Streptococcus pneumoniae translocates into the myocardium and forms unique microlesions that disrupt cardiac function. - Wikidata - awgldk - TriplyDB

Streptococcus pneumoniae translocates into the myocardium and forms unique microlesions that disrupt cardiac function.

Streptococcus pneumoniae translocates into the myocardium and forms unique microlesions that disrupt cardiac function.

http://www.wikidata.org/entity/Q34215330

about

Individuality, phenotypic differentiation, dormancy and 'persistence' in culturable bacterial systems: commonalities shared by environmental, laboratory, and clinical microbiology Circulating Pneumolysin Is a Potent Inducer of Cardiac Injury during Pneumococcal Infection Epidemiology, virulence factors and management of the pneumococcus Cardiotoxicity during invasive pneumococcal disease Streptococcus pneumoniae Cell-Wall-Localized Phosphoenolpyruvate Protein Phosphotransferase Can Function as an Adhesin: Identification of Its Host Target Molecules and Evaluation of Its Potential as a Vaccine Acute myocardial infarction versus other cardiovascular events in community-acquired pneumonia Promiscuous signaling by a regulatory system unique to the pandemic PMEN1 pneumococcal lineage.Increased Zinc Availability Enhances Initial Aggregation and Biofilm Formation of Streptococcus pneumoniae Prevalence, pathogenesis, therapy, and prevention of cardiovascular events in patients with community-acquired pneumonia.Intermediate and long-term risk of new-onset heart failure after hospitalization for pneumonia in elderly adults.Pore-Forming Toxins Induce Macrophage Necroptosis during Acute Bacterial Pneumonia.Visualization of Streptococcus pneumoniae within Cardiac Microlesions and Subsequent Cardiac Remodeling.Bacterial exploitation of phosphorylcholine mimicry suppresses inflammation to promote airway infection.A Non-Human Primate Model of Severe Pneumococcal Pneumonia.Invasive Pneumococcal Disease: Still Lots to Learn and a Need for Standardized Data Collection Instruments.Statin-conferred enhanced cellular resistance against bacterial pore-forming toxins in airway epithelial cells Infiltrated Macrophages Die of Pneumolysin-Mediated Necroptosis following Pneumococcal Myocardial Invasion Anatomical site-specific contributions of pneumococcal virulence determinants.Cardiac Electrical and Structural Changes During Bacterial Infection: An Instructive Model to Study Cardiac Dysfunction in Sepsis.Disentangling competence for genetic transformation and virulence in Streptococcus pneumoniae.Streptococcus pneumoniae in the heart subvert the host response through biofilm-mediated resident macrophage killing.South African guideline for the management of community-acquired pneumonia in adults.Severe Pneumococcal Pneumonia Causes Acute Cardiac Toxicity and Subsequent Cardiac Remodeling.Pneumolysin Mediates Platelet Activation In Vitro.Cigarette Smoke Attenuates the Nasal Host Response to Streptococcus pneumoniae and Predisposes to Invasive Pneumococcal Disease in Mice.Pneumolysin activates neutrophil extracellular trap formation.Risk of exacerbation following pneumonia in adults with heart failure or chronic obstructive pulmonary disease Efferocytosis and Outside-In Signaling by Cardiac Phagocytes. Links to Repair, Cellular Programming, and Intercellular Crosstalk in Heart.Pore-forming toxin-mediated ion dysregulation leads to death receptor-independent necroptosis of lung epithelial cells during bacterial pneumonia.Cell invasion and pyruvate oxidase derived H2O2 are critical for Streptococcus pneumoniae mediated cardiomyocyte killing.Pneumonia as a cardiovascular disease.New Insights into the Immunobiology of Mononuclear Phagocytic Cells and Their Relevance to the Pathogenesis of Cardiovascular Diseases.A Peculiar Case of Invasive Streptococcus pneumoniae.Streptococcus pneumoniae: transmission, colonization and invasion.Multifaceted Role of Pneumolysin in the Pathogenesis of Myocardial Injury in Community-Acquired Pneumonia.Triptych of the Hermit Saints: pneumococcal polysaccharide vaccines for the elderly.IL-17 can be protective or deleterious in murine pneumococcal pneumonia.Severity and properties of cardiac damage caused by Streptococcus pneumoniae are strain dependent

