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MLK3 is required for mitogen activation of B-Raf, ERK and cell proliferationTATA-binding protein (TBP)-like factor (TLF) is a functional regulator of transcription: reciprocal regulation of the neurofibromatosis type 1 and c-fos genes by TLF/TRF2 and TBPBrain lipid binding protein in axon-Schwann cell interactions and peripheral nerve tumorigenesisComputer-based identification of a novel LIMK1/2 inhibitor that synergizes with salirasib to destabilize the actin cytoskeletonC-->U editing of neurofibromatosis 1 mRNA occurs in tumors that express both the type II transcript and apobec-1, the catalytic subunit of the apolipoprotein B mRNA-editing enzymeTumor microenvironment and neurofibromatosis type I: connecting the GAPsA mouse model for Costello syndrome reveals an Ang II-mediated hypertensive conditionEffect of neurofibromatosis type I mutations on a novel pathway for adenylyl cyclase activation requiring neurofibromin and RasSulindac derivatives inhibit cell growth and induce apoptosis in primary cells from malignant peripheral nerve sheath tumors of NF1-patientsMolecular profiling of malignant peripheral nerve sheath tumors associated with neurofibromatosis type 1, based on large-scale real-time RT-PCRAn Update on the Ophthalmologic Features in the PhakomatosesClinical and Neurobiological Relevance of Current Animal Models of Autism Spectrum Disorders.Familial syndromes associated with neuroendocrine tumoursThe molecular biology of WHO grade I astrocytomasGenetically engineered mouse models for functional studies of SKP1-CUL1-F-box-protein (SCF) E3 ubiquitin ligasesThe haploinsufficient hematopoietic microenvironment is critical to the pathological fracture repair in murine models of neurofibromatosis type 1Growth of peripheral and central nervous system tumors is supported by cytoplasmic c-Fos in humans and miceNeurofibromin Loss of Function Drives Excessive Grooming in DrosophilaStructural and biochemical consequences of NF1 associated nontruncating mutations in the Sec14-PH module of neurofibrominDimeric Sfh3 has structural changes in its binding pocket that are associated with a dimer-monomer state transformation induced by substrate bindingThe RasGAP proteins Ira2 and neurofibromin are negatively regulated by Gpb1 in yeast and ETEA in humans.The motor protein kinesin-1 links neurofibromin and merlin in a common cellular pathway of neurofibromatosisStops along the RAS pathway in human genetic diseaseExpression of H-RASV12 in a zebrafish model of Costello syndrome causes cellular senescence in adult proliferating cellsLoss of the nf1 tumor suppressor gene decreases fas antigen expression in myeloid cellsNF2 deficiency promotes tumorigenesis and metastasis by destabilizing adherens junctionsThe neurofibromin GAP-related domain rescues endothelial but not neural crest development in Nf1 miceRas oncogenes: split personalitiesA machine learning classifier trained on cancer transcriptomes detects NF1 inactivation signal in glioblastomaCharacterization of recombinant ELMOD (cell engulfment and motility domain) proteins as GTPase-activating proteins (GAPs) for ARF family GTPases.Genetic predisposition to peripheral nerve neoplasia: diagnostic criteria and pathogenesis of neurofibromatoses, Carney complex, and related syndromesZebrafish neurofibromatosis type 1 genes have redundant functions in tumorigenesis and embryonic development.Cardiac and vascular functions of the zebrafish orthologues of the type I neurofibromatosis gene NFI.Survey of somatic mutations in tuberous sclerosis complex (TSC) hamartomas suggests different genetic mechanisms for pathogenesis of TSC lesionsDownregulation of miR-31, miR-155, and miR-564 in chronic myeloid leukemia cellsGlomus tumors in neurofibromatosis type 1: genetic, functional, and clinical evidence of a novel associationGenome-wide gene expression analysis identifies K-ras as a regulator of alcohol intakeSpred1, a negative regulator of Ras-MAPK-ERK, is enriched in CNS germinal zones, dampens NSC proliferation, and maintains ventricular zone structure.Co-targeting the MAPK and PI3K/AKT/mTOR pathways in two genetically engineered mouse models of schwann cell tumors reduces tumor grade and multiplicity.The Sec14 superfamily and mechanisms for crosstalk between lipid metabolism and lipid signaling
P2860
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P2860
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
NF1 tumor suppressor gene function: narrowing the GAP
@ast
NF1 tumor suppressor gene function: narrowing the GAP
@en
NF1 tumor suppressor gene function: narrowing the GAP
@nl
type
label
NF1 tumor suppressor gene function: narrowing the GAP
@ast
NF1 tumor suppressor gene function: narrowing the GAP
@en
NF1 tumor suppressor gene function: narrowing the GAP
@nl
prefLabel
NF1 tumor suppressor gene function: narrowing the GAP
@ast
NF1 tumor suppressor gene function: narrowing the GAP
@en
NF1 tumor suppressor gene function: narrowing the GAP
@nl
P3181
P1433
P1476
NF1 tumor suppressor gene function: narrowing the GAP
@en
P2093
P304
P3181
P356
10.1016/S0092-8674(01)00245-8
P407
P577
2001-02-23T00:00:00Z