Programmed cellular necrosis mediated by the pore-forming alpha-toxin from Clostridium septicum - Wikidata - awgldk - TriplyDB

Programmed cellular necrosis mediated by the pore-forming alpha-toxin from Clostridium septicum

Programmed cellular necrosis mediated by the pore-forming alpha-toxin from Clostridium septicum

http://www.wikidata.org/entity/Q21089618

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Signaling beyond Punching Holes: Modulation of Cellular Responses by Vibrio cholerae Cytolysin Clostridium perfringens beta-toxin induces necrostatin-inhibitable, calpain-dependent necrosis in primary porcine endothelial cells Spiro[pyrrolidine-3, 3´-oxindole] as potent anti-breast cancer compounds: Their design, synthesis, biological evaluation and cellular target identification A Novel Role of Listeria monocytogenes Membrane Vesicles in Inhibition of Autophagy and Cell Death.Type IIB procollagen NH(2)-propeptide induces death of tumor cells via interaction with integrins alpha(V)beta(3) and alpha(V)beta(5).Streptococcus pneumoniae translocates into the myocardium and forms unique microlesions that disrupt cardiac function.Genetic control of necrosis - another type of programmed cell death.Clostridium Perfringens Toxins Involved in Mammalian Veterinary Diseases.Necrosis is the dominant cell death pathway in uropathogenic Escherichia coli elicited epididymo-orchitis and is responsible for damage of rat testis.HMGB1 in health and disease.Role of the mitochondrion in programmed necrosis The pore-forming α-toxin from clostridium septicum activates the MAPK pathway in a Ras-c-Raf-dependent and independent manner Gallic acid induces necroptosis via TNF-α signaling pathway in activated hepatic stellate cells.Helicobacter pylori VacA induces programmed necrosis in gastric epithelial cells.Pore-Forming Toxins Induce Macrophage Necroptosis during Acute Bacterial Pneumonia.The tuberculosis necrotizing toxin kills macrophages by hydrolyzing NAD Streptolysin S Promotes Programmed Cell Death and Enhances Inflammatory Signaling in Epithelial Keratinocytes during Group A Streptococcus Infection.Vibrio cholerae Porin OmpU Induces Caspase-independent Programmed Cell Death upon Translocation to the Host Cell Mitochondria PFR peptide, one of the antimicrobial peptides identified from the derivatives of lactoferrin, induces necrosis in leukemia cells Coenzyme depletion by members of the aerolysin family of pore-forming toxins leads to diminished ATP levels and cell death.Calpain-dependent cytoskeletal rearrangement exploited for anthrax toxin endocytosis.Clostridial toxins.Alteration of epithelial cell lysosomal integrity induced by bacterial cholesterol-dependent cytolysins Going up in flames: necrotic cell injury and inflammatory diseases.Multifaceted interactions of bacterial toxins with the gastrointestinal mucosa.The expression of HMGB1 on microparticles released during cell activation and cell death in vitro and in vivo.Chemical regulation of signaling pathways to programmed necrosis.Emerging Roles for RIPK1 and RIPK3 in Pathogen-Induced Cell Death and Host Immunity.Pseudomonas aeruginosa ExlA and Serratia marcescens ShlA trigger cadherin cleavage by promoting calcium influx and ADAM10 activation.How the study of Listeria monocytogenes has led to new concepts in biology.The interplay between regulated necrosis and bacterial infection.Plasma membrane wounding and repair in pulmonary diseases.Staphylococcus aureus α toxin potentiates opportunistic bacterial lung infections.Oxygen-glucose deprivation (OGD) and interleukin-1 (IL-1) differentially modulate cathepsin B/L mediated generation of neuroprotective perlecan LG3 by neurons.Sonodynamic effects of hematoporphyrin monomethyl ether on Staphylococcus aureus in vitro.Differential expression of transcription factors and inflammation-, ROS-, and cell death-related genes in organotypic cultures in the modiolus, the organ of Corti and the stria vascularis of newborn rats.A bacterial RTX toxin causes programmed necrotic cell death through calcium-mediated mitochondrial dysfunction.Pro-necrotic Activity of Cationic Mastoparan Peptides in Human Glioblastoma Multiforme Cells Via Membranolytic Action.Pore-forming Toxins Functional Consequences of Calcium Influx Promoted by Bacterial Pore-Forming Toxins

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P2860

Programmed cellular necrosis mediated by the pore-forming alpha-toxin from Clostridium septicum

http://www.wikidata.org/entity/Q21089618

description

2009 nî lūn-bûn

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2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի հուլիսին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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JOURNAL.PPAT.1000516

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Programmed cellular necrosis m ...... oxin from Clostridium septicum

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Anjana Chakravorty

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Catherine L Kennedy

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Danielle J Smith

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Dena Lyras

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P2860

Calcium-dependent signaling pathways in T cells. Potential role of calpain, protein tyrosine phosphatase 1b, and p130Cas in integrin-mediated signaling events

Apoptosis, pyroptosis, and necrosis: mechanistic description of dead and dying eukaryotic cells.

alpha-Toxin is a mediator of Staphylococcus aureus-induced cell death and activates caspases via the intrinsic death pathway independently of death receptor signaling

Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

HMG-1 as a late mediator of endotoxin lethality in mice

Pore-forming activity of alpha-toxin is essential for clostridium septicum-mediated myonecrosis.

Cysteine cathepsins are central contributors of invasion by cultured adenosylmethionine decarboxylase-transformed rodent fibroblasts.

Heat shock pretreatment inhibited the release of Smac/DIABLO from mitochondria and apoptosis induced by hydrogen peroxide in cardiomyocytes and C2C12 myogenic cells.

Potassium is a critical regulator of apoptotic enzymes in vitro and in vivo.

Death by necrosis. Uncontrollable catastrophe, or is there order behind the chaos?

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scholarly article

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10.1371/JOURNAL.PPAT.1000516

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19609357

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2705182

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