependent mechanismPulmonary nanoparticle exposure disrupts systemic microvascular nitric oxide signalingAir particulate matter and cardiovascular disease: the epidemiological, biomedical and clinical evidenceCircadian influences on myocardial infarctionPulmonary allergic reactions impair systemic vascular relaxation in ragweed sensitive miceC₆₀ exposure augments cardiac ischemia/reperfusion injury and coronary artery contraction in Sprague Dawley ratsDiesel exhaust particulate induces pulmonary and systemic inflammation in rats without impairing endothelial function ex vivo or in vivoPulmonary instillation of multi-walled carbon nanotubes promotes coronary vasoconstriction and exacerbates injury in isolated heartsModest vasomotor dysfunction induced by low doses of C60 fullerenes in apolipoprotein E knockout mice with different degree of atherosclerosisEnvironmentally persistent free radicals decrease cardiac function and increase pulmonary artery pressureExpansion of cardiac ischemia/reperfusion injury after instillation of three forms of multi-walled carbon nanotubesPhotochemically altered air pollution mixtures and contractile parameters in isolated murine hearts before and after ischemiaAdenosine-mediated alteration of vascular reactivity and inflammation in a murine model of asthmaEnvironmentally persistent free radicals compromise left ventricular function during ischemia/reperfusion injuryComparative toxicity of size-fractionated airborne particulate matter collected at different distances from an urban highway.Pulmonary diesel particulate increases susceptibility to myocardial ischemia/reperfusion injury via activation of sensory TRPV1 and β1 adrenoreceptors.A murine model of myocardial ischemia-reperfusion injury through ligation of the left anterior descending arteryEffects of particulate air pollution on cardiovascular health: a population health risk assessment.Ozone co-exposure modifies cardiac responses to fine and ultrafine ambient particulate matter in mice: concordance of electrocardiogram and mechanical responsesEnvironmentally persistent free radicals decrease cardiac function before and after ischemia/reperfusion injury in vivoCoronary artery ligation and intramyocardial injection in a murine model of infarction.Mast cells contribute to altered vascular reactivity and ischemia-reperfusion injury following cerium oxide nanoparticle instillation.Multi-walled carbon nanotubes inhibit regenerative axon growth of dorsal root ganglia neurons of mice.Disrupted Nitric Oxide Metabolism from Type II Diabetes and Acute Exposure to Particulate Air Pollution.Myocardial ischemia, reperfusion, and infarction in chronically instrumented, intact, conscious, and unrestrained miceOutbreak of sudden cardiac deaths in a tire manufacturing facility: can it be caused by nanoparticles?The role of air pollution in myocardial remodeling.Cardiovascular remodeling in response to long-term exposure to fine particulate matter air pollutionStage of the estrous cycle does not influence myocardial ischemia-reperfusion injury in rats (Rattus norvegicus).Cardioprotection via preserved mitochondrial structure and function in the mPer2-mutant mouse myocardiumThe utility of the small rodent electrocardiogram in toxicology.Hazard identification of particulate matter on vasomotor dysfunction and progression of atherosclerosis.From particles to patients: oxidative stress and the cardiovascular effects of air pollution.Nanoparticles and the cardiovascular system: a critical review.Atherosclerosis and vasomotor dysfunction in arteries of animals after exposure to combustion-derived particulate matter or nanomaterials.The mechanisms of air pollution and particulate matter in cardiovascular diseases.Air Pollution and Other Environmental Modulators of Cardiac Function.The cardioprotective effect of vanillic acid on hemodynamic parameters, malondialdehyde, and infarct size in ischemia-reperfusion isolated rat heart exposed to PM10Exposure to ultrafine carbon particles at levels below detectable pulmonary inflammation affects cardiovascular performance in spontaneously hypertensive rats.Ultrafine particulate matter exposure impairs vasorelaxant response in superoxide dismutase 2-deficient murine aortic rings.
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Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
description
article
@en
im August 2006 veröffentlichter wissenschaftlicher Artikel
@de
wetenschappelijk artikel
@nl
наукова стаття, опублікована в серпні 2006
@uk
name
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@en
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@nl
type
label
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@en
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@nl
prefLabel
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@en
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@nl
P2093
P2860
P1476
Ultrafine particulate matter exposure augments ischemia-reperfusion injury in mice
@en
P2093
Emily Cozzi
Howard W Stallings
Robert B Devlin
Robert M Lust
Surovi Hazarika
Wayne E Cascio
P2860
P304
P356
10.1152/AJPHEART.01362.2005
P577
2006-03-31T00:00:00Z