P2860

Q26775404-C5691A26-3591-44C2-BBD6-3147DC8273AB Q27320196-8574BE6B-540D-40D7-9ECF-11239CC3AB0C Q28078832-E247FE53-21C0-48CB-848D-5003EBF12F53 Q28081769-E9F10CB8-A999-40A0-8173-FE68941854D7 Q28550871-039E2114-D302-42CB-9A13-074D754791F0 Q28596751-6F41100F-A275-410F-9A09-33A25D380FE9 Q30402403-12ED147F-3F9B-4B86-B396-544ED45B98B7 Q33770464-FCCECC07-E06E-4583-85AF-9D1535125DEC Q33801302-8A5E2932-DE6C-49BB-8FDB-6DCB85500854 Q34490627-7F194C47-4225-4776-B2AD-0F75947B22FE Q35867134-B1B88FFE-74E6-44F1-8DF5-664FAE711F36 Q35970427-754C2716-2842-4E0F-8769-165E2F70994E Q36165636-AAB080A0-70B3-4D74-8B7B-4BD15303E4A9 Q36195060-BD758301-AF46-488B-9B85-597C1C2E44FA Q36351403-A415A48D-1145-4AE5-AB8C-18E9F81A0B85 Q36549172-97C10930-9373-4D35-AEE4-3DFA41D75B7F Q36888581-27589338-AC2B-4D02-BECC-D7F4C7D8E2C1 Q37252799-0F95433D-A18F-44F5-B207-B60CBA576043 Q37364469-9926EE44-FC25-421A-A914-71976B35666B Q38592328-471AA874-3019-46EF-8E48-E67CD15B31CB Q38637237-EE45345D-6AC9-42FE-A3AD-C4AA11EDFBE0 Q39455917-CAF09A1E-E3C3-4768-9F89-4D9D5C4E1594 Q40172815-F3A4B0B1-7E2F-4FE0-974F-773B27E6442C Q40669297-A3B5C398-6004-4521-8E36-15CD3132627D Q40771962-406DDFE7-13E0-4B45-A731-13659B2607E1 Q40839011-C13DB24B-20F1-4968-ADCB-899393599F2B Q42701714-2A886EC2-F057-4F69-A9D4-7030FD773353 Q45341410-7096AA68-9CED-46D3-9B2E-ECB76529A0BA Q46388233-0EFACF1B-1F75-4D06-9774-B5FFE7A3FD17 Q47614256-363C1A49-118B-4151-A0A3-0B6D7DFC1DF1 Q47658087-F0D079F1-B293-4245-A435-BDDC86599F4C Q49531175-55C5873E-16ED-4BF5-AF6C-0BB8AB11B4FA Q49787136-50176894-A3AA-458F-A642-87DB82F40E93 Q52337590-FC4907E7-43E2-4D9D-92DC-043A7521B49E Q53693867-E6C7EBE4-CD8E-4F0A-B9EA-E04CE5E27F09 Q55267236-595B83D3-8CBA-43FE-8DFB-D0CD54B2DDEB Q55362292-65BA0628-1382-4021-9E9B-246A787B01AA Q58741227-5CA06F54-1E4A-48C8-998E-1BD0DCA916AD

P2860

Streptococcus pneumoniae translocates into the myocardium and forms unique microlesions that disrupt cardiac function.

