VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth - Wikidata - awgldk - TriplyDB

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

http://www.wikidata.org/entity/Q24606018

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ABCB5 identifies a therapy-refractory tumor cell population in colorectal cancer patients Cancer stem cells and the tumor microenvironment: interplay in tumor heterogeneity Stem cells and targeted approaches to melanoma cure Tumor cell vasculogenic mimicry: from controversy to therapeutic promise Alternative Treatments For Melanoma: Targeting BCL-2 Family Members to De-Bulk and Kill Cancer Stem Cells ABCB5 maintains melanoma-initiating cells through a proinflammatory cytokine signaling circuit ABCB5 is a limbal stem cell gene required for corneal development and repair Hypoxia-inducing factors as master regulators of stemness properties and altered metabolism of cancer- and metastasis-initiating cells VEGFR-1 Pseudogene Expression and Regulatory Function in Human Colorectal Cancer Cells Transient TNF regulates the self-renewing capacity of stem-like label-retaining cells in sphere and skin equivalent models of melanoma.Molecular pathways: vasculogenic mimicry in tumor cells: diagnostic and therapeutic implications.Molecular characteristics and metastasis predictor genes of triple-negative breast cancer: a clinical study of triple-negative breast carcinomas Chronic inhibition of tumor cell-derived VEGF enhances the malignant phenotype of colorectal cancer cells.Expression of a functional VEGFR-1 in tumor cells is a major determinant of anti-PlGF antibodies efficacy Induction of Vasculogenic Mimicry Overrides VEGF-A Silencing and Enriches Stem-like Cancer Cells in Melanoma Knockdown of VEGF receptor-1 (VEGFR-1) impairs macrophage infiltration, angiogenesis and growth of clear cell renal cell carcinoma (CRCC).Host-derived pericytes and Sca-1+ cells predominate in the MART-1- stroma fraction of experimentally induced melanoma.Novel biomarkers and therapeutic targets for optimizing the therapeutic management of melanomas Iterative sorting reveals CD133+ and CD133- melanoma cells as phenotypically distinct populations.ALDH1A isozymes are markers of human melanoma stem cells and potential therapeutic targets.Understanding melanoma stem cells CD133+ melanoma subpopulations contribute to perivascular niche morphogenesis and tumorigenicity through vasculogenic mimicry.Targeting Syndecan-1, a molecule implicated in the process of vasculogenic mimicry, enhances the therapeutic efficacy of the L19-IL2 immunocytokine in human melanoma xenografts Melanocytes, melanocyte stem cells, and melanoma stem cells.FK506 binding protein 51 positively regulates melanoma stemness and metastatic potential.Effects of Malignant Melanoma Initiating Cells on T-Cell Activation Genetically determined ABCB5 functionality correlates with pigmentation phenotype and melanoma risk.Therapeutic implications of melanoma heterogeneity.Autocrine VEGFR1 and VEGFR2 signaling promotes survival in human glioblastoma models in vitro and in vivo A new perspective of vasculogenic mimicry: EMT and cancer stem cells (Review).Differential mechanisms of tumor progression in clones from a single heterogeneous human melanoma.The multidrug-resistance transporter ABCB5 is expressed in human placenta.Altered gene products involved in the malignant reprogramming of cancer stem/progenitor cells and multitargeted therapies.Melanoma stem cells and metastasis: mimicking hematopoietic cell trafficking?Therapeutic implications of cellular and molecular biology of cancer stem cells in melanoma.Antitumor activity of a novel anti-vascular endothelial growth factor receptor-1 monoclonal antibody that does not interfere with ligand binding.A 'tête-à tête' between cancer stem cells and endothelial progenitor cells in tumor angiogenesis.Aldehyde dehydrogenase isozymes: markers of cancer stem cells in human melanoma.Tumor cell vascular mimicry: Novel targeting opportunity in melanoma.ABCB5-Targeted Chemoresistance Reversal Inhibits Merkel Cell Carcinoma Growth.

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VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

http://www.wikidata.org/entity/Q24606018

description

2011 nî lūn-bûn

@nan

2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2011年の論文

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2011年論文

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2011年論文

@zh-hant

2011年論文

@zh-hk

2011年論文

@zh-mo

2011年論文

@zh-tw

2011年论文

@wuu

name

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@ast

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@en

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@nl

type

label

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@ast

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@en

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@nl

prefLabel

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@ast

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@en

VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@nl

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0008-5472.CAN-10-1660

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Cancer Research

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VEGFR-1 expressed by malignant melanoma-initiating cells is required for tumor growth

@en

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Ana-Maria Waaga-Gasser

http://www.w3.org/2001/XMLSchema#string

Brian J Wilson

http://www.w3.org/2001/XMLSchema#string

George F Murphy

http://www.w3.org/2001/XMLSchema#string

Hans R Widlund

http://www.w3.org/2001/XMLSchema#string

Jie Ma

http://www.w3.org/2001/XMLSchema#string

Karim R Saab

http://www.w3.org/2001/XMLSchema#string

Markus H Frank

http://www.w3.org/2001/XMLSchema#string

Martin Gasser

http://www.w3.org/2001/XMLSchema#string

Natasha Y Frank

http://www.w3.org/2001/XMLSchema#string

Qian Zhan

http://www.w3.org/2001/XMLSchema#string

P2860

Nucleotide sequence and expression of a novel human receptor-type tyrosine kinase gene (flt) closely related to the fms family

Regulation of progenitor cell fusion by ABCB5 P-glycoprotein, a novel human ATP-binding cassette transporter

Inhibition of vascular endothelial cell growth factor activity by an endogenously encoded soluble receptor

Isolation of tumorigenic circulating melanoma cells

Modulation of T-cell activation by malignant melanoma initiating cells

VEGFR1-positive haematopoietic bone marrow progenitors initiate the pre-metastatic niche

Human melanoma-initiating cells express neural crest nerve growth factor receptor CD271

Identification of cells initiating human melanomas

Tenascin-C promotes melanoma progression by maintaining the ABCB5-positive side population

Efficient tumour formation by single human melanoma cells

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1474-85

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scholarly article

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3592572

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10.1158/0008-5472.CAN-10-1660

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4

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21212411

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3083845

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