Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
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Construction and analysis of a modular model of caspase activation in apoptosisModeling a snap-action, variable-delay switch controlling extrinsic cell deathBistability in apoptosis by receptor clusteringModeling the role of p53 pulses in DNA damage- induced cell death decision.Building biological memory by linking positive feedback loops.The role of coupled positive feedback in the expression of the SPI1 type three secretion system in Salmonella.Mathematical models of the transitions between endocrine therapy responsive and resistant states in breast cancer.Dynamic modelling of oestrogen signalling and cell fate in breast cancer cellsProgramming biological models in Python using PySBDynamical behaviors of Rb-E2F pathway including negative feedback loops involving miR449.Data-driven modeling of SRC control on the mitochondrial pathway of apoptosis: implication for anticancer therapy optimization.Bayesian parameter inference by Markov chain Monte Carlo with hybrid fitness measures: theory and test in apoptosis signal transduction networkThe mathematical model of the Bcl-2 family mediated MOMP regulation can perform a non-trivial pattern recognition.Steady states and dynamics of urokinase-mediated plasmin activation in silico and in vitroComputational modeling of signaling pathways mediating cell cycle checkpoint control and apoptotic responses to ionizing radiation-induced DNA damage.Time to Decide? Dynamical Analysis Predicts Partial Tip/Stalk Patterning States Arise during Angiogenesis.Plasmin triggers a switch-like decrease in thrombospondin-dependent activation of TGF-β1.Computational analysis of dynamical responses to the intrinsic pathway of programmed cell death.The indirect activation model of mitochondrial outer membrane permeabilisation (MOMP) initiation requires a trade-off between robustness in the absence of and sensitivity in the presence of stress.Rigor of cell fate decision by variable p53 pulses and roles of cooperative gene expression by p53.Boolean network-based model of the Bcl-2 family mediated MOMP regulation.A cellular stress-directed bistable switch controls the crosstalk between autophagy and apoptosis.
P2860
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P2860
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
description
2008 nî lūn-bûn
@nan
2008 թուականին հրատարակուած գիտական յօդուած
@hyw
2008 թվականին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@ast
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en-gb
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@nl
type
label
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@ast
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en-gb
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@nl
altLabel
Two Independent Positive Feedbacks and Bistability in the Bcl-2 Apoptotic Switch
@en
prefLabel
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@ast
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en-gb
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@nl
P2093
P2860
P1433
P1476
Two independent positive feedbacks and bistability in the Bcl-2 apoptotic switch
@en
P2093
Pingping Shen
Tingzhe Sun
P2860
P356
10.1371/JOURNAL.PONE.0001469
P407
P50
P577
2008-01-01T00:00:00Z