Morphine reduces local cytokine expression and neutrophil infiltration after incision
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AMPK: An emerging target for modification of injury-induced pain plasticity.Adenosine Monophosphate-activated Protein Kinase (AMPK) Activators For the Prevention, Treatment and Potential Reversal of Pathological Pain.Chronic morphine administration delays wound healing by inhibiting immune cell recruitment to the wound site.Peripheral administration of morphine attenuates postincisional pain by regulating macrophage polarization through COX-2-dependent pathway.Caspase-1 modulates incisional sensitization and inflammationEvaluation of the effects of methadone and tramadol on postoperative analgesia and serum interleukin-6 in dogs undergoing orthopaedic surgery.Increased local concentration of complement C5a contributes to incisional pain in mice.Linking traits based on their shared molecular mechanisms.The complement component C5a receptor mediates pain and inflammation in a postsurgical pain model.Sensitization of dural afferents underlies migraine-related behavior following meningeal application of interleukin-6 (IL-6).Resveratrol engages AMPK to attenuate ERK and mTOR signaling in sensory neurons and inhibits incision-induced acute and chronic pain.Direct gene transfer with compacted DNA nanoparticles in retinal pigment epithelial cells: expression, repeat delivery and lack of toxicity.Roles of Gr-1+ leukocytes in postincisional nociceptive sensitization and inflammationmiR-203 regulates nociceptive sensitization after incision by controlling phospholipase A2 activating protein expression.Collecting and measuring wound exudate biochemical mediators in surgical wounds.Dopamine D1 receptors within the basolateral amygdala mediate heroin-induced conditioned immunomodulation.Ventral tegmental area-basolateral amygdala-nucleus accumbens shell neurocircuitry controls the expression of heroin-conditioned immunomodulation.Opioid drug abuse and modulation of immune function: consequences in the susceptibility to opportunistic infections.Rapid, opioid-sensitive mechanisms involved in transient receptor potential vanilloid 1 sensitization.Association of catastrophizing with interleukin-6 responses to acute pain.Keratinocytes express cytokines and nerve growth factor in response to neuropeptide activation of the ERK1/2 and JNK MAPK transcription pathways.Peripheral mechanisms of pain and analgesia.Role of substance P signaling in enhanced nociceptive sensitization and local cytokine production after incision.Injury-induced platelet-derived growth factor receptor-alpha expression mediated by interleukin-1beta (IL-1beta) release and cooperative transactivation by NF-kappaB and ATF-4: IL-1beta facilitates HDAC-1/2 dissociation from promoter.Morphine, but not trauma, sensitizes to systemic Acinetobacter baumannii infection.Should topical opioid analgesics be regarded as effective and safe when applied to chronic cutaneous lesions?Can morphine interfere in the healing process during chronic stress?Targeting AMPK for the Alleviation of Pathological Pain.Transdermal opioid patches for pain treatment in ancient Greece.Tonsillectomy healing.Clinical and immunohistochemical characteristics of mucoceles.Preprotachykinin-A gene disruption attenuates nociceptive sensitivity after opioid administration and incision by peripheral and spinal mechanisms in mice.Chronic morphine administration enhances nociceptive sensitivity and local cytokine production after incisionMyeloperoxidase content is a marker of systemic inflammation in a chronic condition: the example given by the periodontal disease in rats.IL-6- and NGF-induced rapid control of protein synthesis and nociceptive plasticity via convergent signaling to the eIF4F complex.Effect of tramadol on immune responses and nociceptive thresholds in a rat model of incisional pain.Effects of fentanyl on pain and motor behaviors following a collagenase-induced intracerebral hemorrhage in rats.Opioids enhance CXCL1 expression and function after incision in mice.Neutrophils recruited by CXCR1/2 signalling mediate post-incisional pain.Tephrosia sinapou ethyl acetate extract inhibits inflammatory pain in mice: opioid receptor dependent inhibition of TNFα and IL-1β production.
P2860
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P2860
Morphine reduces local cytokine expression and neutrophil infiltration after incision
description
2007 nî lūn-bûn
@nan
2007 թուականին հրատարակուած գիտական յօդուած
@hyw
2007 թվականին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@ast
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en-gb
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@nl
type
label
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@ast
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en-gb
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@nl
prefLabel
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@ast
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en-gb
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@nl
P2093
P2860
P356
P1433
P1476
Morphine reduces local cytokine expression and neutrophil infiltration after incision
@en
P2093
David C Yeomans
DeYong Liang
J David Clark
Martin S Angst
Xiangqi Li
Xiaoyou Shi
Yanli Qiao
P2860
P2888
P356
10.1186/1744-8069-3-28
P407
P577
2007-01-01T00:00:00Z
P5875
P6179
1033819632