Effect of neurofibromatosis type I mutations on a novel pathway for adenylyl cyclase activation requiring neurofibromin and Ras
about
RASopathies: unraveling mechanisms with animal modelsFrom neurodevelopment to neurodegeneration: the interaction of neurofibromin and valosin-containing protein/p97 in regulation of dendritic spine formationFrom Learning to Memory: What Flies Can Tell Us about Intellectual Disability TreatmentNeurofibromin Loss of Function Drives Excessive Grooming in DrosophilaGenes and circuits for olfactory-associated long-term memory in Drosophila.PI3 kinase signaling is involved in Abeta-induced memory loss in Drosophila.The receptor tyrosine kinase Alk controls neurofibromin functions in Drosophila growth and learning.A distinct set of Drosophila brain neurons required for neurofibromatosis type 1-dependent learning and memory.Neurofibromatosis type 1: modeling CNS dysfunction.Neuronal NF1/RAS regulation of cyclic AMP requires atypical PKC activation.Fruit flies and intellectual disability.EGFR signaling promotes self-renewal through the establishment of cell polarity in Drosophila follicle stem cells.Genetic and functional studies implicate synaptic overgrowth and ring gland cAMP/PKA signaling defects in the Drosophila melanogaster neurofibromatosis-1 growth deficiency.Reduced growth of Drosophila neurofibromatosis 1 mutants reflects a non-cell-autonomous requirement for GTPase-Activating Protein activity in larval neurons.Valosin-containing protein and neurofibromin interact to regulate dendritic spine densityNeurofibromatosis-1 heterozygosity impairs CNS neuronal morphology in a cAMP/PKA/ROCK-dependent mannerSyndecan-2 induces filopodia and dendritic spine formation via the neurofibromin-PKA-Ena/VASP pathway.Genetic Dissection of Aversive Associative Olfactory Learning and Memory in Drosophila Larvae.Dopamine deficiency underlies learning deficits in neurofibromatosis-1 miceModeling cognitive dysfunction in neurofibromatosis-1Neurofibromin regulates somatic growth through the hypothalamic-pituitary axisPhysical interaction between neurofibromin and serotonin 5-HT6 receptor promotes receptor constitutive activity.Neurofibromin is the major ras inactivator in dendritic spines.Molecular and cellular mechanisms of learning disabilities: a focus on NF1.Cognitive and behavioral problems in children with neurofibromatosis type 1: challenges and future directions.Neurofibromin is a novel regulator of Ras-induced reactive oxygen species production in mice and humans.The tumor suppressor neurofibromin confers sensitivity to apoptosis by Ras-dependent and Ras-independent pathways.A Drosophila screen identifies neurofibromatosis-1 genetic modifiers involved in systemic and synaptic growth.Modulation of cAMP and ras signaling pathways improves distinct behavioral deficits in a zebrafish model of neurofibromatosis type 1.The mutational spectrum of the NF1 gene in neurofibromatosis type I patients from UAE.
P2860
Q26800379-626AF8DB-323B-4022-B991-45546989D9ABQ27015865-D91BF955-2D3F-4590-B697-56C23F8F41CCQ27020947-D179910A-821F-4F7A-8654-C689CAF13EC3Q27347271-BB6BB995-D8A9-4C5B-A8B9-5363F37FB4ECQ33346322-C3EB3D73-7163-459B-A557-1C68CD9B410FQ33859443-46D10262-348E-4AC9-BD88-A3A15796B376Q34031191-3BBC96A8-0949-47B7-AAE5-0D1969EA7A87Q34051144-508456F2-321B-4741-943C-72C9D20121F6Q34305314-E20C6014-D3C8-4F4F-927D-08ACCB7F2A74Q34575699-B6BBCAE8-DD59-4BCA-8D37-C52555E73F73Q34610614-29C1591F-0E6C-4300-9BD9-E34DBCEF06DFQ34983705-4A195011-308F-4CD0-937E-ACDF1259732EQ35053088-42A1B20D-D385-4F97-803B-6CADBE081F75Q35204747-D6659762-1EF6-4811-BF23-0FFE760CBBA0Q35578562-CAE16C65-AC47-4A64-9FE9-9F61C9BC7C53Q35613659-1DDB8635-1D00-4CEF-A2F6-23779C11E952Q36118445-3776AB7C-8148-46A1-A472-B5531B02D0BDQ36170679-2D8BE362-88D9-4F45-AA3E-532CAD802403Q36717911-B4DFB47F-2B7A-4907-A1FD-5CE23974FBC4Q36756183-7C6125B4-79F5-446D-8894-0C60C1608B6FQ37001127-C717AB63-6B09-4AC8-9548-1A367BA1FBFDQ37379753-637204AC-D8C4-4DB2-B5F1-59397E002B14Q37479474-B7AC1109-91B8-46AC-855B-C846BBF9ABD2Q37719525-A9E9DBCD-17BD-420C-8677-4686BF65A707Q38243763-F459D0DA-0A9D-430D-BA4A-296C4D0D38F0Q39707123-596EB778-6996-4485-9ABF-8C2032C20973Q40210019-DF7BEF32-3F9A-45D4-8A61-D4AD9C2B7913Q42729953-8298FEBC-633B-4C3C-9300-A9472EE69E10Q45264996-35F87191-860F-4B18-B28E-B42A83B3CDCEQ54228124-0C6F91E8-0160-489C-A9F5-E8EF95EBC102
P2860
Effect of neurofibromatosis type I mutations on a novel pathway for adenylyl cyclase activation requiring neurofibromin and Ras
description
2006 nî lūn-bûn
@nan
2006 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
name
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@ast
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@en
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@nl
type
label
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@ast
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@en
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@nl
prefLabel
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@ast
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@en
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@nl
P2093
P2860
P3181
P356
P1476
Effect of neurofibromatosis ty ...... equiring neurofibromin and Ras
@en
P2093
Frances Hannan
James Jiayuan Tong
Peter Nurnberg
Yinghua Zhu
P2860
P304
P3181
P356
10.1093/HMG/DDL023
P407
P50
P577
2006-04-01T00:00:00Z