Variable role of the long terminal repeat Sp1-binding sites in human immunodeficiency virus replication in T lymphocytes.
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Specific NF-kappa B subunits act in concert with Tat to stimulate human immunodeficiency virus type 1 transcriptionRegulation of human immunodeficiency virus type 1 and cytokine gene expression in myeloid cells by NF-kappa B/Rel transcription factorsFunctional roles for the TATA promoter and enhancers in basal and Tat-induced expression of the human immunodeficiency virus type 1 long terminal repeatRegulation of HIV-1 transcription in cells of the monocyte-macrophage lineage.Functional properties of the HIV-1 long terminal repeat containing single-nucleotide polymorphisms in Sp site III and CCAAT/enhancer binding protein site I.Tat-independent replication of human immunodeficiency viruses.Regulation of Mycobacterium tuberculosis-dependent HIV-1 transcription reveals a new role for NFAT5 in the toll-like receptor pathway.Insights into cellular factors that regulate HIV-1 replication in human cellsSelective infection of human T-lymphotropic virus type 1 (HTLV-1)-infected cells by chimeric human immunodeficiency viruses containing HTLV-1 tax response elements in the long terminal repeat.Identification of a replication-competent pathogenic human immunodeficiency virus type 1 with a duplication in the TCF-1alpha region but lacking NF-kappaB binding sitesInduction of AIDS by simian immunodeficiency virus lacking NF-kappaB and SP1 binding elements.Genotypic and phenotypic characterization of long terminal repeat sequences from long-term survivors of human immunodeficiency virus type 1 infection.Multiple NF-κB sites in HIV-1 subtype C long terminal repeat confer superior magnitude of transcription and thereby the enhanced viral predominance.Second-site long terminal repeat (LTR) revertants of replication-defective human immunodeficiency virus: effects of revertant TATA box motifs on virus infectivity, LTR-directed expression, in vitro RNA synthesis, and binding of basal transcription fHuman immunodeficiency virus type 1 infection of the brain.Replication of type 1 human immunodeficiency viruses containing linker substitution mutations in the -201 to -130 region of the long terminal repeatThe RRE of human immunodeficiency virus type 1 contributes to cell-type-specific viral tropism.Genetic differences accounting for evolution and pathogenicity of simian immunodeficiency virus from a sooty mangabey monkey after cross-species transmission to a pig-tailed macaque.An interaction between the DNA-binding domains of RelA(p65) and Sp1 mediates human immunodeficiency virus gene activationEffects of the human immunodeficiency virus type 1 Tat protein on the expression of inflammatory cytokinesContribution of NF-kappa B and Sp1 binding motifs to the replicative capacity of human immunodeficiency virus type 1: distinct patterns of viral growth are determined by T-cell typesHuman immunodeficiency virus Tat transactivation: induction of a tissue-specific enhancer in a nonpermissive cell line.PU.1 binding to ets motifs within the equine infectious anemia virus long terminal repeat (LTR) enhancer: regulation of LTR activity and virus replication in macrophages.Epstein-Barr virus nuclear antigen 2 transactivates the long terminal repeat of human immunodeficiency virus type 1.Efficient transcription and replication of simian immunodeficiency virus in the absence of NF-kappaB and Sp1 binding elements.Genetic instability of live, attenuated human immunodeficiency virus type 1 vaccine strainsProtein kinase C-zeta mediates NF-kappa B activation in human immunodeficiency virus-infected monocytes.O-linked N-acetylglucosaminylation of Sp1 inhibits the human immunodeficiency virus type 1 promoter.A proliferative p53-responsive element mediates tumor necrosis factor alpha induction of the human immunodeficiency virus type 1 long terminal repeatDifferential role of long terminal repeat control elements for the regulation of basal and Tat-mediated transcription of the human immunodeficiency virus in stimulated and unstimulated primary human macrophages.Differential growth kinetics are exhibited by human immunodeficiency virus type 1 TAR mutants.Transcription factor AP-2 regulates human immunodeficiency virus type 1 gene expression.c-Myc and Sp1 contribute to proviral latency by recruiting histone deacetylase 1 to the human immunodeficiency virus type 1 promoter.
P2860
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P2860
Variable role of the long terminal repeat Sp1-binding sites in human immunodeficiency virus replication in T lymphocytes.
description
1991 nî lūn-bûn
@nan
1991年の論文
@ja
1991年論文
@yue
1991年論文
@zh-hant
1991年論文
@zh-hk
1991年論文
@zh-mo
1991年論文
@zh-tw
1991年论文
@wuu
1991年论文
@zh
1991年论文
@zh-cn
name
Variable role of the long term ...... replication in T lymphocytes.
@ast
Variable role of the long term ...... replication in T lymphocytes.
@en
type
label
Variable role of the long term ...... replication in T lymphocytes.
@ast
Variable role of the long term ...... replication in T lymphocytes.
@en
prefLabel
Variable role of the long term ...... replication in T lymphocytes.
@ast
Variable role of the long term ...... replication in T lymphocytes.
@en
P2093
P2860
P1433
P1476
Variable role of the long term ...... replication in T lymphocytes.
@en
P2093
Buckler-White A
Theodore TS
P2860
P304
P407
P577
1991-03-01T00:00:00Z