STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
about
Mitsugumin 29 regulates t-tubule architecture in the failing heart.Store-operated Ca2+ entry supports contractile function in hearts of hibernators.LRRC10 is required to maintain cardiac function in response to pressure overload.Cardiac Stim1 Silencing Impairs Adaptive Hypertrophy and Promotes Heart Failure Through Inactivation of mTORC2/Akt Signaling.STIM1-dependent Ca(2+) microdomains are required for myofilament remodeling and signaling in the heart.Transient Receptor Potential Canonical (TRPC)/Orai1-dependent Store-operated Ca2+ Channels: NEW TARGETS OF ALDOSTERONE IN CARDIOMYOCYTES.Enhanced store-operated Ca2+ influx and ORAI1 expression in ventricular fibroblasts from human failing heart.ADP-Induced Ca2+ Signaling and Proliferation of Rat Ventricular Myofibroblasts Depend on Phospholipase C-Linked TRP Channels Activation Within Lipid Rafts.Calcium and Excitation-Contraction Coupling in the HeartTGFBI functions similar to periostin but is uniquely dispensable during cardiac injuryGenomic structural variations lead to dysregulation of important coding and non-coding RNA species in dilated cardiomyopathy.Stromal interaction molecule 1 haploinsufficiency causes maladaptive response to pressure overload.Suppression of Stim1 reduced intracellular calcium concentration and attenuated hypoxia/reoxygenation induced apoptosis in H9C2 cells.A proteolytic fragment of histone deacetylase 4 protects the heart from failure by regulating the hexosamine biosynthetic pathway.Cardiac Remodeling and Disease: Current Understanding of STIM1/Orai1-Mediated Store-Operated Ca2+ Entry in Cardiac Function and Pathology.Cardiac Remodeling and Disease: SOCE and TRPC Signaling in Cardiac Pathology.Cardiovascular and Hemostatic Disorders: SOCE in Cardiovascular Cells: Emerging Targets for Therapeutic Intervention.Tissue Specificity: Store-Operated Ca2+ Entry in Cardiac Myocytes.Role of STIM1 (Stromal Interaction Molecule 1) in Hypertrophy-Related Contractile Dysfunction.Caveolae-localized L-type Ca2+ channels do not contribute to function or hypertrophic signalling in the mouse heart.Essential role for smooth muscle cell stromal interaction molecule-1 in myocardial infarction.The Complex Role of Store Operated Calcium Entry Pathways and Related Proteins in the Function of Cardiac, Skeletal and Vascular Smooth Muscle Cells.
P2860
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P2860
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
description
2015 nî lūn-bûn
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name
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@ast
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@en
type
label
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@ast
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@en
prefLabel
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@ast
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@en
P2093
P2860
P50
P1476
STIM1 elevation in the heart results in aberrant Ca²⁺ handling and cardiomyopathy
@en
P2093
Adam R Burr
Allen J York
Catherine A Makarewich
Hongyu Zhang
Michelle A Sargent
Robert N Correll
Sanjeewa A Goonasekera
Steven R Houser
P2860
P356
10.1016/J.YJMCC.2015.07.032
P577
2015-08-01T00:00:00Z