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Diseases caused by mutations in ORAI1 and STIM1Critical roles for WDR72 in calcium transport and matrix protein removal during enamel maturationMultiple Calcium Export Exchangers and Pumps Are a Prominent Feature of Enamel Organ Cells.Evolutionary Analysis Predicts Sensitive Positions of MMP20 and Validates Newly- and Previously-Identified MMP20 Mutations Causing Amelogenesis ImperfectaThe Na+/Ca2+, K+ exchanger NCKX4 is required for efficient cone-mediated visionHypomaturation amelogenesis imperfecta caused by a novel SLC24A4 mutation.Cell cycle control, DNA damage repair, and apoptosis-related pathways control pre-ameloblasts differentiation during tooth development.Store-operated Ca2+ Entry Modulates the Expression of Enamel Genes.Root anomalies and dentin dysplasia in autosomal recessive hyperphosphatemic familial tumoral calcinosis (HFTC).Ameloblast Modulation and Transport of Cl⁻, Na⁺, and K⁺ during AmelogenesisA targeted next-generation sequencing assay for the molecular diagnosis of genetic disorders with orodental involvementStore-operated Ca2+ entry regulates Ca2+-activated chloride channels and eccrine sweat gland functionReduced Protein Expression of the Na+/Ca2++K+-Exchanger (SLC24A4) in Apical Plasma Membranes of Maturation Ameloblasts of Fluorotic Mice.Store-operated Ca2+ entry controls ameloblast cell function and enamel development.Deletion of Slc26a1 and Slc26a7 Delays Enamel Mineralization in Mice.DENTAL ENAMEL FORMATION AND IMPLICATIONS FOR ORAL HEALTH AND DISEASE.Ca2+ transport and signalling in enamel cells.Enamel: Molecular identity of its transepithelial ion transport system.Amelogenesis Imperfecta: 1 Family, 2 Phenotypes, and 2 Mutated Genes.Amelogenesis Imperfecta; Genes, Proteins, and Pathways.A Functional Study of Mutations in K+-dependent Na+-Ca2+ Exchangers Associated with Amelogenesis Imperfecta and Non-syndromic Oculocutaneous Albinism.Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.Dental enamel cells express functional SOCE channels.Novel FAM83H mutations in patients with amelogenesis imperfecta.Orai1 expression pattern in tooth and craniofacial ectodermal tissues and potential functions during ameloblast differentiation.A homozygous STIM1 mutation impairs store-operated calcium entry and natural killer cell effector function without clinical immunodeficiency.Defective SLC24A4 causes hypomineralized amelogenesis imperfecta (AI)Stromal interaction molecule 1 haploinsufficiency causes maladaptive response to pressure overload.Defective SLC24A4 does not exchange extracellular 4Na+ for cytosolic Ca2+; K+Evidence That Calcium Entry Into Calcium-Transporting Dental Enamel Cells Is Regulated by Cholecystokinin, Acetylcholine and ATP.
P2860
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P2860
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@ast
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en-gb
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@nl
type
label
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@ast
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en-gb
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@nl
prefLabel
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@ast
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en-gb
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@nl
P2093
P2860
P921
P356
P1476
STIM1 and SLC24A4 Are Critical for Enamel Maturation
@en
P2093
A S Richardson
J P Simmer
M Yildirim
P2860
P304
P356
10.1177/0022034514527971
P407
P433
P577
2014-07-01T00:00:00Z