A thrombospondin-dependent pathway for a protective ER stress response - Wikidata - awgldk - TriplyDB

A thrombospondin-dependent pathway for a protective ER stress response

A thrombospondin-dependent pathway for a protective ER stress response

http://www.wikidata.org/entity/Q24337738

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Endoplasmic reticulum stress: a novel mechanism and therapeutic target for cardiovascular diseases Neuronal endoplasmic reticulum stress in axon injury and neurodegeneration Keep your heart in shape: molecular chaperone networks for treating heart disease Targeting myocardial remodelling to develop novel therapies for heart failure: a position paper from the Working Group on Myocardial Function of the European Society of Cardiology Calreticulin induces dilated cardiomyopathy Gene Expression and MicroRNA Expression Analysis in Small Arteries of Spontaneously Hypertensive Rats. Evidence for ER Stress Cardiac fibrosis in myocardial infarction-from repair and remodeling to regeneration Nerve growth factor improves functional recovery by inhibiting endoplasmic reticulum stress-induced neuronal apoptosis in rats with spinal cord injury β-adrenergic receptor-dependent alterations in murine cardiac transcript expression are differentially regulated by gefitinib in vivo Thrombospondin-4 controls matrix assembly during development and repair of myotendinous junctions.Translational profiles of medullary myofibroblasts during kidney fibrosis.Control of organization and function of muscle and tendon by thrombospondin-4.Roles for ATF6 and the sarco/endoplasmic reticulum protein quality control system in the heart.Painful nerve injury upregulates thrombospondin-4 expression in dorsal root ganglia Gene expression profiling in tibial muscular dystrophy reveals unfolded protein response and altered autophagy.Hydrogen sulfide attenuates high fat diet-induced cardiac dysfunction via the suppression of endoplasmic reticulum stress.Revisiting the matricellular concept.Genetic Analysis of Connective Tissue Growth Factor as an Effector of Transforming Growth Factor β Signaling and Cardiac Remodeling.Genetic responses to seasonal variation in altitudinal stress: whole-genome resequencing of great tit in eastern Himalayas Members of the thrombospondin gene family bind stromal interaction molecule 1 and regulate calcium channel activity Deficiency of Thrombospondin-4 in Mice Does Not Affect Skeletal Growth or Bone Mass Acquisition, but Causes a Transient Reduction of Articular Cartilage Thickness Modulation of cardiac fibrosis by Krüppel-like factor 6 through transcriptional control of thrombospondin 4 in cardiomyocytes.A H(a)rd Way to Adapt in Cardiac Hypertrophy.Proangiogenic Properties of Thrombospondin-4.STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy Lost in transgenesis: a user's guide for genetically manipulating the mouse in cardiac research.Human Umbilical Tissue-Derived Cells Promote Synapse Formation and Neurite Outgrowth via Thrombospondin Family Proteins Hrd1 and ER-Associated Protein Degradation, ERAD, are Critical Elements of the Adaptive ER Stress Response in Cardiac Myocytes Thrombospondins in the transition from myocardial infarction to heart failure.New concepts of endoplasmic reticulum function in the heart: programmed to conserve.Matricellular proteins in drug delivery: Therapeutic targets, active agents, and therapeutic localization Antagonism of the thromboxane-prostanoid receptor is cardioprotective against right ventricular pressure overload.ATF6 [corrected] and thrombospondin 4: the dynamic duo of the adaptive endoplasmic reticulum stress response Microarray analysis of active cardiac remodeling genes in a familial hypertrophic cardiomyopathy mouse model rescued by a phospholamban knockout.Assessing the contribution of thrombospondin-4 induction and ATF6α activation to endoplasmic reticulum expansion and phenotypic modulation in bladder outlet obstruction.Thrombospondin expression in myofibers stabilizes muscle membranes.Endoplasmic Reticulum Stress and Related Pathological Processes.Cardiomyocyte-specific transforming growth factor β suppression blocks neutrophil infiltration, augments multiple cytoprotective cascades, and reduces early mortality after myocardial infarction.Thrombospondins, potential drug targets for cardiovascular diseases.Thrombospondins: old players, new games

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P2860

A thrombospondin-dependent pathway for a protective ER stress response

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2012 nî lūn-bûn

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2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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name

A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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A thrombospondin-dependent pathway for a protective ER stress response

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Marjorie Maillet

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Michelle A Sargent

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P2860

Characterization of human thrombospondin-4

Interaction of cartilage oligomeric matrix protein/thrombospondin 5 with aggrecan

Pseudoachondroplasia and multiple epiphyseal dysplasia due to mutations in the cartilage oligomeric matrix protein gene

Identification and characterization of thrombospondin-4, a new member of the thrombospondin gene family

Thrombospondin-2 Is Essential for Myocardial Matrix Integrity: Increased Expression Identifies Failure-Prone Cardiac Hypertrophy

Characterization of cartilage oligomeric matrix protein (COMP) in human normal and pseudoachondroplasia musculoskeletal tissues

Trinucleotide expansion mutations in the cartilage oligomeric matrix protein (COMP) gene

Mutations in exon 17B of cartilage oligomeric matrix protein (COMP) cause pseudoachondroplasia

Multiple molecular chaperones complex with misfolded large oligomeric glycoproteins in the endoplasmic reticulum

Thrombospondin-4 is a putative tumour-suppressor gene in colorectal cancer that exhibits age-related methylation

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10.1016/J.CELL.2012.03.050

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Jeffery D. Molkentin

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225289670

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Activating transcription factor 6

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3372931

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