Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells - Wikidata - awgldk - TriplyDB

Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells

Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells

http://www.wikidata.org/entity/Q24642091

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CCN5: biology and pathophysiology The Transcriptomic Evolution of Mammalian Pregnancy: Gene Expression Innovations in Endometrial Stromal Fibroblasts Investigating microenvironmental regulation of human chordoma cell behaviour Epigenetic activation of the prostaglandin receptor EP4 promotes resistance to endocrine therapy for breast cancer Deficiency of CCN5/WISP-2-Driven Program in breast cancer Promotes Cancer Epithelial cells to mesenchymal stem cells and Breast Cancer growth.WISP1/CCN4 inhibits adipocyte differentiation through repression of PPARγ activity.New insights into the role of EMT in tumor immune escape.Non-canonical HIF-2α function drives autonomous breast cancer cell growth via an AREG-EGFR/ErbB4 autocrine loop.Cysteine-rich 61-connective tissue growth factor-nephroblastoma-overexpressed 5 (CCN5)/Wnt-1-induced signaling protein-2 (WISP-2) regulates microRNA-10b via hypoxia-inducible factor-1α-TWIST signaling networks in human breast cancer cells The CCN proteins: important signaling mediators in stem cell differentiation and tumorigenesis.Intrinsic bevacizumab resistance is associated with prolonged activation of autocrine VEGF signaling and hypoxia tolerance in colorectal cancer cells and can be overcome by nintedanib, a small molecule angiokinase inhibitor.Polycationic calixarene PTX013, a potent cytotoxic agent against tumors and drug resistant cancer.CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway Association of the metastatic phenotype with CCN family members among breast and oral cancer cells.MCF-7 Human Breast Cancer Cells Form Differentiated Microtissues in Scaffold-Free Hydrogels.CCN5/WISP-2: A micromanager of breast cancer progression Modification of MCF-10A cells with pioglitazone and serum-rich growth medium increases soluble factors in the conditioned medium, likely reducing BT-474 cell growth.WISP-2 in human gastric cancer and its potential metastatic suppressor role in gastric cancer cells mediated by JNK and PLC-γ pathways The use of chelated radionuclide (samarium-153-ethylenediaminetetramethylenephosphonate) to modulate phenotype of tumor cells and enhance T cell-mediated killing Wnt signaling in mammary glands: plastic cell fates and combinatorial signaling.Molecular and pathological signatures of epithelial-mesenchymal transitions at the cancer invasion front Predictive factors of sensitivity to elisidepsin, a novel Kahalalide F-derived marine compound CCL25 mediates migration, invasion and matrix metalloproteinase expression by breast cancer cells in a CCR9-dependent fashion.Hypoxia-inducible factor-mediated induction of WISP-2 contributes to attenuated progression of breast cancer.Dual roles of CCN proteins in breast cancer progression.Loss of WISP2/CCN5 in estrogen-dependent MCF7 human breast cancer cells promotes a stem-like cell phenotype.Glucocorticoids induce CCN5/WISP-2 expression and attenuate invasion in oestrogen receptor-negative human breast cancer cells.Role of CCN5 (WNT1 inducible signaling pathway protein 2) in pancreatic islets.Targeting WNT1-inducible signaling pathway protein 2 alters human breast cancer cell susceptibility to specific lysis through regulation of KLF-4 and miR-7 expression.Priming and potentiation of DNA damage response by fibronectin in human colon cancer cells and tumor-derived myofibroblasts.Potential effects of chrysin on MDA-MB-231 cells.MicroRNA-183 regulates Ezrin expression in lung cancer cells.CCN5/WISP-2 restores ER-∝ in normal and neoplastic breast cells and sensitizes triple negative breast cancer cells to tamoxifen The Novel Secreted Adipokine WNT1-inducible Signaling Pathway Protein 2 (WISP2) Is a Mesenchymal Cell Activator of Canonical WNT.The expression of Wnt-1 inducible signaling pathway protein-2 in astrocytoma: Correlation between pathological grade and clinical outcome.CCN5/WISP-2 promotes growth arrest of triple-negative breast cancer cells through accumulation and trafficking of p27(Kip1) via Skp2 and FOXO3a regulation.Genome-Wide Transcriptome Analysis of Estrogen Receptor-Positive and Human Epithelial Growth Factor Receptor 2-Positive Breast Cancers by Ribonucleic Acid Sequencing.Leptin-induced ER-α-positive breast cancer cell viability and migration is mediated by suppressing CCN5-signaling via activating JAK/AKT/STAT-pathway.Differential in Vitro Biological Action, Coregulator Interactions, and Molecular Dynamic Analysis of Bisphenol A (BPA), BPAF, and BPS Ligand-ERα Complexes.

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Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells

http://www.wikidata.org/entity/Q24642091

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2008 nî lūn-bûn

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2008 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2008年の論文

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Molecular and Cellular Biology

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Role of WISP-2/CCN5 in the mai ...... e in human breast cancer cells

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Asmaà Fritah

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Christian Gespach

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Cécile Saucier

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Gérard Redeuilh

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Marc Bracke

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Michèle Sabbah

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Olivier De Wever

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Sylvain Drouot

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P2860

WISP genes are members of the connective tissue growth factor family that are up-regulated in wnt-1-transformed cells and aberrantly expressed in human colon tumors

The Hallmarks of Cancer

Involvement of chemokine receptors in breast cancer metastasis

Proposal for a unified CCN nomenclature.

Histological grading and prognosis in breast cancer; a study of 1409 cases of which 359 have been followed for 15 years

CCN5 is a growth arrest-specific gene that regulates smooth muscle cell proliferation and motility

A structural approach to the role of CCN (CYR61/CTGF/NOV) proteins in tumourigenesis

How many more breast cancer predisposition genes are there?

Epithelial-mesenchymal transitions in tumour progression

The transcription factor snail is a repressor of E-cadherin gene expression in epithelial tumour cells

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1114-23

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scholarly article

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4598609

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10.1128/MCB.01335-07

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3

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28

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Rosette Lidereau

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2008-02-01T00:00:00Z

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5777310

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18070926

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2223394

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