Aminopyridines potentiate synaptic and neuromuscular transmission by targeting the voltage-activated calcium channel beta subunit - Wikidata - awgldk - TriplyDB

Aminopyridines potentiate synaptic and neuromuscular transmission by targeting the voltage-activated calcium channel beta subunit

Aminopyridines potentiate synaptic and neuromuscular transmission by targeting the voltage-activated calcium channel beta subunit

http://www.wikidata.org/entity/Q24657140

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Pyridostigmine but not 3,4-diaminopyridine exacerbates ACh receptor loss and myasthenia induced in mice by muscle-specific kinase autoantibody Contribution of Kv1.2 voltage-gated potassium channel to D2 autoreceptor regulation of axonal dopamine overflow Paraneoplastic syndromes associated with lung cancer Reduction in voltage-gated K+ channel activity in primary sensory neurons in painful diabetic neuropathy: role of brain-derived neurotrophic factor The effect of calcium channel blockers on the outcome of acute myeloid leukemia.Potentiation of high voltage-activated calcium channels by 4-aminopyridine depends on subunit composition.Up-regulation of Cavβ3 subunit in primary sensory neurons increases voltage-activated Ca2+ channel activity and nociceptive input in neuropathic pain Aging-Related Hyperexcitability in CA3 Pyramidal Neurons Is Mediated by Enhanced A-Type K+ Channel Function and Expression.Molecular Basis of Regulating High Voltage-Activated Calcium Channels by S-Nitrosylation.Neurogenic Causes of Detrusor Underactivity.Presynaptic CaV2.1 calcium channels carrying familial hemiplegic migraine mutation R192Q allow faster recovery from synaptic depression in mouse calyx of Held.Inhibition of neuronal degenerin/epithelial Na+ channels by the multiple sclerosis drug 4-aminopyridine.Dalfampridine: review of its efficacy in improving gait in patients with multiple sclerosis.Distinct intrinsic and synaptic properties of pre-sympathetic and pre-parasympathetic output neurons in Barrington's nucleus.The association between Lambert-Eaton myasthenic syndrome and small cell lung carcinoma Enhancing neural transmission in multiple sclerosis (4-aminopyridine therapy).Treatment in Lambert-Eaton myasthenic syndrome.Prolonged-release fampridine improves walking in a proportion of patients with multiple sclerosis.Advanced therapeutic directions to treat the underactive bladder.GABAB receptor-mediated frequency-dependent and circadian changes in synaptic plasticity modulate retinal input to the suprachiasmatic nucleus.Lambert-Eaton myasthenic syndrome (LEMS): a rare autoimmune presynaptic disorder often associated with cancer.4-Аminopyridine sequesters intracellular Ca(2+) which triggers exocytosis in excitable and non-excitable cells.Mechanism of highly synchronized bilateral hippocampal activity.Involvement of basal and calcium-activated protein kinase C in neurotransmitter secretion in mouse motor synapses.Do premotor interneurons act in parallel on spinal motoneurons and on dorsal horn spinocerebellar and spinocervical tract neurons in the cat?Suppression of Inflammatory Demyelinaton and Axon Degeneration through Inhibiting Kv3 Channels.Blockade of fast A-type and TEA-sensitive potassium channels provide an antiparkinsonian effect in a 6-OHDA animal model.Pharmacological treatments available for the management of underactive bladder in neurological conditions.Pretreatment with potassium channel blockers of 4-aminopyridine and tetraethylammonium attenuates behavioural symptoms of Parkinsonism induced by intrastriatal injection of 6-hydroxydopamine; the role of lipid peroxidation.The α2δ-1-NMDA Receptor Complex Is Critically Involved in Neuropathic Pain Development and Gabapentin Therapeutic Actions.4-aminopyridine induces apoptosis of human acute myeloid leukemia cells via increasing [Ca2+]i through P2X7 receptor pathway.Reply to Meriney and Lacomis: Comment on direct aminopyridine effects on voltage-gated Ca channels Reported direct aminopyridine effects on voltage-gated calcium channels is a high-dose pharmacological off-target effect of no clinical relevance

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Aminopyridines potentiate synaptic and neuromuscular transmission by targeting the voltage-activated calcium channel beta subunit

http://www.wikidata.org/entity/Q24657140

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

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2009年论文

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name

Aminopyridines potentiate syna ...... d calcium channel beta subunit

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Aminopyridines potentiate syna ...... d calcium channel beta subunit

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Aminopyridines potentiate syna ...... d calcium channel beta subunit

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type

label

Aminopyridines potentiate syna ...... d calcium channel beta subunit

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Aminopyridines potentiate syna ...... d calcium channel beta subunit

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Aminopyridines potentiate syna ...... d calcium channel beta subunit

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prefLabel

Aminopyridines potentiate syna ...... d calcium channel beta subunit

@ast

Aminopyridines potentiate syna ...... d calcium channel beta subunit

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Aminopyridines potentiate syna ...... d calcium channel beta subunit

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Journal of Biological Chemistry

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Aminopyridines potentiate syna ...... d calcium channel beta subunit

@en

P2093

De-Pei Li

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Hui-Lin Pan

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Shao-Rui Chen

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Zi-Zhen Wu

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P2860

Structure and functional expression of an omega-conotoxin-sensitive human N-type calcium channel

Effects of phrixotoxins on the Kv4 family of potassium channels and implications for the role of Ito1 in cardiac electrogenesis

Pain perception in mice lacking the beta3 subunit of voltage-activated calcium channels

Reduced expression of A-type potassium channels in primary sensory neurons induces mechanical hypersensitivity

IgG from patients with Lambert-Eaton syndrome blocks voltage-dependent calcium channels.

Twenty years of dendrotoxins.

The I-II loop of the Ca2+ channel alpha1 subunit contains an endoplasmic reticulum retention signal antagonized by the beta subunit.

Pharmacological characterization of five cloned voltage-gated K+ channels, types Kv1.1, 1.2, 1.3, 1.5, and 3.1, stably expressed in mammalian cell lines.

Conus geographus toxins that discriminate between neuronal and muscle sodium channels.

Sea anemone peptides with a specific blocking activity against the fast inactivating potassium channel Kv3.4.

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52

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284

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38030488

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19850918

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2794761

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