Sam68 enhances the cytoplasmic utilization of intron-containing RNA and is functionally regulated by the nuclear kinase Sik/BRK.
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The STAR RNA binding proteins GLD-1, QKI, SAM68 and SLM-2 bind bipartite RNA motifsLocalization of nucleoporin Tpr to the nuclear pore complex is essential for Tpr mediated regulation of the export of unspliced RNA.The Tpr protein regulates export of mRNAs with retained introns that traffic through the Nxf1 pathway.Regulated association of protein kinase B/Akt with breast tumor kinasehRIP, a cellular cofactor for Rev function, promotes release of HIV RNAs from the perinuclear regionSam68 regulates translation of target mRNAs in male germ cells, necessary for mouse spermatogenesisSam68 is absolutely required for Rev function and HIV-1 productionSam68 exerts separable effects on cell cycle progression and apoptosisPicornaviruses and nuclear functions: targeting a cellular compartment distinct from the replication site of a positive-strand RNA virusExpression of the jaagsiekte sheep retrovirus envelope glycoprotein is sufficient to induce lung tumors in sheepEvolutionary conservation of a molecular machinery for export and expression of mRNAs with retained intronsAn adaptor role for cytoplasmic Sam68 in modulating Src activity during cell polarization.Tap and NXT promote translation of unspliced mRNA.Analysis of synergy between divergent simple retrovirus posttranscriptional control elementsMethods for analyzing peptides and proteins on a chromatographic timescale by electron-transfer dissociation mass spectrometryThe Wilms' tumor 1 (WT1) gene (+KTS isoform) functions with a CTE to enhance translation from an unspliced RNA with a retained intron.An intron with a constitutive transport element is retained in a Tap messenger RNA.The shuttling SR protein 9G8 plays a role in translation of unspliced mRNA containing a constitutive transport element.Brk/PTK6 signaling in normal and cancer cell models.Translational regulation of HIV-1 replication by HIV-1 Rev cellular cofactors Sam68, eIF5A, hRIP, and DDX3.Building a better understanding of the intracellular tyrosine kinase PTK6 - BRK by BRKSRp40 and SRp55 promote the translation of unspliced human immunodeficiency virus type 1 RNAPhosphorylation of nucleoporin Tpr governs its differential localization and is required for its mitotic functionRev-free HIV-1 gene delivery system for targeting Rev-RRE-Crm1 nucleocytoplasmic RNA transport pathwayBreast tumor kinase and extracellular signal-regulated kinase 5 mediate Met receptor signaling to cell migration in breast cancer cells.Gammaretroviral pol sequences act in cis to direct polysome loading and NXF1/NXT-dependent protein production by gag-encoded RNA.A novel function for Sam68: enhancement of HIV-1 RNA 3' end processing.Specificity of the STAR/GSG domain protein Qk1: implications for the regulation of myelination.RAKing in AKT: a tumor suppressor function for the intracellular tyrosine kinase FRK.A cell-based method for screening RNA-protein interactions: identification of constitutive transport element-interacting proteins.Cytoplasmic retention of protein tyrosine kinase 6 promotes growth of prostate tumor cells.Sam68 relocalization into stress granules in response to oxidative stress through complexing with TIA-1Sam68 marks the transcriptionally active stages of spermatogenesis and modulates alternative splicing in male germ cells.Gene regulation at the RNA layer: RNA binding proteins in intercellular signaling networks.Murine MusD retrotransposon: structure and molecular evolution of an "intracellularized" retrovirus.PTK6 inhibition promotes apoptosis of Lapatinib-resistant Her2(+) breast cancer cells by inducing Bim.Nuclear Protein Sam68 Interacts with the Enterovirus 71 Internal Ribosome Entry Site and Positively Regulates Viral Protein Translation.Primary sequence and secondary structure motifs in spleen necrosis virus RU5 confer translational utilization of unspliced human immunodeficiency virus type 1 reporter RNA.Selective translational repression of HIV-1 RNA by Sam68DeltaC occurs by altering PABP1 binding to unspliced viral RNAMechanisms employed by retroviruses to exploit host factors for translational control of a complicated proteome.
P2860
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P2860
Sam68 enhances the cytoplasmic utilization of intron-containing RNA and is functionally regulated by the nuclear kinase Sik/BRK.
description
2003 nî lūn-bûn
@nan
2003 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK
@nl
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@ast
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@en
type
label
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK
@nl
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@ast
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@en
prefLabel
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK
@nl
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@ast
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@en
P2093
P2860
P1476
Sam68 enhances the cytoplasmic ...... by the nuclear kinase Sik/BRK.
@en
P2093
Brian W Guzik
David Rekosh
John H Coyle
Lucia Eisner-Smerage
Stephen J Taylor
Yeou-Cherng Bor
P2860
P304
P356
10.1128/MCB.23.1.92-103.2003
P407
P577
2003-01-01T00:00:00Z