Specific contribution of human T-type calcium channel isotypes (alpha(1G), alpha(1H) and alpha(1I)) to neuronal excitability
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Caveolin-3 regulates protein kinase A modulation of the Ca(V)3.2 (alpha1H) T-type Ca2+ channelsSignal processing by T-type calcium channel interactions in the cerebellumA Recurrent Mutation in CACNA1G Alters Cav3.1 T-Type Calcium-Channel Conduction and Causes Autosomal-Dominant Cerebellar AtaxiaGating effects of mutations in the Cav3.2 T-type calcium channel associated with childhood absence epilepsyT-type calcium channels contribute to colonic hypersensitivity in a rat model of irritable bowel syndromeBiology of Parkinson's disease: pathogenesis and pathophysiology of a multisystem neurodegenerative disorderHippocampal CA3 transcriptome signature correlates with initial precipitating injury in refractory mesial temporal lobe epilepsyFunctional expression of T-type Ca2+ channels in spinal motoneurons of the adult turtleGene transcription and splicing of T-type channels are evolutionarily-conserved strategies for regulating channel expression and gatingAlternative splicing of the rat Ca(v)3.3 T-type calcium channel gene produces variants with distinct functional properties(1).Structural determinants of the high affinity extracellular zinc binding site on Cav3.2 T-type calcium channels.Functional impact of alternative splicing of human T-type Cav3.3 calcium channels.Contrasting anesthetic sensitivities of T-type Ca2+ channels of reticular thalamic neurons and recombinant Ca(v)3.3 channels.Forward suppression in the auditory cortex is caused by the Ca(v)3.1 calcium channel-mediated switch from bursting to tonic firing at thalamocortical projections.CaV3.2 is the major molecular substrate for redox regulation of T-type Ca2+ channels in the rat and mouse thalamus.I-II loop structural determinants in the gating and surface expression of low voltage-activated calcium channels.Analgesic effect of a mixed T-type channel inhibitor/CB2 receptor agonist.Role of antispasmodics in the treatment of irritable bowel syndrome.Transient and big are key features of an invertebrate T-type channel (LCav3) from the central nervous system of Lymnaea stagnalisTranscriptional regulation of α1H T-type calcium channel under hypoxiaRegulation of endogenous conductances in GnRH neurons by estrogensBursting of thalamic neurons and states of vigilance.Calcium current homeostasis and synaptic deficits in hippocampal neurons from Kelch-like 1 knockout mice.Role for T-type Ca2+ channels in sleep waves.Imaging of thalamocortical dysrhythmia in neuropsychiatry.Functional roles of cytoplasmic loops and pore lining transmembrane helices in the voltage-dependent inactivation of HVA calcium channels.Diabetic neuropathy enhances voltage-activated Ca2+ channel activity and its control by M4 muscarinic receptors in primary sensory neurons.CaV1.2/CaV3.x channels mediate divergent vasomotor responses in human cerebral arteries.Multiple T-type Ca2+ current subtypes in electrophysiologically characterized hamster dorsal horn neurons: possible role in spinal sensory integration.Availability of low-threshold Ca2+ current in retinal ganglion cells.Kisspeptin and Gonadotropin-Releasing Hormone Neuronal Excitability: Molecular Mechanisms Driven by 17β-Estradiol.Elimination of the actin-binding domain in kelch-like 1 protein induces T-type calcium channel modulation only in the presence of action potential waveforms.Phosphorylation of the Cav3.2 T-type calcium channel directly regulates its gating propertiesCa-α1T, a fly T-type Ca2+ channel, negatively modulates sleep.Voltage-gated calcium channels and idiopathic generalized epilepsies.Ionic mechanisms of endogenous bursting in CA3 hippocampal pyramidal neurons: a model study.mRNA expression of ion channels in GnRH neurons: subtype-specific regulation by 17β-estradiol17β-oestradiol regulation of gonadotrophin-releasing hormone neuronal excitability.Molecular identity and functional properties of a novel T-type Ca2+ channel cloned from the sensory epithelia of the mouse inner earNumb-mediated neurite outgrowth is isoform-dependent, and requires activation of voltage-dependent calcium channels.
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P2860
Specific contribution of human T-type calcium channel isotypes (alpha(1G), alpha(1H) and alpha(1I)) to neuronal excitability
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2002 nî lūn-bûn
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2002 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2002 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2002年の論文
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2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
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name
Specific contribution of human ...... (1I)) to neuronal excitability
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Specific contribution of human ...... (1I)) to neuronal excitability
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Specific contribution of human ...... (1I)) to neuronal excitability
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type
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Specific contribution of human ...... (1I)) to neuronal excitability
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Specific contribution of human ...... (1I)) to neuronal excitability
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Specific contribution of human ...... (1I)) to neuronal excitability
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Specific contribution of human ...... (1I)) to neuronal excitability
@ast
Specific contribution of human ...... (1I)) to neuronal excitability
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Specific contribution of human ...... (1I)) to neuronal excitability
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P3181
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Specific contribution of human ...... (1I)) to neuronal excitability
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Edward Perez-Reyes
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P3181
P356
10.1113/JPHYSIOL.2001.013269
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P577
2002-04-01T00:00:00Z