B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice. - Wikidata - awgldk - TriplyDB

B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.

B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.

http://www.wikidata.org/entity/Q30490538

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Quantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environment Neurotrauma and inflammation: CNS and PNS responses Adrenergic-mediated loss of splenic marginal zone B cells contributes to infection susceptibility after stroke Fenbendazole improves pathological and functional recovery following traumatic spinal cord injury.Natural IgM antibodies that bind neoepitopes exposed as a result of spinal cord injury , drive secondary injury by activating complement The paradox of chronic neuroinflammation, systemic immune suppression, autoimmunity after traumatic chronic spinal cord injury The PPAR alpha agonist gemfibrozil is an ineffective treatment for spinal cord injured mice.Endogenous antibodies promote rapid myelin clearance and effective axon regeneration after nerve injury.Progression from IgD+ IgM+ to isotype-switched B cells is site specific during coronavirus-induced encephalomyelitis.A mouse model of ischemic spinal cord injury with delayed paralysis caused by aortic cross-clamping.A Rehabilomics framework for personalized and translational rehabilitation research and care for individuals with disabilities: Perspectives and considerations for spinal cord injury.The alternative and terminal pathways of complement mediate post-traumatic spinal cord inflammation and injury Genome-wide gene expression profiling of stress response in a spinal cord clip compression injury model B-lymphocyte-mediated delayed cognitive impairment following stroke.Fcγ receptor-mediated inflammation inhibits axon regeneration.Anti-inflammatory treatments during the chronic phase of spinal cord injury improve locomotor function in adult mice.Pathogenic natural antibodies propagate cerebral injury following ischemic stroke in mice.Challenging the role of adaptive immunity in neurotrauma: Rag1(-/-) mice lacking mature B and T cells do not show neuroprotection after closed head injury.Analgesia or addiction?: implications for morphine use after spinal cord injury.Proteomic Analysis of the Spatio-temporal Based Molecular Kinetics of Acute Spinal Cord Injury Identifies a Time- and Segment-specific Window for Effective Tissue Repair Macrophage migration inhibitory factor (MIF) is essential for inflammatory and neuropathic pain and enhances pain in response to stress Traumatic spinal cord injury in mice with human immune systems Antibody profiling identifies novel antigenic targets in spinal cord injury patients.Identification of autoantibodies to glial fibrillary acidic protein in spinal cord injury patients B-cell maturation antigen, a proliferation-inducing ligand, and B-cell activating factor are candidate mediators of spinal cord injury-induced autoimmunity IVIg attenuates complement and improves spinal cord injury outcomes in mice.Neurotoxic or Neuroprotective? Current Controversies in SCI-Induced Autoimmunity Complement activation in the injured central nervous system: another dual-edged sword?Intervertebral disk degeneration in dogs: consequences, diagnosis, treatment, and future directions.MicroRNAs: Roles in Regulating Neuroinflammation.Does B lymphocyte-mediated autoimmunity contribute to post-stroke dementia?The role of peripheral immune cells in the CNS in steady state and disease.Phagocytosis of neuronal debris by microglia is associated with neuronal damage in multiple sclerosis.An optimized method for enumerating CNS derived memory B cells during viral-induced inflammation.Activated GL7+ B cells are maintained within the inflamed CNS in the absence of follicle formation during viral encephalomyelitis.Modulating the immune response in spinal cord injury.B-Cell Depletion with CD20 Antibodies as New Approach in the Treatment of Inflammatory and Immunological Events Associated with Spinal Cord Injury.Hormonal therapy in traumatic spinal cord injury.Involvement of the choroid plexus in the inflammatory response after acute spinal cord injury in dogs: an immunohistochemical study.B cells and autoantibodies: complex roles in CNS injury

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P2860

B cells produce pathogenic antibodies and impair recovery after spinal cord injury in mice.

http://www.wikidata.org/entity/Q30490538

description

2009 nî lūn-bûn

@nan

2009 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

@wuu

name

B cells produce pathogenic ant ...... er spinal cord injury in mice.

@ast

B cells produce pathogenic ant ...... er spinal cord injury in mice.

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type

label

B cells produce pathogenic ant ...... er spinal cord injury in mice.

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B cells produce pathogenic ant ...... er spinal cord injury in mice.

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B cells produce pathogenic ant ...... er spinal cord injury in mice.

@ast

B cells produce pathogenic ant ...... er spinal cord injury in mice.

@en

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Journal of Clinical Investigation

P1476

B cells produce pathogenic ant ...... er spinal cord injury in mice.

@en

P2093

Daniel P Ankeny

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Phillip G Popovich

http://www.w3.org/2001/XMLSchema#string

Zhen Guan

http://www.w3.org/2001/XMLSchema#string

P2860

Characterization of early and terminal complement proteins associated with polymorphonuclear leukocytes in vitro and in vivo after spinal cord injury

Deficiency in complement C1q improves histological and functional locomotor outcome after spinal cord injury

Reduced neuronal cell death after experimental brain injury in mice lacking a functional alternative pathway of complement activation

Anti-inflammatory activity of IVIG mediated through the inhibitory Fc receptor.

Method for quantitation of IgG subclass antibodies in mouse serum by enzyme-linked immunosorbent assay.

Immunomodulation of autoimmune and inflammatory diseases with intravenous immune globulin.

Autoimmune channelopathies and related neurological disorders.

Complement plays an important role in spinal cord injury and represents a therapeutic target for improving recovery following trauma.

Innate autoimmunity.

Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation.

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2990-2999

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10.1172/JCI39780

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10

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119

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2009-09-21T00:00:00Z

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19770513

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2752085

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