about
3',4'-Dihydroxyflavonol reduces superoxide and improves nitric oxide function in diabetic rat mesenteric arteries.An evaluation of pharmacology curricula in Australian science and health-related degree programs.Hydrogen Sulfide in the RVLM and PVN has No Effect on Cardiovascular RegulationChronic NaHS Treatment Is Vasoprotective in High-Fat-Fed ApoE(-/-) Mice.Chronic NaHS treatment decreases oxidative stress and improves endothelial function in diabetic mice.Decreased vascular H2S production is associated with vascular oxidative stress in rats fed a high-fat western diet.Mechanism of vasorelaxation and role of endogenous hydrogen sulfide production in mouse aorta.Prevention of ischaemia-induced coronary vascular dysfunction.Hydrogen sulfide protects endothelial nitric oxide function under conditions of acute oxidative stress in vitro.H2S causes contraction and relaxation of major arteries of the rabbit.3',4'-Dihydroxyflavonol restores endothelium-dependent relaxation in small mesenteric artery from rats with type 1 and type 2 diabetesAn investigation of the mechanisms of hydrogen sulfide-induced vasorelaxation in rat middle cerebral arteriesEndothelium-dependent nitroxyl-mediated relaxation is resistant to superoxide anion scavenging and preserved in diabetic rat aortaHydrogen sulfide treatment reduces blood pressure and oxidative stress in angiotensin II-induced hypertensive miceVasorelaxation elicited by endogenous and exogenous hydrogen sulfide in mouse mesenteric arteriesInfluence of type-4 dipeptidyl peptidase inhibition on endothelium-dependent relaxation of aortae from a db/db mouse model of type 2 diabetes: a comparison with the effect of glimepiride
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description
researcher
@en
wetenschapper
@nl
name
J L Hart
@en
J L Hart
@nl
type
label
J L Hart
@en
J L Hart
@nl
prefLabel
J L Hart
@en
J L Hart
@nl
P108
P106
P31
P496
0000-0002-9473-0383