Associations and interactions between bare lymphocyte syndrome factors
about
Identification of the ankyrin repeat proteins ANKRA and RFXANK as novel partners of class IIa histone deacetylasesInterferon-gamma induces major histocompatibility class II transactivator (CIITA), which mediates collagen repression and major histocompatibility class II activation by human aortic smooth muscle cellsZXDC, a novel zinc finger protein that binds CIITA and activates MHC gene transcriptionThe zinc finger proteins ZXDA and ZXDC form a complex that binds CIITA and regulates MHC II gene transcriptionThe insulator factor CTCF controls MHC class II gene expression and is required for the formation of long-distance chromatin interactionsSumoylation of the zinc finger protein ZXDC enhances the function of its transcriptional activation domainNew functions of the major histocompatibility complex class II-specific transcription factor RFXANK revealed by a high-resolution mutagenesis studyAnalysis of ankyrin repeats reveals how a single point mutation in RFXANK results in bare lymphocyte syndromeThe ankyrin repeat as molecular architecture for protein recognitionMitogen-activated protein kinase ERK1/2 regulates the class II transactivatorInterferon gamma repression of collagen (COL1A2) transcription is mediated by the RFX5 complexExpression of the three human major histocompatibility complex class II isotypes exhibits a differential dependence on the transcription factor RFXAPTwo distinct domains within CIITA mediate self-association: involvement of the GTP-binding and leucine-rich repeat domainsSelf-association of CIITA and its transactivation potential.Promoter-specific functions of CIITA and the MHC class II enhanceosome in transcriptional activationPhosphorylation of CIITA directs its oligomerization, accumulation and increased activity on MHCII promoters.Expression of the MHC class II transactivator (CIITA) type IV promoter in B lymphocytes and regulation by IFN-gamma.Inhibition of human T cell leukemia virus type 2 replication by the suppressive action of class II transactivator and nuclear factor Y.Gamma interferon modulates myogenesis through the major histocompatibility complex class II transactivator, CIITA.Genome-wide CIITA-binding profile identifies sequence preferences that dictate function versus recruitment.Major histocompatibility complex class II transactivator CIITA is a viral restriction factor that targets human T-cell lymphotropic virus type 1 Tax-1 function and inhibits viral replication.Uncovering direct and indirect molecular determinants of chromatin loops using a computational integrative approach.CATERPILLERs, pyrin and hereditary immunological disorders.Interferon-γ resets muscle cell fate by stimulating the sequential recruitment of JARID2 and PRC2 to promoters to repress myogenesis.The MHC-II transactivator CIITA, a restriction factor against oncogenic HTLV-1 and HTLV-2 retroviruses: similarities and differences in the inhibition of Tax-1 and Tax-2 viral transactivators.Cohesin regulates MHC class II genes through interactions with MHC class II insulators.Oct-1 maintains an intermediate, stable state of HLA-DRA promoter repression in Rb-defective cells: an Oct-1-containing repressosome that prevents NF-Y binding to the HLA-DRA promoter.Differential splicing generates Tvl-1/RFXANK isoforms with different functions.Kaposi's Sarcoma-Associated Herpesvirus Latency-Associated Nuclear Antigen Inhibits Major Histocompatibility Complex Class II Expression by Disrupting Enhanceosome Assembly through Binding with the Regulatory Factor X ComplexNLRC5 cooperates with the RFX transcription factor complex to induce MHC class I gene expression.Downregulation of CIITA function by protein kinase a (PKA)-mediated phosphorylation: mechanism of prostaglandin E, cyclic AMP, and PKA inhibition of class II major histocompatibility complex expression in monocytic linesKinetics of a gamma interferon response: expression and assembly of CIITA promoter IV and inhibition by methylation.Major histocompatibility complex class II transcriptional platform: assembly of nuclear factor Y and regulatory factor X (RFX) on DNA requires RFX5 dimers.The class II transactivator requires brahma-related gene 1 to activate transcription of major histocompatibility complex class II genes.CIITA regulates transcription onset viaSer5-phosphorylation of RNA Pol II.Activation of a methylated promoter mediated by a sequence-specific DNA-binding protein, RFX.Collagen alpha1(I) gene (COL1A1) is repressed by RFX familyThe S box of major histocompatibility complex class II promoters is a key determinant for recruitment of the transcriptional co-activator CIITA.Solution structure of the heterotrimeric complex between the interaction domains of RFX5 and RFXAP from the RFX gene regulatory complex.Molecular and cellular correlates of the CIITA-mediated inhibition of HTLV-2 Tax-2 transactivator function resulting in loss of viral replication.
P2860
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P2860
Associations and interactions between bare lymphocyte syndrome factors
description
2000 nî lūn-bûn
@nan
2000 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Associations and interactions between bare lymphocyte syndrome factors
@ast
Associations and interactions between bare lymphocyte syndrome factors
@en
Associations and interactions between bare lymphocyte syndrome factors
@nl
type
label
Associations and interactions between bare lymphocyte syndrome factors
@ast
Associations and interactions between bare lymphocyte syndrome factors
@en
Associations and interactions between bare lymphocyte syndrome factors
@nl
prefLabel
Associations and interactions between bare lymphocyte syndrome factors
@ast
Associations and interactions between bare lymphocyte syndrome factors
@en
Associations and interactions between bare lymphocyte syndrome factors
@nl
P2093
P2860
P1476
Associations and interactions between bare lymphocyte syndrome factors
@en
P2093
A M DeSandro
U M Nagarajan
P2860
P304
P356
10.1128/MCB.20.17.6587-6599.2000
P407
P577
2000-09-01T00:00:00Z