Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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ORAI1 mutations abolishing store-operated Ca2+ entry cause anhidrotic ectodermal dysplasia with immunodeficiency.

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Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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2017 nî lūn-bûn

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2017年の論文

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2017年論文

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2017年論文

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2017年論文

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Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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Stim1 Regulates Enamel Mineralization and Ameloblast Modulation

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I Takahashi

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K Kuremoto

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K Yoshizaki

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M Nakanishi

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M Nikaido

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M Oh-Hora

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N Ryu

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Y Furukawa

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Y Takano

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P2860

STIM1 and SLC24A4 Are Critical for Enamel Maturation

Dual functions for the endoplasmic reticulum calcium sensors STIM1 and STIM2 in T cell activation and tolerance

Capacitative calcium entry: from concept to molecules

Hypomaturation enamel defects in Klk4 knockout/LacZ knockin mice

Improving bioscience research reporting: the ARRIVE guidelines for reporting animal research

STIM1 mutation associated with a syndrome of immunodeficiency and autoimmunity

ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia.

The cystic fibrosis transmembrane conductance regulator (CFTR) is expressed in maturation stage ameloblasts, odontoblasts and bone cells

CRAC channelopathies.

Calcium transport across the dental enamel epithelium.

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1422-1429

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scholarly article

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10.1177/0022034517719872

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English

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Naoto Haruyama

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2017-07-21T00:00:00Z

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28732182

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Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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P2860

Stim1 Regulates Enamel Mineralization and Ameloblast Modulation.

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