C/EBPα Dysregulation in AML and ALL - Wikidata - awgldk - TriplyDB

C/EBPα Dysregulation in AML and ALL

C/EBPα Dysregulation in AML and ALL

http://www.wikidata.org/entity/Q22065117

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Aberrant nuclear factor-kappa B activity in acute myeloid leukemia: from molecular pathogenesis to therapeutic target CEBPA exerts a specific and biologically important proapoptotic role in pancreatic β cells through its downstream network targets Suv39h1 mediates AP-2α-dependent inhibition of C/EBPα expression during adipogenesis.The Cebpa +37-kb enhancer directs transgene expression to myeloid progenitors and to long-term hematopoietic stem cells.CEBPA-dependent HK3 and KLF5 expression in primary AML and during AML differentiation Zinc finger protein 382 is downregulated by promoter hypermethylation in pediatric acute myeloid leukemia patients Dual role of hematopoietic progenitor kinase 1 (HPK1) as a positive regulator of 1α,25-dihydroxyvitamin D-induced differentiation and cell cycle arrest of AML cells and as a mediator of vitamin D resistance CDC25A governs proliferation and differentiation of FLT3-ITD acute myeloid leukemia.RARα-PLZF oncogene inhibits C/EBPα function in myeloid cells.Neutropenia-associated ELANE mutations disrupting translation initiation produce novel neutrophil elastase isoforms.Molecular-based classification of acute myeloid leukemia and its role in directing rational therapy: personalized medicine for profoundly promiscuous proliferations.Concise review: erythroid versus myeloid lineage commitment: regulating the master regulators.The functionally diverse roles of tribbles.C/EBPα in normal and malignant myelopoiesis.The tumor suppressor gene DAPK2 is induced by the myeloid transcription factors PU.1 and C/EBPα during granulocytic differentiation but repressed by PML-RARα in APL.Affinity purification strategies for proteomic analysis of transcription factor complexes.Transcription Factor GFI1B in Health and Disease.The hypoxia-inducible factor-C/EBPα axis controls ethanol-mediated hepcidin repression.The presence of C/EBPα and its degradation are both required for TRIB2-mediated leukaemia.Disentangling the microRNA regulatory milieu in multiple myeloma: integrative genomics analysis outlines mixed miRNA-TF circuits and pathway-derived networks modulated in t(4;14) patients.Concordant acute myeloblastic leukemia in monozygotic twins with germline and shared somatic mutations in the gene for CCAAT-enhancer-binding protein α with 13 years difference at onset.CD2-positive B-cell precursor acute lymphoblastic leukemia with an early switch to the monocytic lineage.TRIB2 contributes to cisplatin resistance in small cell lung cancer.Evaluation of CCAAT/Enhancer Binding Protein (C/EBP) Alpha (CEBPA) and Runt-Related Transcription Factor 1 (RUNX1) Expression in Patients with De Novo Acute Myeloid Leukemia.MicroRNA-155 induces AML in combination with the loss of C/EBPA in mice.

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C/EBPα Dysregulation in AML and ALL

http://www.wikidata.org/entity/Q22065117

description

2011 nî lūn-bûn

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2011 թուականին հրատարակուած գիտական յօդուած

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2011 թվականին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年论文

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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C/EBPα Dysregulation in AML and ALL

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CRITREVONCOG.V16.I1-2.90

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Critical Reviews in Oncogenesis

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C/EBPα Dysregulation in AML and ALL

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Alan Friedman

http://www.w3.org/2001/XMLSchema#string

Ido Paz-Priel

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The 2008 revision of the World Health Organization (WHO) classification of myeloid neoplasms and acute leukemia: rationale and important changes

Dominant-negative mutations of CEBPA, encoding CCAAT/enhancer binding protein-alpha (C/EBPalpha), in acute myeloid leukemia

Prevalence and prognostic implications of CEBPA mutations in pediatric acute myeloid leukemia (AML): a report from the Children's Oncology Group

Cell-cycle regulator E2F1 and microRNA-223 comprise an autoregulatory negative feedback loop in acute myeloid leukemia

Structural basis for DNA recognition by the basic region leucine zipper transcription factor CCAAT/enhancer-binding protein alpha

CEBPA mutations in younger adults with acute myeloid leukemia and normal cytogenetics: prognostic relevance and analysis of cooperating mutations.

Mutations and treatment outcome in cytogenetically normal acute myeloid leukemia.

ATF-2 and C/EBPalpha can form a heterodimeric DNA binding complex in vitro. Functional implications for transcriptional regulation

MicroRNA expression profiling in relation to the genetic heterogeneity of acute myeloid leukemia

E2F repression by C/EBPalpha is required for adipogenesis and granulopoiesis in vivo

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93-102

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scholarly article

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10.1615/CRITREVONCOG.V16.I1-2.90

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1-2

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16

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2011-01-01T00:00:00Z

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51860655

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22150310

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3243939

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