The DNA-binding defect observed in major histocompatibility complex class II regulatory mutants concerns only one member of a family of complexes binding to the X boxes of class II promoters
about
RFXAP, a novel subunit of the RFX DNA binding complex is mutated in MHC class II deficiencyRFX1, a transactivator of hepatitis B virus enhancer I, belongs to a novel family of homodimeric and heterodimeric DNA-binding proteinsAssociations and interactions between bare lymphocyte syndrome factorsA functionally essential domain of RFX5 mediates activation of major histocompatibility complex class II promoters by promoting cooperative binding between RFX and NF-YFunction of major histocompatibility complex class II promoters requires cooperative binding between factors RFX and NF-YRFX1 is identical to enhancer factor C and functions as a transactivator of the hepatitis B virus enhancerRFX1, a single DNA-binding protein with a split dimerization domain, generates alternative complexesMHC Class II-Deficient Combined Immunodeficiency: A Disease of Gene RegulationMutant human cells defective in induction of major histocompatibility complex class II genes by interferon gamma.Affinity enrichment and functional characterization of TRAX1, a novel transcription activator and X1-sequence-binding protein of HLA-DRAFunctional complementation of major histocompatibility complex class II regulatory mutants by the purified X-box-binding protein RFX.Predicting Autism Spectrum Disorder Using Blood-based Gene Expression Signatures and Machine Learning.The major histocompatibility complex class II promoter-binding protein RFX (NF-X) is a methylated DNA-binding protein.The MHC class II transactivator (CIITA) requires conserved leucine charged domains for interactions with the conserved W box promoter element.Differential splicing generates Tvl-1/RFXANK isoforms with different functions.Functional effects of a natural polymorphism in the transcriptional regulatory sequence of HLA-DQB1.Transcriptional regulation of MHC class II genes.Biological implications of HLA-DR expression in tumours.MHC class II gene silencing in trophoblast cells is caused by inhibition of CIITA expression.Locus- and allele-specific DNA-protein interactions in the HLA-DQB1 X box.Interaction of EF-C/RFX-1 with the inverted repeat of viral enhancer regions is required for transactivation.
P2860
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P2860
The DNA-binding defect observed in major histocompatibility complex class II regulatory mutants concerns only one member of a family of complexes binding to the X boxes of class II promoters
description
1992 nî lūn-bûn
@nan
1992 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
The DNA-binding defect observe ...... X boxes of class II promoters
@ast
The DNA-binding defect observe ...... X boxes of class II promoters
@en
The DNA-binding defect observe ...... X boxes of class II promoters
@nl
type
label
The DNA-binding defect observe ...... X boxes of class II promoters
@ast
The DNA-binding defect observe ...... X boxes of class II promoters
@en
The DNA-binding defect observe ...... X boxes of class II promoters
@nl
prefLabel
The DNA-binding defect observe ...... X boxes of class II promoters
@ast
The DNA-binding defect observe ...... X boxes of class II promoters
@en
The DNA-binding defect observe ...... X boxes of class II promoters
@nl
P2093
P2860
P356
P1476
The DNA-binding defect observe ...... X boxes of class II promoters
@en
P2093
C Herrero Sanchez
P2860
P304
P356
10.1128/MCB.12.9.4076
P407
P577
1992-09-01T00:00:00Z