FIF [fibroblast growth factor-2 (FGF-2)-interacting-factor], a nuclear putatively antiapoptotic factor, interacts specifically with FGF-2
about
Functional identification of Api5 as a suppressor of E2F-dependent apoptosis in vivoThe antiapoptotic protein AAC-11 interacts with and regulates Acinus-mediated DNA fragmentationDirect and heterologous approaches to identify the LET-756/FGF interactomeA nuclear target for interleukin-1alpha: interaction with the growth suppressor necdin modulates proliferation and collagen expressionHelical Repeat Structure of Apoptosis Inhibitor 5 Reveals Protein-Protein Interaction ModulesApoptosis inhibitor-5 overexpression is associated with tumor progression and poor prognosis in patients with cervical cancerSignaling by fibroblast growth factors: the inside story.Api5 contributes to E2F1 control of the G1/S cell cycle phase transition.API5 confers tumoral immune escape through FGF2-dependent cell survival pathway.High molecular weight FGF2: the biology of a nuclear growth factor.Apoptosis Inhibitor 5 Increases Metastasis via Erk-mediated MMP expressionFibroblast growth factor signaling in embryonic and cancer stem cells.Long Noncoding RNA LOC100129973 Suppresses Apoptosis by Targeting miR-4707-5p and miR-4767 in Vascular Endothelial Cells.API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.Targeting AAC-11 in cancer therapy.A nuclear odyssey: fibroblast growth factor-2 (FGF-2) as a regulator of nuclear homeostasis in the nervous system.Nucleoprotein of influenza A virus negatively impacts antiapoptotic protein API5 to enhance E2F1-dependent apoptosis and virus replication.Nuclear trafficking of secreted factors and cell-surface receptors: new pathways to regulate cell proliferation and differentiation, and involvement in cancers.Translocation of FGF-1 and FGF-2 across vesicular membranes occurs during G1-phase by a common mechanismApi5 a new cofactor of estrogen receptor alpha involved in breast cancer outcome.Exacerbation of myocardial injury in transgenic mice overexpressing FGF-2 is T cell dependent.Apoptosis inhibitor 5 is an endogenous inhibitor of caspase-2.Rice APOPTOSIS INHIBITOR5 coupled with two DEAD-box adenosine 5'-triphosphate-dependent RNA helicases regulates tapetum degeneration.High molecular weight fibroblast growth factor 2 induces apoptosis by interacting with complement component 1 Q subcomponent-binding protein in vitro
P2860
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P2860
FIF [fibroblast growth factor-2 (FGF-2)-interacting-factor], a nuclear putatively antiapoptotic factor, interacts specifically with FGF-2
description
2000 nî lūn-bûn
@nan
2000 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2000年の論文
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name
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@ast
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en-gb
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@nl
type
label
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@ast
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en-gb
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@nl
prefLabel
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@ast
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en-gb
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@nl
P2093
P921
P356
P1476
FIF [fibroblast growth factor- ...... eracts specifically with FGF-2
@en
P2093
C Zanibellato
L Van den Berghe
P304
P356
10.1210/MEND.14.11.0556
P407
P577
2000-11-01T00:00:00Z