Innate immune recognition of an AT-rich stem-loop DNA motif in the Plasmodium falciparum genome - Wikidata - awgldk - TriplyDB

Innate immune recognition of an AT-rich stem-loop DNA motif in the Plasmodium falciparum genome

Innate immune recognition of an AT-rich stem-loop DNA motif in the Plasmodium falciparum genome

http://www.wikidata.org/entity/Q24611264

about

Cyclic GMP-AMP synthase is a cytosolic DNA sensor that activates the type I interferon pathway Intrinsic host restriction factors of human cytomegalovirus replication and mechanisms of viral escape Dendritic Cells and Their Multiple Roles during Malaria Infection Beyond empiricism: informing vaccine development through innate immunity research The Regulation of CD4(+) T Cell Responses during Protozoan Infections RNA mediated Toll-like receptor stimulation in health and disease Recognition of cytosolic DNA by cGAS and other STING-dependent sensors Type I interferon programs innate myeloid dynamics and gene expression in the virally infected nervous system Young lives lost as B cells falter: what we are learning about antibody responses in malaria.Plasmodium falciparum merozoite surface protein 1 blocks the proinflammatory protein S100P International Union of Basic and Clinical Pharmacology. XCVI. Pattern recognition receptors in health and disease Nucleic Acid-Sensing Receptors: Rheostats of Autoimmunity and Autoinflammation Induction of interferon-stimulated genes by IRF3 promotes replication of Toxoplasma gondii Type I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental Malaria NLRC3, a member of the NLR family of proteins, is a negative regulator of innate immune signaling induced by the DNA sensor STING The N terminus of cGAS de-oligomerizes the cGAS:DNA complex and lifts the DNA size restriction of core-cGAS activity.The T-Cell Inhibitory Molecule Butyrophilin-Like 2 Is Up-regulated in Mild Plasmodium falciparum Infection and Is Protective During Experimental Cerebral Malaria.Splenic differentiation and emergence of CCR5+CXCL9+CXCL10+ monocyte-derived dendritic cells in the brain during cerebral malaria.The role of the spleen in malaria.A surprising role for uric acid: the inflammatory malaria response.Type I IFN signaling in CD8- DCs impairs Th1-dependent malaria immunity.Plasmacytoid dendritic cells appear inactive during sub-microscopic Plasmodium falciparum blood-stage infection, yet retain their ability to respond to TLR stimulation Malaria immunity in man and mosquito: insights into unsolved mysteries of a deadly infectious disease.Dual engagement of the NLRP3 and AIM2 inflammasomes by plasmodium-derived hemozoin and DNA during malaria Interferons and interferon regulatory factors in malaria Plasmodium falciparum-infected erythrocytes and IL-12/IL-18 induce diverse transcriptomes in human NK cells: IFN-α/β pathway versus TREM signaling.Inflammasomes in host response to protozoan parasites.The CD14+CD16+ inflammatory monocyte subset displays increased mitochondrial activity and effector function during acute Plasmodium vivax malaria.STING specifies IRF3 phosphorylation by TBK1 in the cytosolic DNA signaling pathway STING/MPYS mediates host defense against Listeria monocytogenes infection by regulating Ly6C(hi) monocyte migration.STING recognition of cytoplasmic DNA instigates cellular defense.Plasmodium falciparum-derived uric acid precipitates induce maturation of dendritic cells.Defining the range of pathogens susceptible to Ifitm3 restriction using a knockout mouse model Transcriptional analysis of murine macrophages infected with different Toxoplasma strains identifies novel regulation of host signaling pathways Suppression of systemic autoimmunity by the innate immune adaptor STING.Exposure-dependent control of malaria-induced inflammation in children.STING-dependent cytosolic DNA sensing mediates innate immune recognition of immunogenic tumors.Coinfection with Blood-Stage Plasmodium Promotes Systemic Type I Interferon Production during Pneumovirus Infection but Impairs Inflammation and Viral Control in the Lung.Mild Plasmodium falciparum malaria following an episode of severe malaria is associated with induction of the interferon pathway in Malawian children.Genome-wide Analysis of Host-Plasmodium yoelii Interactions Reveals Regulators of the Type I Interferon Response.

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Innate immune recognition of an AT-rich stem-loop DNA motif in the Plasmodium falciparum genome

http://www.wikidata.org/entity/Q24611264

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2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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Parisa Kalantari

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Peggy Parroche

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P2860

Genome sequence of the human malaria parasite Plasmodium falciparum

Peroxisomes are signaling platforms for antiviral innate immunity

RIG-I-dependent sensing of poly(dA:dT) through the induction of an RNA polymerase III-transcribed RNA intermediate

The adaptor protein MITA links virus-sensing receptors to IRF3 transcription factor activation

RNA polymerase III detects cytosolic DNA and induces type I interferons through the RIG-I pathway

DAI (DLM-1/ZBP1) is a cytosolic DNA sensor and an activator of innate immune response

IPS-1, an adaptor triggering RIG-I- and Mda5-mediated type I interferon induction

AIM2 recognizes cytosolic dsDNA and forms a caspase-1-activating inflammasome with ASC

Cardif is an adaptor protein in the RIG-I antiviral pathway and is targeted by hepatitis C virus

STING is an endoplasmic reticulum adaptor that facilitates innate immune signalling

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Ricardo Tostes Gazzinelli

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51550830

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Plasmodium falciparum

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