CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway - Wikidata - awgldk - TriplyDB

CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway

CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway

http://www.wikidata.org/entity/Q35102072

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Mechanisms of TGFβ-Induced Epithelial-Mesenchymal Transition Taking aim at the extracellular matrix: CCN proteins as emerging therapeutic targets Gender differences in transcriptional signature of developing rat testes and ovaries following embryonic exposure to 2,3,7,8-TCDD Deficiency of CCN5/WISP-2-Driven Program in breast cancer Promotes Cancer Epithelial cells to mesenchymal stem cells and Breast Cancer growth.Genome-wide RNA-Sequencing analysis identifies a distinct fibrosis gene signature in the conjunctiva after glaucoma surgery CCN1/CYR61: the very model of a modern matricellular protein.A comprehensive characterization of genome-wide copy number aberrations in colorectal cancer reveals novel oncogenes and patterns of alterations TGF-β signaling in breast cancer cell invasion and bone metastasis.CCN5 attenuates profibrotic phenotypes of fibroblasts through the Smad6-CCN2 pathway: Potential role in epidural ﬁbrosis.CCN5/WISP-2: A micromanager of breast cancer progression Early gene expression in salivary gland after isoproterenol treatment.WISP2 regulates preadipocyte commitment and PPARγ activation by BMP4.Restricted adipogenesis in hypertrophic obesity: the role of WISP2, WNT, and BMP4 Loss of WISP2/CCN5 in estrogen-dependent MCF7 human breast cancer cells promotes a stem-like cell phenotype.Overexpressing the novel autocrine/endocrine adipokine WISP2 induces hyperplasia of the heart, white and brown adipose tissues and prevents insulin resistance Molecular Therapy for Degenerative Disc Disease: Clues from Secretome Analysis of the Notochordal Cell-Rich Nucleus Pulposus.ZEB/miR-200 feedback loop: at the crossroads of signal transduction in cancer.Glucocorticoids induce CCN5/WISP-2 expression and attenuate invasion in oestrogen receptor-negative human breast cancer cells.Somatic gene copy number alterations in colorectal cancer: new quest for cancer drivers and biomarkers.Role of CCN5 (WNT1 inducible signaling pathway protein 2) in pancreatic islets.Emerging roles of CCN proteins in vascular development and pathology.Human pancreatic cancer progression: an anarchy among CCN-siblings.Targeting WNT1-inducible signaling pathway protein 2 alters human breast cancer cell susceptibility to specific lysis through regulation of KLF-4 and miR-7 expression.Anti-fibrotic effect of CCN3 accompanied by altered gene expression profile of the CCN family CCN5/WISP-2 promotes growth arrest of triple-negative breast cancer cells through accumulation and trafficking of p27(Kip1) via Skp2 and FOXO3a regulation.CCN5/WISP2 and metabolic diseases.Leptin-induced ER-α-positive breast cancer cell viability and migration is mediated by suppressing CCN5-signaling via activating JAK/AKT/STAT-pathway.The potential application of strategic released apigenin from polymeric carrier in pulmonary fibrosis.CCN2 and CCN5 exerts opposing effect on fibroblast proliferation and transdifferentiation induced by TGF-β.

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CCN5, a novel transcriptional repressor of the transforming growth factor β signaling pathway

http://www.wikidata.org/entity/Q35102072

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

CCN5, a novel transcriptional ...... wth factor β signaling pathway

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CCN5, a novel transcriptional ...... wth factor β signaling pathway

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CCN5, a novel transcriptional ...... wth factor β signaling pathway

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CCN5, a novel transcriptional ...... wth factor β signaling pathway

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Molecular and Cellular Biology

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CCN5, a novel transcriptional ...... wth factor β signaling pathway

@en

P2093

Céline Prunier

http://www.w3.org/2001/XMLSchema#string

Gérard Redeuilh

http://www.w3.org/2001/XMLSchema#string

Joël Lachuer

http://www.w3.org/2001/XMLSchema#string

Kathleen Lambein

http://www.w3.org/2001/XMLSchema#string

Michèle Sabbah

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Nathalie Ferrand

http://www.w3.org/2001/XMLSchema#string

Nicolas Nazaret

http://www.w3.org/2001/XMLSchema#string

Olivier De Wever

http://www.w3.org/2001/XMLSchema#string

Sylvie Dumont

http://www.w3.org/2001/XMLSchema#string

Virginie Megalophonos

http://www.w3.org/2001/XMLSchema#string

P2860

WISP genes are members of the connective tissue growth factor family that are up-regulated in wnt-1-transformed cells and aberrantly expressed in human colon tumors

mHDA1/HDAC5 histone deacetylase interacts with and represses MEF2A transcriptional activity

Receptor-interacting protein 140 directly recruits histone deacetylases for gene silencing

Proposal for a unified CCN nomenclature.

c-Jun interacts with the corepressor TG-interacting factor (TGIF) to suppress Smad2 transcriptional activity

Role of WISP-2/CCN5 in the maintenance of a differentiated and noninvasive phenotype in human breast cancer cells

A structural approach to the role of CCN (CYR61/CTGF/NOV) proteins in tumourigenesis

Epithelial-mesenchymal transitions in tumour progression

Smad4 and FAST-1 in the assembly of activin-responsive factor

Microarray analysis reveals differential gene expression patterns in tumors of the pineal region

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1459-1469

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10.1128/MCB.01316-10

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7

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31

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2011-01-24T00:00:00Z

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49781489

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21262769

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3135290

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