M-LDH serves as a sarcolemmal K(ATP) channel subunit essential for cell protection against ischemia
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Chronic mild hypoxia protects heart-derived H9c2 cells against acute hypoxia/reoxygenation by regulating expression of the SUR2A subunit of the ATP-sensitive K+ channelDystrophin is required for the normal function of the cardio-protective K(ATP) channel in cardiomyocytesInfection with AV-SUR2A protects H9C2 cells against metabolic stress: a mechanism of SUR2A-mediated cytoprotection independent from the K(ATP) channel activityThe glycolytic enzymes, glyceraldehyde-3-phosphate dehydrogenase, triose-phosphate isomerase, and pyruvate kinase are components of the K(ATP) channel macromolecular complex and regulate its functionHigh glucose regulates the activity of cardiac sarcolemmal ATP-sensitive K+ channels via 1,3-bisphosphoglycerate: a novel link between cardiac membrane excitability and glucose metabolismHypoxia-induced preconditioning in adult stimulated cardiomyocytes is mediated by the opening and trafficking of sarcolemmal KATP channelsGlyceraldehyde 3-phosphate dehydrogenase serves as an accessory protein of the cardiac sarcolemmal K(ATP) channelMg2+ protects adult beating cardiomyocytes against ischaemiaNicotinamide-rich diet protects the heart against ischaemia-reperfusion in mice: a crucial role for cardiac SUR2AATP-sensitive K+ channel knockout induces cardiac proteome remodeling predictive of heart disease susceptibility.The glycolytic enzymes glyceraldehyde 3-phosphate dehydrogenase and enolase interact with the renal epithelial K+ channel ROMK2 and regulate its function.Human oocytes express ATP-sensitive K(+) channels3-D structural and functional characterization of the purified KATP channel complex Kir6.2-SUR1.K(ATP) channel-dependent metaboproteome decoded: systems approaches to heart failure prediction, diagnosis, and therapy.Cellular remodeling in heart failure disrupts K(ATP) channel-dependent stress toleranceMuscle KATP channels: recent insights to energy sensing and myoprotectionNicotinamide-rich diet improves physical endurance by up-regulating SUR2A in the heartRemodeling of atrial ATP-sensitive K⁺ channels in a model of salt-induced elevated blood pressure.Measuring and evaluating the role of ATP-sensitive K+ channels in cardiac muscle.A spontaneous increase in intracellular Ca2+ in metaphase II human oocytes in vitro can be prevented by drugs targeting ATP-sensitive K+ channelsATP-sensitive K+ channel channel/enzyme multimer: metabolic gating in the heart.Overexpression of SUR2A generates a cardiac phenotype resistant to ischemia.AMP-activated protein kinase mediates preconditioning in cardiomyocytes by regulating activity and trafficking of sarcolemmal ATP-sensitive K(+) channels.KATP Channels in the Cardiovascular System.Phenylephrine preconditioning in embryonic heart H9c2 cells is mediated by up-regulation of SUR2B/Kir6.2: A first evidence for functional role of SUR2B in sarcolemmal KATP channels and cardioprotection.Testosterone protects female embryonic heart H9c2 cells against severe metabolic stress by activating estrogen receptors and up-regulating IES SUR2B.Ageing, gender and cardiac sarcolemmal K(ATP) channels.Comparative proteomic analysis of the ATP-sensitive K+ channel complex in different tissue typesReview. SUR1: a unique ATP-binding cassette protein that functions as an ion channel regulator.Proteomic profiling of KATP channel-deficient hypertensive heart maps risk for maladaptive cardiomyopathic outcome.The role of ATP-sensitive potassium channels in cellular function and protection in the cardiovascular systemThe Regulation and Function of Lactate Dehydrogenase A: Therapeutic Potential in Brain Tumor.Limitations in intense exercise performance of athletes - effect of speed endurance training on ion handling and fatigue development.M-LDH physically associated with sarcolemmal K ATP channels mediates cytoprotection in heart embryonic H9C2 cellsA dual mechanism of cytoprotection afforded by M-LDH in embryonic heart H9C2 cells.Identification and physiological activity of survival factor released from cardiomyocytes during ischaemia and reperfusion.A patient suffering from hypokalemic periodic paralysis is deficient in skeletal muscle ATP-sensitive K channels.Targeted expression of Kir6.2 in mitochondria confers protection against hypoxic stress.Large conductance Ca2+-activated K+ channels sense acute changes in oxygen tension in alveolar epithelial cellsDifferential K(ATP) channel pharmacology in intact mouse heart.
P2860
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P2860
M-LDH serves as a sarcolemmal K(ATP) channel subunit essential for cell protection against ischemia
description
2002 nî lūn-bûn
@nan
2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@ast
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@en
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@nl
type
label
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@ast
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@en
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@nl
prefLabel
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@ast
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@en
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@nl
P2093
P2860
P356
P1433
P1476
M-LDH serves as a sarcolemmal ...... ll protection against ischemia
@en
P2093
Aleksandar Jovanović
Anthony M Davies
Grant R Budas
Harri J Ranki
Russell M Crawford
Sofija Jovanović
Timothy J Wilson
P2860
P304
P356
10.1093/EMBOJ/CDF388
P407
P577
2002-08-01T00:00:00Z