Human papillomaviruses modulate expression of microRNA 203 upon epithelial differentiation to control levels of p63 proteins - Wikidata - awgldk - TriplyDB

Human papillomaviruses modulate expression of microRNA 203 upon epithelial differentiation to control levels of p63 proteins

Human papillomaviruses modulate expression of microRNA 203 upon epithelial differentiation to control levels of p63 proteins

http://www.wikidata.org/entity/Q24629203

about

MicroRNA 203 expression in keratinocytes is dependent on regulation of p53 levels by E6 MicroRNAs Expressed during Viral Infection: Biomarker Potential and Therapeutic Considerations Cervical cancer screening in developing countries at a crossroad: Emerging technologies and policy choices Not miR-ly micromanagers: the functions and regulatory networks of microRNAs in mammalian skin Multistep model of cervical cancer: participation of miRNAs and coding genes The microRNA feedback regulation of p63 in cancer progression The JAK-STAT transcriptional regulator, STAT-5, activates the ATM DNA damage pathway to induce HPV 31 genome amplification upon epithelial differentiation Cutaneous papillomavirus E6 oncoproteins associate with MAML1 to repress transactivation and NOTCH signaling Quantifying mRNA and microRNA with qPCR in cervical carcinogenesis: a validation of reference genes to ensure accurate data miR-375 activates p21 and suppresses telomerase activity by coordinately regulating HPV E6/E7, E6AP, CIP2A, and 14-3-3ζ Identification of C/EBPα as a novel target of the HPV8 E6 protein regulating miR-203 in human keratinocytes.Human papillomavirus 16 E5 modulates the expression of host microRNAs Altered microRNA expression associated with chromosomal changes contributes to cervical carcinogenesis.Viral oncogenes, noncoding RNAs, and RNA splicing in human tumor viruses miRNAs in the pathogenesis of oncogenic human viruses.Small molecule inhibitors of human papillomavirus protein - protein interactions.Suppression of STAT-1 expression by human papillomaviruses is necessary for differentiation-dependent genome amplification and plasmid maintenance.p63 is necessary for the activation of human papillomavirus late viral functions upon epithelial differentiation.Regulation of cellular miRNA expression by human papillomaviruses.Viruses and microRNAs: RISCy interactions with serious consequences.Epstein-Barr virus downregulates microRNA 203 through the oncoprotein latent membrane protein 1: a contribution to increased tumor incidence in epithelial cells.Two less common human microRNAs miR-875 and miR-3144 target a conserved site of E6 oncogene in most high-risk human papillomavirus subtypes Productive Lifecycle of Human Papillomaviruses that Depends Upon Squamous Epithelial Differentiation.MicroRNA-203 represses selection and expansion of oncogenic Hras transformed tumor initiating cells.Post-Transcriptional Regulation of KLF4 by High-Risk Human Papillomaviruses Is Necessary for the Differentiation-Dependent Viral Life Cycle.The fanconi anemia pathway limits human papillomavirus replication Cancer associated human papillomaviruses.Low expression levels of putative HPV encoded microRNAs in cervical samples A Decade of Global mRNA and miRNA Profiling of HPV-Positive Cell Lines and Clinical Specimens.Virology and molecular pathogenesis of HPV (human papillomavirus)-associated oropharyngeal squamous cell carcinoma Methylation-mediated transcriptional repression of microRNAs during cervical carcinogenesis.Galectin-7 Regulates Keratinocyte Proliferation and Differentiation through JNK-miR-203-p63 Signaling.In-depth analysis of the interaction of HIV-1 with cellular microRNA biogenesis and effector mechanisms.Human papillomaviruses modulate microRNA 145 expression to directly control genome amplification.Nip the HPV encoded evil in the cancer bud: HPV reshapes TRAILs and signaling landscapes Low folate levels are associated with methylation-mediated transcriptional repression of miR-203 and miR-375 during cervical carcinogenesis.Cellular transformation by human papillomaviruses: lessons learned by comparing high- and low-risk viruses HPV16 E5 and KGFR/FGFR2b interplay in differentiating epithelial cells miR-2861 acts as a tumor suppressor via targeting EGFR/AKT2/CCND1 pathway in cervical cancer induced by human papillomavirus virus 16 E6 MicroRNA profiling of Sendai virus-infected A549 cells identifies miR-203 as an interferon-inducible regulator of IFIT1/ISG56.

P2860

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P2860

Human papillomaviruses modulate expression of microRNA 203 upon epithelial differentiation to control levels of p63 proteins

http://www.wikidata.org/entity/Q24629203

description

2010 nî lūn-bûn

@nan

2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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type

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Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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Human papillomaviruses modulat ...... control levels of p63 proteins

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P1433

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Journal of Virology

P1476

Human papillomaviruses modulat ...... control levels of p63 proteins

@en

P2093

Laimonis A Laimins

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Marta Melar-New

http://www.w3.org/2001/XMLSchema#string

P2860

MicroRNAs: novel regulators involved in the pathogenesis of psoriasis?

The nuclear RNase III Drosha initiates microRNA processing

p63 regulates proliferation and differentiation of developmentally mature keratinocytes

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

The role of RNAi and microRNAs in animal virus replication and antiviral immunity

MicroRNAs modulate hematopoietic lineage differentiation

SCF TrCP1 activates and ubiquitylates TAp63gamma

miR-203 represses 'stemness' by repressing DeltaNp63

Human papillomaviruses activate the ATM DNA damage pathway for viral genome amplification upon differentiation

Dicer function is essential for lung epithelium morphogenesis.

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5212-21

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scholarly article

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220993

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10.1128/JVI.00078-10

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10

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84

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2010-05-01T00:00:00Z

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20219920

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2863797

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