about
The thioredoxin reductase-1 inhibitor aurothioglucose attenuates lung injury and improves survival in a murine model of acute respiratory distress syndromeThe thioredoxin system in neonatal lung diseaseMaternal docosahexaenoic acid supplementation decreases lung inflammation in hyperoxia-exposed newborn mice.DHA suppresses chronic apoptosis in the lung caused by perinatal inflammationPrenatal inflammation exacerbates hyperoxia-induced functional and structural changes in adult mice.Attenuation of miR-17∼92 Cluster in Bronchopulmonary Dysplasia.Lipopolysaccharide-induced cyclooxygenase-2 expression in mouse transformed Clara cellsNeonatal hyperoxic exposure persistently alters lung secretoglobins and annexin A1Maternal dietary docosahexaenoic acid supplementation attenuates fetal growth restriction and enhances pulmonary function in a newborn mouse model of perinatal inflammation.Thioredoxin Reductase Inhibition Attenuates Neonatal Hyperoxic Lung Injury and Enhances Nuclear Factor E2-Related Factor 2 Activation.miR-29b supplementation decreases expression of matrix proteins and improves alveolarization in mice exposed to maternal inflammation and neonatal hyperoxia.Of mice and men: correlations between microRNA-17∼92 cluster expression and promoter methylation in severe bronchopulmonary dysplasia.Maternal high fat diet exposure is associated with increased hepcidin levels, decreased myelination, and neurobehavioral changes in male offspring.Riboflavin supplementation does not attenuate hyperoxic lung injury in transgenic (spc-mt)hGR mice.Alterations of the thioredoxin system by hyperoxia: implications for alveolar development.Methods for the determination of plasma or tissue glutathione levels.Arginase II is a target of miR-17-5p and regulates miR-17-5p expression in human pulmonary artery smooth muscle cells.Hyperoxia exposure alters hepatic eicosanoid metabolism in newborn mice.Selenium: implications for outcomes in extremely preterm infants.Aurothioglucose Does Not Improve Alveolarization or Elicit Sustained Nrf2 Activation in C57Bl/6 Models of Bronchopulmonary Dysplasia.Nitrite potentiates the vasodilatory signaling of S-nitrosothiols.Hemodynamic Effects of Glutathione-Liganded Binuclear Dinitrosyl Iron Complex: Evidence for Nitroxyl Generation and Modulation by Plasma Albumin.The thioredoxin reductase inhibitor auranofin induces heme oxygenase-1 in lung epithelial cells via Nrf2-dependent mechanismsSelenium supplementation of lung epithelial cells enhances nuclear factor E2-related factor 2 (Nrf2) activation following thioredoxin reductase inhibitionAlterations in VASP phosphorylation and profilin1 and cofilin1 expression in hyperoxic lung injury and BPDThioredoxin Reductase-1 Inhibition Augments Endogenous Glutathione-Dependent Antioxidant Responses in Experimental Bronchopulmonary DysplasiaL-NAME releases nitric oxide and potentiates subsequent nitroglycerin-mediated vasodilation.Neonatal comorbidities and gasotransmittersDetection of dinitrosyl iron complexes by ozone-based chemiluminescenceThiol-Redox Regulation in Lung Development and Vascular RemodelingAntioxidants & bronchopulmonary dysplasia: Beating the system or beating a dead horse?The discovery BPD (D-BPD) program: study protocol of a prospective translational multicenter collaborative study to investigate determinants of chronic lung disease in very low birth weight infantsOxygen Toxicity in the Neonate: Thinking Beyond the BalanceCyclic O3 exposure synergizes with aging leading to memory impairment in male APOE ε3, but not APOE ε4, targeted replacement mice
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description
researcher ORCID ID = 0000-0002-5156-4282
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wetenschapper
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name
Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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Trent E Tipple
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P31
P496
0000-0002-5156-4282