Deficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injury
about
Human neural stem cells differentiate and promote locomotor recovery in an early chronic spinal cord injury NOD-scid mouse modelInflammogenesis of Secondary Spinal Cord InjuryEmerging and Novel Functions of Complement Protein C1qMolecular and cellular changes in the lumbar spinal cord following thoracic injury: regulation by treadmill locomotor trainingAge-dependent modulation of cortical transcriptomes in spinal cord injury and repairAnalysis of host-mediated repair mechanisms after human CNS-stem cell transplantation for spinal cord injury: correlation of engraftment with recoverySyndromics: a bioinformatics approach for neurotrauma researchB cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.The complement cascade: Yin-Yang in neuroinflammation--neuro-protection and -degeneration.Comparison of immunopathology and locomotor recovery in C57BL/6, BUB/BnJ, and NOD-SCID mice after contusion spinal cord injuryThe alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injuryContribution of complement activation pathways to neuropathology differs among mouse models of Alzheimer's disease.New insights of an old defense system: structure, function, and clinical relevance of the complement system.Genome-wide gene expression profiling of stress response in a spinal cord clip compression injury modelComplement protein C1q modulates neurite outgrowth in vitro and spinal cord axon regeneration in vivo.The Ryk receptor is expressed in glial and fibronectin-expressing cells after spinal cord injury.Cornel Iridoid Glycoside Improves Locomotor Impairment and Decreases Spinal Cord Damage in Rats.MicroRNA dysregulation in spinal cord injury: causes, consequences and therapeutics.Complement activation in the injured central nervous system: another dual-edged sword?Inflammatory cascades mediate synapse elimination in spinal cord compression.Myelin as an inflammatory mediator: Myelin interactions with complement, macrophages, and microglia in spinal cord injury.Targeted modulation of the neuroinflammatory response after spinal cord injury: the ongoing quest for the "holy grail".Complement Protein C3 Suppresses Axon Growth and Promotes Neuron Loss.Apoptosis signal-regulating kinase 1 mediates striatal degeneration via the regulation of C1q.Delayed post-injury administration of C5a improves regeneration and functional recovery after spinal cord injury in mice.Selective killing of spinal cord neural stem cells impairs locomotor recovery in a mouse model of spinal cord injury.Cell cycle and complement inhibitors may be specific for treatment of spinal cord injury in aged and young mice: Transcriptomic analyses.
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P2860
Deficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injury
description
2008 nî lūn-bûn
@nan
2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Deficiency in complement C1q i ...... tcome after spinal cord injury
@ast
Deficiency in complement C1q i ...... tcome after spinal cord injury
@en
Deficiency in complement C1q i ...... tcome after spinal cord injury
@nl
type
label
Deficiency in complement C1q i ...... tcome after spinal cord injury
@ast
Deficiency in complement C1q i ...... tcome after spinal cord injury
@en
Deficiency in complement C1q i ...... tcome after spinal cord injury
@nl
prefLabel
Deficiency in complement C1q i ...... tcome after spinal cord injury
@ast
Deficiency in complement C1q i ...... tcome after spinal cord injury
@en
Deficiency in complement C1q i ...... tcome after spinal cord injury
@nl
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P2860
P3181
P1476
Deficiency in complement C1q i ...... tcome after spinal cord injury
@en
P2093
Adrian M Burgos
Aileen J Anderson
Frank P T Hamers
Hal X Nguyen
Manuel D Galvan
Mitra J Hooshmand
Sabina Luchetti
P2860
P304
P3181
P356
10.1523/JNEUROSCI.2823-08.2008
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P577
2008-12-17T00:00:00Z