Role of SP1-binding domains in in vivo transcriptional regulation of the human immunodeficiency virus type 1 long terminal repeat
about
The cellular factor TRP-185 regulates RNA polymerase II binding to HIV-1 TAR RNARole of flanking E box motifs in human immunodeficiency virus type 1 TATA element functionDifferent members of the Sp1 multigene family exert opposite transcriptional regulation of the long terminal repeat of HIV-1Synergistic activation of the human immunodeficiency virus type 1 promoter by the viral Tat protein and cellular transcription factor Sp1Human transcription factor YY1 represses human immunodeficiency virus type 1 transcription and virion productionT helper cell activation and human retroviral pathogenesisFunctional roles for the TATA promoter and enhancers in basal and Tat-induced expression of the human immunodeficiency virus type 1 long terminal repeatIn vitro and in vivo binding of human immunodeficiency virus type 1 Tat protein and Sp1 transcription factorCOUP-TF interacting protein 2 represses the initial phase of HIV-1 gene transcription in human microglial cellsInhibition of phosphodiesterase type IV suppresses human immunodeficiency virus type 1 replication and cytokine production in primary T cells: involvement of NF-kappaB and NFATA novel HIV-1-encoded microRNA enhances its viral replication by targeting the TATA box region.Regulation of HIV-1 transcription in cells of the monocyte-macrophage lineage.Opinion: Inhibition of Blood-Brain Barrier Repair as a Mechanism in HIV-1 Disease.Modulation of Sp1 phosphorylation by human immunodeficiency virus type 1 Tat.Functional interactions between C/EBP, Sp1, and COUP-TF regulate human immunodeficiency virus type 1 gene transcription in human brain cells.The human immunodeficiency virus type 1 Tat protein up-regulates the promoter activity of the beta-chemokine monocyte chemoattractant protein 1 in the human astrocytoma cell line U-87 MG: role of SP-1, AP-1, and NF-kappaB consensus sites.Combinatorial latency reactivation for HIV-1 subtypes and variantsMolecular pathways in virus-induced cytokine production.Promoter scanning for transcription inhibition with DNA-binding polyamides.The ZiN/POZ domain of ZF5 is required for both transcriptional activation and repression.The activation region of the Tat protein of human immunodeficiency virus type-1 functions in yeast.Genetic selection for context-dependent stochastic phenotypes: Sp1 and TATA mutations increase phenotypic noise in HIV-1 gene expression.Efficient trans-activation by the HIV-2 Tat protein requires a duplicated TAR RNA structure.In vivo transcriptional regulation of the human immunodeficiency virus in the central nervous system in transgenic miceIdentification of a replication-competent pathogenic human immunodeficiency virus type 1 with a duplication in the TCF-1alpha region but lacking NF-kappaB binding sitesInduction of AIDS by simian immunodeficiency virus lacking NF-kappaB and SP1 binding elements.Genotypic and phenotypic characterization of long terminal repeat sequences from long-term survivors of human immunodeficiency virus type 1 infection.Lineage-specific and ubiquitous biological roles of the mammalian transcription factor LSFcis-acting sequences located downstream of the human immunodeficiency virus type 1 promoter affect its chromatin structure and transcriptional activityUpstream U3 sequences in simian immunodeficiency virus are selectively deleted in vivo in the absence of an intact nef gene.Naturally occurring genotypes of the human immunodeficiency virus type 1 long terminal repeat display a wide range of basal and Tat-induced transcriptional activities.Second-site long terminal repeat (LTR) revertants of replication-defective human immunodeficiency virus: effects of revertant TATA box motifs on virus infectivity, LTR-directed expression, in vitro RNA synthesis, and binding of basal transcription fHuman immunodeficiency virus type 1 infection of the brain.Human immunodeficiency viruses containing heterologous enhancer/promoters are replication competent and exhibit different lymphocyte tropisms.Replication of type 1 human immunodeficiency viruses containing linker substitution mutations in the -201 to -130 region of the long terminal repeatActivation of a heterologous promoter by human immunodeficiency virus type 1 Tat requires Sp1 and is distinct from the mode of activation by acidic transcriptional activators.Increased spacing between Sp1 and TATAA renders human immunodeficiency virus type 1 replication defective: implication for Tat function.Variable role of the long terminal repeat Sp1-binding sites in human immunodeficiency virus replication in T lymphocytes.Linker-scanning mutational analysis of the transcriptional activity of the human immunodeficiency virus type 1 long terminal repeatCNS-specific regulatory elements in brain-derived HIV-1 strains affect responses to latency-reversing agents with implications for cure strategies.
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P2860
Role of SP1-binding domains in in vivo transcriptional regulation of the human immunodeficiency virus type 1 long terminal repeat
description
1989 nî lūn-bûn
@nan
1989 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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P2093
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Role of SP1-binding domains in ...... us type 1 long terminal repeat
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P2093
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1989-06-01T00:00:00Z