Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice - Wikidata - awgldk - TriplyDB

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

http://www.wikidata.org/entity/Q24632417

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Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutations Basic exploratory research versus guideline-compliant studies used for hazard evaluation and risk assessment: bisphenol A as a case study Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4 Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruption Exposure to environmentally relevant doses of the xenoestrogen bisphenol-A alters development of the fetal mouse mammary gland Endocrine-disrupting chemicals: an Endocrine Society scientific statement Why public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol A Does breast cancer start in the womb?Oral exposure to bisphenol a increases dimethylbenzanthracene-induced mammary cancer in rats Induction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposure In vivo effects of bisphenol A in laboratory rodent studies An overview of the effects of organic compounds on women's reproductive health and birth outcomes Prenatal Exposure to BPA and Offspring Outcomes: The Diabesogenic Behavior of BPA Progesterone and Overlooked Endocrine Pathways in Breast Cancer Pathogenesis Environmental causes of cancer: endocrine disruptors as carcinogens EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals CtBP1 associates metabolic syndrome and breast carcinogenesis targeting multiple miRNAs Use of amniotic fluid for determining pregnancies at risk of preterm birth and for studying diseases of potential environmental etiology Prenatal environmental exposures, epigenetics, and disease Estrogens in the wrong place at the wrong time: Fetal BPA exposure and mammary cancer Exposure of neonatal rats to the endocrine disrupter Bisphenol A affects ontogenic expression pattern of testicular steroid receptors and their coregulators The role of Bisphenol A in shaping the brain, epigenome and behavior.Bisphenol-A and diethylstilbestrol exposure induces the expression of breast cancer associated long noncoding RNA HOTAIR in vitro and in vivo.Bisphenol A: Perinatal exposure and body weight Neoplasia as development gone awry: the role of endocrine disruptors.Perinatal exposure to bisphenol a increases adult mammary gland progesterone response and cell number.Perinatal exposure to oestradiol and bisphenol A alters the prostate epigenome and increases susceptibility to carcinogenesis Circulating serum xenoestrogens and mammographic breast density Prenatal exposure to BPA alters the epigenome of the rat mammary gland and increases the propensity to neoplastic development Gestational high-fat diet and bisphenol A exposure heightens mammary cancer risk.Female reproductive disorders: the roles of endocrine-disrupting compounds and developmental timing Epigenetic influences of low-dose bisphenol A in primary human breast epithelial cells.Combinations of physiologic estrogens with xenoestrogens alter calcium and kinase responses, prolactin release, and membrane estrogen receptor trafficking in rat pituitary cells.Estrogenic potency of bisphenol S, polyethersulfone and their metabolites generated by the rat liver S9 fractions on a MVLN cell using a luciferase reporter gene assay.Endocrine disruptors, environmental oxygen, epigenetics and pregnancy Low-dose BPA exposure alters the mesenchymal and epithelial transcriptomes of the mouse fetal mammary gland.Perinatal exposure to environmentally relevant levels of bisphenol A decreases fertility and fecundity in CD-1 mice Exposure to the Endocrine Disruptor Bisphenol A Alters Susceptibility for Mammary Cancer In utero exposure to diethylstilbestrol (DES) or bisphenol-A (BPA) increases EZH2 expression in the mammary gland: an epigenetic mechanism linking endocrine disruptors to breast cancer

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P2860

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

http://www.wikidata.org/entity/Q24632417

description

2005 nî lūn-bûn

@nan

2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2005年の論文

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2005年論文

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2005年論文

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2005年論文

@zh-hk

2005年論文

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2005年論文

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2005年论文

@wuu

name

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@ast

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@en

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@nl

type

label

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@ast

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@en

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@nl

prefLabel

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@ast

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@en

Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

@nl

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Perinatal exposure to bisphenol-A alters peripubertal mammary gland development in mice

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Ana M Soto

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Beverly S Rubin

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Carlos Sonnenschein

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Caroline M Markey

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Enrique H Luque

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Monica Muñoz-de-Toro

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Perinaaz R Wadia

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P2860

Urinary concentrations of bisphenol A and 4-nonylphenol in a human reference population

Relative binding affinity-serum modified access (RBA-SMA) assay predicts the relative in vivo bioactivity of the xenoestrogens bisphenol A and octylphenol

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

Perinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levels

Estrogen receptor null mice: what have we learned and where will they lead us?

Interaction of estrogenic chemicals and phytoestrogens with estrogen receptor beta

Exposure to bisphenol A advances puberty

Estrogenicity of resin-based composites and sealants used in dentistry

Paralogous mouse Hox genes, Hoxa9, Hoxb9, and Hoxd9, function together to control development of the mammary gland in response to pregnancy

Mammary gland development in adult mice requires epithelial and stromal estrogen receptor alpha

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4138-47

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10.1210/EN.2005-0340

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146

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7821610

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15919749

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2834307

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