http://www.wikidata.org/entity/Q34215330

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2014年の論文

@ja

2014年論文

@yue

2014年論文

@zh-hant

2014年論文

@zh-hk

2014年論文

@zh-mo

2014年論文

@zh-tw

2014年论文

@wuu

name

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@ast

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@en

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@nl

type

label

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@ast

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@en

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@nl

prefLabel

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@ast

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@en

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@nl

P1433

Q34215330-2760850C-E6D5-4CA4-B682-0C273D81A2E8

P1476

Q34215330-B1B313F7-9657-4C45-951A-C45489B7962C

P2093

Q34215330-08C2D96E-41FB-4204-9C02-25F7CA2193BE

Q34215330-22629514-ADFF-4EA0-9668-092D9BC0CA6E

Q34215330-29A5B46D-1107-4DB5-8B20-95CEC0D37C15

Q34215330-520387BA-9B7D-4032-ADEF-C2005E59F193

Q34215330-593B48CB-AEBB-4C29-BA10-019EE2E64F6F

Q34215330-6BB7C58D-5181-4D16-831B-AC20E43079B9

Q34215330-6BEDA286-D347-4306-88A1-D7C84DB6DC1F

Q34215330-77BE5FE9-1B0A-42FB-9D9A-60E131F844B3

Q34215330-874AF6FD-6A3D-4E79-8B1F-949E12C93B2D

Q34215330-9D282E9A-23AC-425B-B6B2-38935EF4B458

P2860

Q34215330-07D8D103-2572-4D14-9194-7D22AD28DF2B

Q34215330-0DCE67FA-6520-400B-B63E-83CEF99B6A2F

Q34215330-11510299-39A2-4666-ABD0-581C26654EB9

Q34215330-15BDB289-4AA2-449D-AF64-31C074705DF7

Q34215330-15F76ACC-28D9-4B25-94DB-68CC68287FFF

Q34215330-184528DF-F82A-495F-AF28-F49EC94565C3

Q34215330-199686F2-79EF-49E5-B262-013B44C3A503

Q34215330-21BCA3F6-DEC4-427E-97CD-E8E1E9E0F009

Q34215330-26868D2D-5C59-4DF7-893D-3A4F24AE9CDE

Q34215330-2C079033-B3A4-470C-B473-0AE7ECD8AF69

P304

Q34215330-118FCC8A-AE42-4F0A-BE98-8CA3854FF169

P31

Q34215330-821F901A-410B-4AA4-A79C-E13E3F3BE052

P356

Q34215330-AFA70921-1B78-444F-961B-3E601121A7F0

P433

Q34215330-885E78C0-9358-45CE-9824-E2EA72B26518

P478

Q34215330-D2A025AF-81E5-4437-8A61-32913648C1E1

P50

Q34215330-46D63478-69A3-40EA-87E0-09525DFF7D54

Q34215330-52D0514E-8307-4C80-80D1-31D136EA67AE

Q34215330-6FE8E570-03F4-4AA8-AF58-D04E80E34E3F

Q34215330-AAE684EF-9A3E-4597-91DC-037D1005438E

P577

Q34215330-363D9A1C-9CBF-44D0-AEDC-EBD115015723

P5875

Q34215330-5E0AC10D-8482-4D61-91E5-22D9BF457499

P698

Q34215330-70DDF291-A521-44BD-9F99-B24DED9013E8

P932

Q34215330-1B41479D-B6EA-49CB-AD77-D3CA814F36FF

P356

JOURNAL.PPAT.1004383

P1433

P1476

Streptococcus pneumoniae trans ...... that disrupt cardiac function.

@en

P2093

Andres Esteban

http://www.w3.org/2001/XMLSchema#string

Armand O Brown

http://www.w3.org/2001/XMLSchema#string

Beth Mann

http://www.w3.org/2001/XMLSchema#string

Carlos J Orihuela

http://www.w3.org/2001/XMLSchema#string

Claude J LeSaux

http://www.w3.org/2001/XMLSchema#string

Ganesh V Halade

http://www.w3.org/2001/XMLSchema#string

Geli Gao

http://www.w3.org/2001/XMLSchema#string

Gregory J Bagby

http://www.w3.org/2001/XMLSchema#string

Jane S Hankins

http://www.w3.org/2001/XMLSchema#string

Jessica Humann

http://www.w3.org/2001/XMLSchema#string

P2860

Programmed cellular necrosis mediated by the pore-forming alpha-toxin from Clostridium septicum

Cutting edge: recognition of Gram-positive bacterial cell wall components by the innate immune system occurs via Toll-like receptor 2

Complete Genome Sequence of a Virulent Isolate of Streptococcus pneumoniae

The NLRP3 inflammasome contributes to brain injury in pneumococcal meningitis and is activated through ATP-dependent lysosomal cathepsin B release

Streptococcus pneumoniae anchor to activated human cells by the receptor for platelet-activating factor

Clostridium perfringens beta-toxin induces necrostatin-inhibitable, calpain-dependent necrosis in primary porcine endothelial cells

Bacterial flagellin triggers cardiac innate immune responses and acute contractile dysfunction

Cardiac complications in patients with community-acquired pneumonia: a systematic review and meta-analysis of observational studies

beta-Arrestin 1 participates in platelet-activating factor receptor-mediated endocytosis of Streptococcus pneumoniae.

Laminin receptor initiates bacterial contact with the blood brain barrier in experimental meningitis models.

P304

e1004383

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1371/JOURNAL.PPAT.1004383

http://www.w3.org/2001/XMLSchema#string

P433

9

http://www.w3.org/2001/XMLSchema#string

P478

10

http://www.w3.org/2001/XMLSchema#string

P50

Elaine I Tuomanen

Merry L. Lindsey

P577

2014-09-18T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

265791062

http://www.w3.org/2001/XMLSchema#string

P698

25232870

http://www.w3.org/2001/XMLSchema#string

P932

4169480

http://www.w3.org/2001/XMLSchema#string