IL-6- and NGF-induced rapid control of protein synthesis and nociceptive plasticity via convergent signaling to the eIF4F complex.
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Efficient retrograde transport of pseudorabies virus within neurons requires local protein synthesis in axonsInterleukin-6: an emerging regulator of pathological painAMPK: An emerging target for modification of injury-induced pain plasticity.Nociceptive TRP Channels: Sensory Detectors and Transducers in Multiple Pain PathologiesCalcium-Sensing Receptor Gene: Regulation of ExpressionCommonalities between pain and memory mechanisms and their meaning for understanding chronic painTranslational control of nociception via 4E-binding protein 1Adenosine Monophosphate-activated Protein Kinase (AMPK) Activators For the Prevention, Treatment and Potential Reversal of Pathological Pain.Quantitative, time-resolved proteomic analysis by combining bioorthogonal noncanonical amino acid tagging and pulsed stable isotope labeling by amino acids in cell cultureLocal synthesis of dynein cofactors matches retrograde transport to acutely changing demandsLocal translation and retrograde axonal transport of CREB regulates IL-6-induced nociceptive plasticity.Deletion of interleukin-6 signal transducer gp130 in small sensory neurons attenuates mechanonociception and down-regulates TRPA1 expression.Peripheral nerve regeneration and NGF-dependent neurite outgrowth of adult sensory neurons converge on STAT3 phosphorylation downstream of neuropoietic cytokine receptor gp130.mTORC1 inhibition induces pain via IRS-1-dependent feedback activation of ERK.Balancing GRK2 and EPAC1 levels prevents and relieves chronic pain.Evolution: the advantage of 'maladaptive' pain plasticity.Spinal dopaminergic projections control the transition to pathological pain plasticity via a D1/D5-mediated mechanism.Fragile X mental retardation protein (FMRP) and the spinal sensory system.gp130 cytokines are positive signals triggering changes in gene expression and axon outgrowth in peripheral neurons following injury.Self-injurious behaviour in intellectual disability syndromes: evidence for aberrant pain signalling as a contributing factor.Sensitization of dural afferents underlies migraine-related behavior following meningeal application of interleukin-6 (IL-6).Non-canonical amino acid labeling in vivo to visualize and affinity purify newly synthesized proteins in larval zebrafishResveratrol engages AMPK to attenuate ERK and mTOR signaling in sensory neurons and inhibits incision-induced acute and chronic pain.ERK2 Alone Drives Inflammatory Pain But Cooperates with ERK1 in Sensory Neuron SurvivalGeneration of a pain memory in the primary afferent nociceptor triggered by PKCε activation of CPEB.Nerve growth factor enhances the excitability of rat sensory neurons through activation of the atypical protein kinase C isoform, PKMζ.Protease-activated receptor 2 activation is sufficient to induce the transition to a chronic pain stateThe pharmacology of nociceptor primingAxonal transport of neural membrane protein 35 mRNA increases axon growth.Sustained exposure to cytokines and hypoxia enhances excitability of oxygen-sensitive type I cells in rat carotid body: correlation with the expression of HIF-1α protein and adrenomedullinLocal action of the proinflammatory cytokines IL-1β and IL-6 on intracranial meningeal nociceptors.Isolation and analyses of axonal ribonucleoprotein complexesCellular, molecular, and epigenetic mechanisms in non-associative conditioning: implications for pain and memoryEarly exercise rehabilitation of muscle weakness in acute respiratory failure patientsThe use of metformin is associated with decreased lumbar radiculopathy pain.Bidirectional regulation of P body formation mediated by eIF4F complex formation in sensory neurons.Impaired NGF/TrkA Signaling Causes Early AD-Linked Presynaptic Dysfunction in Cholinergic Primary NeuronsIon channels and migraineMitogen-activated protein kinase phosphatase-3 (MKP-3) in the surgical wound is necessary for the resolution of postoperative pain in mice.Paracrine control of vascular innervation in health and disease.
P2860
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P2860
IL-6- and NGF-induced rapid control of protein synthesis and nociceptive plasticity via convergent signaling to the eIF4F complex.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@en
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@nl
type
label
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@en
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@nl
prefLabel
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@en
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@nl
P2093
P2860
P1476
IL-6- and NGF-induced rapid co ...... ignaling to the eIF4F complex.
@en
P2093
Dipti V Tillu
Gregory O Dussor
Katherine A Peebles
Marina N Asiedu
Natalie Ertz
Ohannes K Melemedjian
P2860
P304
15113-15123
P356
10.1523/JNEUROSCI.3947-10.2010
P407
P577
2010-11-01T00:00:00Z