about
Threats to the mind: aging, amyloid, and hypertensionWater deprivation induces neurovascular and cognitive dysfunction through vasopressin-induced oxidative stressNADPH-oxidase-derived reactive oxygen species mediate the cerebrovascular dysfunction induced by the amyloid beta peptide.Brain and circulating levels of Aβ1-40 differentially contribute to vasomotor dysfunction in the mouse brain.Age-dependent neurovascular dysfunction and damage in a mouse model of cerebral amyloid angiopathy.Obligatory Role of EP1 Receptors in the Increase in Cerebral Blood Flow Produced by Hypercapnia in the Mice.Endothelial Dysfunction and Amyloid-β-Induced Neurovascular Alterations.Prohibitin viral gene transfer protects hippocampal CA1 neurons from ischemia and ameliorates postischemic hippocampal dysfunction.Brain Perivascular Macrophages Initiate the Neurovascular Dysfunction of Alzheimer Aβ Peptides.Brain perivascular macrophages: characterization and functional roles in health and disease.Commentary on Myers et al.: growing role of the innate immunity receptor CD36 in central nervous system diseases.Neurovascular protection by ischemic tolerance: role of nitric oxide and reactive oxygen species.Apoε4 disrupts neurovascular regulation and undermines white matter integrity and cognitive functionNeutrophil adhesion in brain capillaries reduces cortical blood flow and impairs memory function in Alzheimer's disease mouse models
P50
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P50
description
researcher ORCID ID = 0000-0001-7601-663X
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Laibaik Park
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Laibaik Park
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Laibaik Park
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Laibaik Park
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Laibaik Park
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Laibaik Park
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Laibaik Park
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Laibaik Park
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P1153
34572129200
P31
P496
0000-0001-7601-663X