about
Plastics derived endocrine disruptors (BPA, DEHP and DBP) induce epigenetic transgenerational inheritance of obesity, reproductive disease and sperm epimutationsBasic exploratory research versus guideline-compliant studies used for hazard evaluation and risk assessment: bisphenol A as a case studyEstrogenic chemicals in plastic and oral contraceptives disrupt development of the fetal mouse prostate and urethra.Exposure to a low dose of bisphenol A during fetal life or in adulthood alters maternal behavior in miceBisphenol A is released from used polycarbonate animal cages into water at room temperatureThe estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistanceUrinary concentrations of bisphenol A and 4-nonylphenol in a human reference populationAn extensive new literature concerning low-dose effects of bisphenol A shows the need for a new risk assessmentEffects of environmental agents on the attainment of puberty: considerations when assessing exposure to environmental chemicals in the National Children's Study.Altered breast development in young girls from an agricultural environmentHormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responsesThe estrogenic endocrine disrupting chemical bisphenol A (BPA) and obesityPerinatal exposure to bisphenol-A alters peripubertal mammary gland development in miceBisphenol-A exposure in utero leads to epigenetic alterations in the developmental programming of uterine estrogen responsePolycarbonate bottle use and urinary bisphenol A concentrationsDevelopmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4Bisphenol-A and the great divide: a review of controversies in the field of endocrine disruptionExposure to environmentally relevant doses of the xenoestrogen bisphenol-A alters development of the fetal mouse mammary glandEndocrine-disrupting chemicals: an Endocrine Society scientific statementExposure of the U.S. population to bisphenol A and 4-tertiary-octylphenol: 2003-2004Low phytoestrogen levels in feed increase fetal serum estradiol resulting in the "fetal estrogenization syndrome" and obesity in CD-1 miceOral exposure to bisphenol a increases dimethylbenzanthracene-induced mammary cancer in ratsPrenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in lifeOnset of spermatogenesis is accelerated by gestational administration of 1,2,3,4,6,7-hexachlorinated naphthalene in male rat offspringInduction of mammary gland ductal hyperplasias and carcinoma in situ following fetal bisphenol A exposureIn vivo effects of bisphenol A in laboratory rodent studiesCombining xenoestrogens at levels below individual no-observed-effect concentrations dramatically enhances steroid hormone action.Disposition of orally administered 2,2-Bis(4-hydroxyphenyl)propane (Bisphenol A) in pregnant rats and the placental transfer to fetusesParent bisphenol A accumulation in the human maternal-fetal-placental unitEarly cerebral activities of the environmental estrogen bisphenol A appear to act via the somatostatin receptor subtype sst(2)Effects of perinatal exposure to bisphenol A on play behavior of female and male juvenile rats.Effects of perinatal exposure to bisphenol A on sociosexual behavior of female and male rats.Androgens and environmental antiandrogens affect reproductive development and play behavior in the Sprague-Dawley ratPerinatal exposure to low doses of bisphenol A affects body weight, patterns of estrous cyclicity, and plasma LH levelsThe mouse uterotrophic assay: a reevaluation of its validity in assessing the estrogenicity of bisphenol AAltered profiles of spontaneous novelty seeking, impulsive behavior, and response to D-amphetamine in rats perinatally exposed to bisphenol A.The masculinization of the fetus during pregnancy due to inhalation of diesel exhaust.Large effects from small exposures. I. Mechanisms for endocrine-disrupting chemicals with estrogenic activityThe need to decide if all estrogens are intrinsically similarDevelopmental increases in rat hepatic microsomal UDP-glucuronosyltransferase activities toward xenoestrogens and decreases during pregnancy.
P2860
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P2860
description
1999 nî lūn-bûn
@nan
1999 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Exposure to bisphenol A advances puberty
@ast
Exposure to bisphenol A advances puberty
@en
Exposure to bisphenol A advances puberty
@nl
type
label
Exposure to bisphenol A advances puberty
@ast
Exposure to bisphenol A advances puberty
@en
Exposure to bisphenol A advances puberty
@nl
prefLabel
Exposure to bisphenol A advances puberty
@ast
Exposure to bisphenol A advances puberty
@en
Exposure to bisphenol A advances puberty
@nl
P2093
P3181
P356
P1433
P1476
Exposure to bisphenol A advances puberty
@en
P2093
A K Hotchkiss
F S vom Saal
J G Vandenbergh
K A Thayer
K L Howdeshell
P2888
P3181
P356
10.1038/44517
P407
P577
1999-10-21T00:00:00Z
P6179
1032623467