API5 confers cancer stem cell-like properties through the FGF2-NANOG axis. - Wikidata - awgldk - TriplyDB

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

http://www.wikidata.org/entity/Q37627441

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API5 induces cisplatin resistance through FGFR signaling in human cancer cells.Identification of neoepitopes recognized by tumor-infiltrating lymphocytes (TILs) from patients with glioma.A novel function of API5 (apoptosis inhibitor 5), TLR4-dependent activation of antigen presenting cells

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API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

http://www.wikidata.org/entity/Q37627441

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 16 January 2017

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

@en

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

@nl

type

label

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

@en

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

@nl

prefLabel

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

@en

API5 confers cancer stem cell-like properties through the FGF2-NANOG axis.

@nl

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API5 confers cancer stem cell-like properties through the FGF2-NANOG axis

@en

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B I Lee

http://www.w3.org/2001/XMLSchema#string

C H Choi

http://www.w3.org/2001/XMLSchema#string

H Cho

http://www.w3.org/2001/XMLSchema#string

H S Jang

http://www.w3.org/2001/XMLSchema#string

H-C Park

http://www.w3.org/2001/XMLSchema#string

H-J Lee

http://www.w3.org/2001/XMLSchema#string

J-H Kim

http://www.w3.org/2001/XMLSchema#string

K H Noh

http://www.w3.org/2001/XMLSchema#string

K-H Song

http://www.w3.org/2001/XMLSchema#string

M Son

http://www.w3.org/2001/XMLSchema#string

P2860

Functional identification of Api5 as a suppressor of E2F-dependent apoptosis in vivo

AAC-11 overexpression induces invasion and protects cervical cancer cells from apoptosis

FIF [fibroblast growth factor-2 (FGF-2)-interacting-factor], a nuclear putatively antiapoptotic factor, interacts specifically with FGF-2

AAC-11, a novel cDNA that inhibits apoptosis after growth factor withdrawal

The antiapoptotic protein AAC-11 interacts with and regulates Acinus-mediated DNA fragmentation

Inhibition of tumor angiogenesis and growth by a small-molecule multi-FGF receptor blocker with allosteric properties

Molecular mechanism of SSR128129E, an extracellularly acting, small-molecule, allosteric inhibitor of FGF receptor signaling

Phenotypic characterization of human colorectal cancer stem cells

Prospective identification of tumorigenic breast cancer cells

Stages of embryonic development of the zebrafish

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e285

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scholarly article

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10.1038/ONCSIS.2016.87

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1

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6

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Stephen M. Hewitt

Joon-Yong Chung

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2017-01-16T00:00:00Z

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28092370

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5294250

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