Cleavage of the matricellular protein SPARC by matrix metalloproteinase 3 produces polypeptides that influence angiogenesis
about
Antiangiogenic gene therapy of cancer: recent developmentsUsing proteomics to uncover extracellular matrix interactions during cardiac remodeling.Inhibition of Pterygium Fibroblast Migration and Outgrowth by Bevacizumab and Cyclosporine A Involves Down-Regulation of Matrix Metalloproteinases-3 and -13TP53INP1 decreases pancreatic cancer cell migration by regulating SPARC expressionActivity-dependent shedding of the NMDA receptor glycine binding site by matrix metalloproteinase 3: a PUTATIVE mechanism of postsynaptic plasticityActivation of matrix metalloproteinase-3 and agrin cleavage in cerebral ischemia/reperfusionDirect comparison of human mesenchymal stem cells derived from adipose tissues and bone marrow in mediating neovascularization in response to vascular ischemia.Synergistic effect of stromelysin-1 (matrix metalloproteinase-3) promoter (-1171 5A->6A) polymorphism in oral submucous fibrosis and head and neck lesionsProcessing of the matricellular protein hevin in mouse brain is dependent on ADAMTS4Loss of SPARC-mediated VEGFR-1 suppression after injury reveals a novel antiangiogenic activity of VEGF-AAging-related alterations in the extracellular matrix modulate the microenvironment and influence tumor progression.The novel SPARC family member SMOC-2 potentiates angiogenic growth factor activity.Allelic variation of the MMP3 promoter affects transcription activity through the transcription factor C-MYB in human brain arteriovenous malformations.IgLON cell adhesion molecules are shed from the cell surface of cortical neurons to promote neuronal growth.Anti-cancer role of SPARC, an inhibitor of adipogenesisRevisiting the matricellular concept.Proteolysis of the matricellular protein hevin by matrix metalloproteinase-3 produces a SPARC-like fragment (SLF) associated with neovasculature in a murine glioma model.Matricellular proteins in cardiac adaptation and disease.Lenalidomide inhibits the malignant clone and up-regulates the SPARC gene mapping to the commonly deleted region in 5q- syndrome patients.X-ray fluorescence microscopy reveals large-scale relocalization and extracellular translocation of cellular copper during angiogenesis.Bone matrix osteonectin limits prostate cancer cell growth and survival.A Novel Collagen Matricryptin Reduces Left Ventricular Dilation Post-Myocardial Infarction by Promoting Scar Formation and Angiogenesis.Targeting integrins for the control of tumour angiogenesis.Extracellular influences on tumour angiogenesis in the aged host.Forced expression of MMP9 rescues the loss of angiogenesis and abrogates metastasis of pancreatic tumors triggered by the absence of host SPARC.Matricellular proteins as regulators of cancer metastasis to bone.Differences in gene expression profiles from asbestos-treated SPARC-null and wild-type mouse lungs.Bone marrow-derived cathepsin K cleaves SPARC in bone metastasis.A novel GGA-binding site is required for intracellular sorting mediated by stabilin-1.Role of immune responses in the pathogenesis of low-risk MDS and high-risk MDS: implications for immunotherapy.Role of SPARC in bone remodeling and cancer-related bone metastasis.Astrocyte-secreted matricellular proteins in CNS remodelling during development and diseasePrognostic significance of immunohistochemical epithelial-mesenchymal transition markers in skin melanoma patients.Matrix Metalloproteinases in Myocardial Infarction and Heart Failure.The SPARC protein: an overview of its role in lung cancer and pulmonary fibrosis and its potential role in chronic airways disease.Proteolytic Isoforms of SPARC Induce Adipose Stromal Cell Mobilization in Obesity.The copper binding domain of SPARC mediates cell survival in vitro via interaction with integrin beta1 and activation of integrin-linked kinase.Cyclic AMP phosphodiesterase 4D (PDE4D) Tethers EPAC1 in a vascular endothelial cadherin (VE-Cad)-based signaling complex and controls cAMP-mediated vascular permeabilityAnti-angiogenic peptides identified in thrombospondin type I domains.Effect of cyclic tensile load on the regulation of the expression of matrix metalloproteases (MMPs -1, -3) and structural components in synovial cells.
P2860
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P2860
Cleavage of the matricellular protein SPARC by matrix metalloproteinase 3 produces polypeptides that influence angiogenesis
description
2003 nî lūn-bûn
@nan
2003 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Cleavage of the matricellular ...... es that influence angiogenesis
@ast
Cleavage of the matricellular ...... es that influence angiogenesis
@en
Cleavage of the matricellular ...... es that influence angiogenesis
@en-gb
Cleavage of the matricellular ...... es that influence angiogenesis
@nl
type
label
Cleavage of the matricellular ...... es that influence angiogenesis
@ast
Cleavage of the matricellular ...... es that influence angiogenesis
@en
Cleavage of the matricellular ...... es that influence angiogenesis
@en-gb
Cleavage of the matricellular ...... es that influence angiogenesis
@nl
prefLabel
Cleavage of the matricellular ...... es that influence angiogenesis
@ast
Cleavage of the matricellular ...... es that influence angiogenesis
@en
Cleavage of the matricellular ...... es that influence angiogenesis
@en-gb
Cleavage of the matricellular ...... es that influence angiogenesis
@nl
P2093
P2860
P921
P356
P1476
Cleavage of the matricellular ...... es that influence angiogenesis
@en
P2093
Donald H Maurice
E Helene Sage
James A Bassuk
Melissa Steadele
Pauli Puolakkainen
Sarah E Funk
Thao Truong
P2860
P304
P356
10.1074/JBC.M302946200
P407
P577
2003-09-26T00:00:00Z