Human papillomaviruses activate the ATM DNA damage pathway for viral genome amplification upon differentiation
about
Parvovirus minute virus of mice induces a DNA damage response that facilitates viral replicationHuman papillomaviruses modulate expression of microRNA 203 upon epithelial differentiation to control levels of p63 proteinsPapillomavirus E6 Oncoproteins Take Common Structural Approaches to Solve Different Biological ProblemsThe DNA damage response induced by infection with human cytomegalovirus and other virusesModulation of DNA damage and repair pathways by human tumour virusesThe Role of the DNA Damage Response throughout the Papillomavirus Life CycleThe acetyltransferase Tip60 is a critical regulator of the differentiation-dependent amplification of human papillomaviruses.The JAK-STAT transcriptional regulator, STAT-5, activates the ATM DNA damage pathway to induce HPV 31 genome amplification upon epithelial differentiationNuclear accumulation of the papillomavirus E1 helicase blocks S-phase progression and triggers an ATM-dependent DNA damage responseThe papillomavirus E1 helicase activates a cellular DNA damage response in viral replication fociATM and ATR activities maintain replication fork integrity during SV40 chromatin replication.Papillomavirus genomes associate with BRD4 to replicate at fragile sites in the host genome.Kaposi Sarcoma Herpesvirus (KSHV) Latency-Associated Nuclear Antigen (LANA) recruits components of the MRN (Mre11-Rad50-NBS1) repair complex to modulate an innate immune signaling pathway and viral latencyDual expression of Epstein-Barr virus, latent membrane protein-1 and human papillomavirus-16 E6 transform primary mouse embryonic fibroblasts through NF-κB signalingAn E2F1-mediated DNA damage response contributes to the replication of human cytomegalovirus.Productive replication of human papillomavirus 31 requires DNA repair factor Nbs1.Construction of a full transcription map of human papillomavirus type 18 during productive viral infection.The DNA damage response in viral-induced cellular transformationHuman papillomaviruses-related cancers. Presence and prevention strategies in the Middle east and north African regionsNuclear export of human papillomavirus type 31 E1 is regulated by Cdk2 phosphorylation and required for viral genome maintenance.Snapshots: chromatin control of viral infection.CD66+ cells in cervical precancers are partially differentiated progenitors with neoplastic traitsRecruitment of Brd4 to the human papillomavirus type 16 DNA replication complex is essential for replication of viral DNA.Persistence of human papillomavirus infection: keys to malignant progressionViral carcinogenesis: factors inducing DNA damage and virus integrationPapillomaviruses use recombination-dependent replication to vegetatively amplify their genomes in differentiated cellsCHK2 kinase in the DNA damage response and beyondA conserved amphipathic helix in the N-terminal regulatory region of the papillomavirus E1 helicase is required for efficient viral DNA replication.p38MAPK and MK2 pathways are important for the differentiation-dependent human papillomavirus life cycle.DNA damage repair genes controlling human papillomavirus (HPV) episome levels under conditions of stability and extreme instabilityBrd4 is displaced from HPV replication factories as they expand and amplify viral DNAProteomic analysis and identification of cellular interactors of the giant ubiquitin ligase HERC2.Re-expression of HPV16 E2 in SiHa (human cervical cancer) cells potentiates NF-κB activation induced by TNF-α concurrently increasing senescence and survivalSuppression of STAT-1 expression by human papillomaviruses is necessary for differentiation-dependent genome amplification and plasmid maintenance.p63 is necessary for the activation of human papillomavirus late viral functions upon epithelial differentiation.Recent Insights into the Control of Human Papillomavirus (HPV) Genome Stability, Loss, and Degradation.Tobacco smoke activates human papillomavirus 16 p97 promoter and cooperates with high-risk E6/E7 for oxidative DNA damage in lung cells.Current trends in the etiology and diagnosis of HPV-related head and neck cancers.Activation of NF-κB by human papillomavirus 16 E1 limits E1-dependent viral replication through degradation of E1.SV40 utilizes ATM kinase activity to prevent non-homologous end joining of broken viral DNA replication products
P2860
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P2860
Human papillomaviruses activate the ATM DNA damage pathway for viral genome amplification upon differentiation
description
2009 nî lūn-bûn
@nan
2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Human papillomaviruses activat ...... ification upon differentiation
@ast
Human papillomaviruses activat ...... ification upon differentiation
@en
Human papillomaviruses activat ...... ification upon differentiation
@nl
type
label
Human papillomaviruses activat ...... ification upon differentiation
@ast
Human papillomaviruses activat ...... ification upon differentiation
@en
Human papillomaviruses activat ...... ification upon differentiation
@nl
prefLabel
Human papillomaviruses activat ...... ification upon differentiation
@ast
Human papillomaviruses activat ...... ification upon differentiation
@en
Human papillomaviruses activat ...... ification upon differentiation
@nl
P2860
P3181
P1433
P1476
Human papillomaviruses activat ...... ification upon differentiation
@en
P2093
Cary A Moody
Laimonis A Laimins
P2860
P304
P3181
P356
10.1371/JOURNAL.PPAT.1000605
P407
P577
2009-10-01T00:00:00Z