Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair - Wikidata - awgldk - TriplyDB

Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair

Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair

http://www.wikidata.org/entity/Q21145008

about

Increased learning and brain long-term potentiation in aged mice lacking DNA polymerase μ Sustained active site rigidity during synthesis by human DNA polymerase μ Nonhomologous end joining: a good solution for bad ends A human iPSC model of Ligase IV deficiency reveals an important role for NHEJ-mediated-DSB repair in the survival and genomic stability of induced pluripotent stem cells and emerging haematopoietic progenitors.Choosing the right path: does DNA-PK help make the decision?Polμ deficiency increases resistance to oxidative damage and delays liver aging.Essential role for polymerase specialization in cellular nonhomologous end joining.Cell autonomous and nonautonomous mechanisms drive hematopoietic stem/progenitor cell loss in the absence of DNA repair.Increased neuronal death and disturbed axonal growth in the Polμ-deficient mouse embryonic retina.Myelodysplastic syndrome: an inability to appropriately respond to damaged DNA?DNA polymerases and cancer.Gadd45b deficiency promotes premature senescence and skin aging.Polymerases in nonhomologous end joining: building a bridge over broken chromosomes.Biological and therapeutic relevance of nonreplicative DNA polymerases to cancer.DNA polymerases in nonhomologous end joining: are there any benefits to standing out from the crowd?DNA expansions generated by human Polμ on iterative sequences.Mutational signatures of non-homologous and polymerase theta-mediated end-joining in embryonic stem cells.Vitamin transporters in mice brain with aging.

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Altered hematopoiesis in mice lacking DNA polymerase mu is due to inefficient double-strand break repair

http://www.wikidata.org/entity/Q21145008

description

2009 nî lūn-bûn

@nan

2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Altered hematopoiesis in mice ...... ent double-strand break repair

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JOURNAL.PGEN.1000389

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Altered hematopoiesis in mice ...... ent double-strand break repair

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Antonio Bernad

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Beatriz Escudero

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Daniel Lucas

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Enrique Samper

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Gloria Terrados

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José Manuel Ligos

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Juan Camilo Estrada

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Luis Blanco

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P2860

Two novel human and mouse DNA polymerases of the polX family

Association of DNA polymerase mu (pol mu) with Ku and ligase IV: role for pol mu in end-joining double-strand break repair.

Flk-2 is a marker in hematopoietic stem cell differentiation: a simple method to isolate long-term stem cells

A gradient of template dependence defines distinct biological roles for family X polymerases in nonhomologous end joining.

DNA double-stranded breaks induce histone H2AX phosphorylation on serine 139

Interplay of p53 and DNA-repair protein XRCC4 in tumorigenesis, genomic stability and development

Deletion of Ku86 causes early onset of senescence in mice

Immunoglobulin kappa light chain gene rearrangement is impaired in mice deficient for DNA polymerase mu

Chromosomal stability and the DNA double-stranded break connection

Deficiencies in DNA damage repair limit the function of haematopoietic stem cells with age

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e1000389

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scholarly article

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10.1371/JOURNAL.PGEN.1000389

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Jose Carlos Segovia

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2009-02-01T00:00:00Z

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24027201

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19229323

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