T-type calcium channels contribute to colonic hypersensitivity in a rat model of irritable bowel syndrome
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The role of visceral hypersensitivity in irritable bowel syndrome: pharmacological targets and novel treatmentsThe Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic PotentialMain ion channels and receptors associated with visceral hypersensitivity in irritable bowel syndromeImmunohistological demonstration of CaV3.2 T-type voltage-gated calcium channel expression in soma of dorsal root ganglion neurons and peripheral axons of rat and mouse.Analgesic effect of a mixed T-type channel inhibitor/CB2 receptor agonist.Characterization of novel cannabinoid based T-type calcium channel blockers with analgesic effects.1,4-Dihydropyridine derivatives with T-type calcium channel blocking activity attenuate inflammatory and neuropathic pain.Role of antispasmodics in the treatment of irritable bowel syndrome.Retrieval of context-associated memory is dependent on the Ca(v)3.2 T-type calcium channel.Functional characterization and analgesic effects of mixed cannabinoid receptor/T-type channel ligands.NMP-7 inhibits chronic inflammatory and neuropathic pain via block of Cav3.2 T-type calcium channels and activation of CB2 receptors.Small organic molecule disruptors of Cav3.2 - USP5 interactions reverse inflammatory and neuropathic pain.Z944: a first in class T-type calcium channel modulator for the treatment of pain.Drug management of visceral pain: concepts from basic research.Dorsal root ganglion neurons and tyrosine hydroxylase--an intriguing association with implications for sensation and pain.Inflammatory mediator bradykinin increases population of sensory neurons expressing functional T-type Ca(2+) channels.Functional chromaffin cell plasticity in response to stress: focus on nicotinic, gap junction, and voltage-gated Ca2+ channels.Comparative effects of α2δ-1 ligands in mouse models of colonic hypersensitivity.Pharmacological Inhibition of Voltage-gated Ca(2+) Channels for Chronic Pain Relief.Assessment of the effectiveness and safety of Ethosuximide in the Treatment of non-Diabetic Peripheral Neuropathic Pain: EDONOT-protocol of a randomised, parallel, controlled, double-blinded and multicentre clinical trial.Efficacy of otilonium bromide in irritable bowel syndrome: a pooled analysisT-type Ca2+ channels in normal and abnormal brain functions.Calcium-permeable ion channels in pain signaling.Role of T-type channels in vasomotor function: team player or chameleon?T-type calcium channels in chronic pain: mouse models and specific blockers.Colonic smooth muscle cells and colonic motility patterns as a target for irritable bowel syndrome therapy: mechanisms of action of otilonium bromide.A cell-permeant peptide corresponding to the cUBP domain of USP5 reverses inflammatory and neuropathic pain.Targeting voltage-gated calcium channels in neurological and psychiatric diseases.Regulation of the T-type Ca(2+) channel Cav3.2 by hydrogen sulfide: emerging controversies concerning the role of H2 S in nociception.Analgesic effect of a broad-spectrum dihydropyridine inhibitor of voltage-gated calcium channels.Ion channels, ion channel receptors, and visceral hypersensitivity in irritable bowel syndrome.Assessment of the effectiveness and safety of ethosuximide in the treatment of abdominal pain related to irritable bowel syndrome - IBSET: protocol of a randomised, parallel, controlled, double-blind and multicentre trial.Recent advances in the development of T-type calcium channel blockers for pain intervention.Synthesis and characterization of a disubstituted piperazine derivative with T-type channel blocking action and analgesic properties.A common haplotype containing functional CACNA1H variants is frequently coinherited with increased TPSAB1 copy number.Modulation of enteric neurons by interleukin-6 and corticotropin-releasing factor contributes to visceral hypersensitivity and altered colonic motility in a rat model of irritable bowel syndrome.Chronic deficit in nitric oxide elicits oxidative stress and augments T-type calcium-channel contribution to vascular tone of rodent arteries and arterioles.Experimental Models of Irritable Bowel Syndrome and the Role of the Enteric Neurotransmission.Peripheral contribution of NGF and ASIC1a to colonic hypersensitivity in a rat model of irritable bowel syndrome.Ameliorative effect of chlorpromazine hydrochloride on visceral hypersensitivity in rats: possible involvement of 5-HT2A receptor.
P2860
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P2860
T-type calcium channels contribute to colonic hypersensitivity in a rat model of irritable bowel syndrome
description
2011 nî lūn-bûn
@nan
2011 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
T-type calcium channels contri ...... el of irritable bowel syndrome
@ast
T-type calcium channels contri ...... el of irritable bowel syndrome
@en
T-type calcium channels contri ...... el of irritable bowel syndrome
@nl
type
label
T-type calcium channels contri ...... el of irritable bowel syndrome
@ast
T-type calcium channels contri ...... el of irritable bowel syndrome
@en
T-type calcium channels contri ...... el of irritable bowel syndrome
@nl
prefLabel
T-type calcium channels contri ...... el of irritable bowel syndrome
@ast
T-type calcium channels contri ...... el of irritable bowel syndrome
@en
T-type calcium channels contri ...... el of irritable bowel syndrome
@nl
P2093
P2860
P50
P3181
P356
P1476
T-type calcium channels contri ...... el of irritable bowel syndrome
@en
P2093
Abdelkrim Alloui
Agathe Gelot
Alain Eschalier
Denis Ardid
Emilie Muller
Fabrice Marger
Joël Nargeot
Juan F Sanguesa Ferrer
Terrance P Snutch
P2860
P304
11268-11273
P3181
P356
10.1073/PNAS.1100869108
P407
P577
2011-06-20T00:00:00Z