about
Global regulation of gene expression and cell differentiation in Caulobacter crescentus in response to nutrient availabilityStructural Basis of a Rationally Rewired Protein-Protein Interface Critical to Bacterial SignalingTemporal and evolutionary dynamics of two-component signaling pathwaysA DNA damage checkpoint in Caulobacter crescentus inhibits cell division through a direct interaction with FtsWBacillus subtilis SMC complexes juxtapose chromosome arms as they travel from origin to terminus.Evolution of two-component signal transduction systemsHigh-resolution mapping of the spatial organization of a bacterial chromosomeA bacterial toxin inhibits DNA replication elongation through a direct interaction with the β sliding clamp.Permanent genetic memory with >1-byte capacityModularity of the bacterial cell cycle enables independent spatial and temporal control of DNA replicationA DNA damage-induced, SOS-independent checkpoint regulates cell division in Caulobacter crescentus.Determinants of homodimerization specificity in histidine kinases.Protein evolution. Pervasive degeneracy and epistasis in a protein-protein interface.Nutritional Control of DNA Replication Initiation through the Proteolysis and Regulated Translation of DnaA.Evolving new protein-protein interaction specificity through promiscuous intermediates.Rapid pairing and resegregation of distant homologous loci enables double-strand break repair in bacteria.Condensin promotes the juxtaposition of DNA flanking its loading site in Bacillus subtilis.Keeping Signals Straight: How Cells Process Information and Make Decisions.The bacterial cell cycle regulator GcrA is a σ70 cofactor that drives gene expression from a subset of methylated promoters.Global analysis of double-strand break processing reveals in vivo properties of the helicase-nuclease complex AddAB.Identification of the PhoB Regulon and Role of PhoU in the Phosphate Starvation Response of Caulobacter crescentusHelix bundle loops determine whether histidine kinases autophosphorylate in cis or in trans.Proteotoxic stress induces a cell-cycle arrest by stimulating Lon to degrade the replication initiator DnaAContact-dependent killing by Caulobacter crescentus via cell surface-associated, glycine zipper proteins.Polarity and cell fate asymmetry in Caulobacter crescentus.Determinants of specificity in two-component signal transduction.New approaches to understanding the spatial organization of bacterial genomes.Unsupervised Extraction of Stable Expression Signatures from Public Compendia with an Ensemble of Neural Networks.SMC Progressively Aligns Chromosomal Arms in Caulobacter crescentus but Is Antagonized by Convergent Transcription.Transcription rate and transcript length drive formation of chromosomal interaction domain boundaries.Adaptive mutations that prevent crosstalk enable the expansion of paralogous signaling protein familiesA dynamic complex of signaling proteins uses polar localization to regulate cell-fate asymmetry in Caulobacter crescentusAsymmetric cell division: a persistent issue?Dynamics of two Phosphorelays controlling cell cycle progression in Caulobacter crescentus.Finding intron/exon splice junctions using INFO, INterruption Finder and Organizer.ClpAP is an auxiliary protease for DnaA degradation in Caulobacter crescentus.A DNA damage checkpoint in Caulobacter crescentus inhibits cell division through a direct interaction with FtsW.Structural insights into the unique mechanism of transcription activation by Caulobacter crescentus GcrA.The small membrane protein MgrB regulates PhoQ bifunctionality to control PhoP target gene expression dynamics.A Bacterial Chromosome Structuring Protein Binds Overtwisted DNA to Stimulate Type II Topoisomerases and Enable DNA Replication
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description
researcher
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wetenschapper
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name
Laub MT
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Laub MT
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type
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Laub MT
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Laub MT
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Laub MT
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Laub MT
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P108
P106
P31
P496
0000-0002-8288-7607